35 research outputs found
Олесь Бабій - співець слави січових стрільців
The Salamanca Formation of the San Jorge Basin (Patagonia, Argentina) preserves critical records of Southern Hemisphere Paleocene biotas, but its age remains poorly resolved, with estimates ranging from Late Cretaceous to middle Paleocene. We report a multi-disciplinary geochronologic study of the Salamanca Formation and overlying Río Chico Group in the western part of the basin. New constraints include (1) an 40Ar/39Ar age determination of 67.31 ± 0.55 Ma from a basalt flow underlying the Salamanca Formation, (2) micropaleontological results indicating an early Danian age for the base of the Salamanca Formation, (3) laser ablation HR-MC-ICP-MS (high resolution-multi collector-inductively coupled plasma-mass spectrometry) U-Pb ages and a high-resolution TIMS (thermal ionization mass spectrometry) age of 61.984 ± 0.041(0.074)[0.100] Ma for zircons from volcanic ash beds in the Peñas Coloradas Formation (Río Chico Group), and (4) paleomagnetic results indicating that the Salamanca Formation in this area is entirely of normal polarity, with reversals occurring in the Río Chico Group. Placing these new age constraints in the context of a sequence stratigraphic model for the basin, we correlate the Salamanca Formation in the study area to Chrons C29n and C28n, with the Banco Negro Inferior (BNI), a mature widespread fossiliferous paleosol unit at the top of the Salamanca Formation, corresponding to the top of Chron C28n. The diverse paleobotanical assemblages from this area are here assigned to C28n (64.67–63.49 Ma), ∼2–3 million years older than previously thought, adding to growing evidence for rapid Southern Hemisphere floral recovery after the Cretaceous-Paleogene extinction. Important Peligran and “Carodnia” zone vertebrate fossil assemblages from coastal BNI and Peñas Coloradas exposures are likely older than previously thought and correlate to the early Torrejonian and early Tiffanian North American Land Mammal Ages, respectively
Partially Materialized Digest Scheme: An Efficient Verification Method for Outsourced Databases
Variational description of multi-fluid hydrodynamics: Uncharged fluids
We present a formalism for Newtonian multi-fluid hydrodynamics derived from
an unconstrained variational principle. This approach provides a natural way of
obtaining the general equations of motion for a wide range of hydrodynamic
systems containing an arbitrary number of interacting fluids and superfluids.
In addition to spatial variations we use ``time shifts'' in the variational
principle, which allows us to describe dissipative processes with entropy
creation, such as chemical reactions, friction or the effects of external
non-conservative forces. The resulting framework incorporates the
generalization of the entrainment effect originally discussed in the case of
the mixture of two superfluids by Andreev and Bashkin. In addition to the
conservation of energy and momentum, we derive the generalized conservation
laws of vorticity and helicity, and the special case of Ertel's theorem for the
single perfect fluid.
We explicitly discuss the application of this framework to thermally
conducting fluids, superfluids, and superfluid neutron star matter. The
equations governing thermally conducting fluids are found to be more general
than the standard description, as the effect of entrainment usually seems to be
overlooked in this context. In the case of superfluid He4 we recover the
Landau--Khalatnikov equations of the two-fluid model via a translation to the
``orthodox'' framework of superfluidity, which is based on a rather awkward
choice of variables. Our two-fluid model for superfluid neutron star matter
allows for dissipation via mutual friction and also ``transfusion'' via
beta-reactions between the neutron fluid and the proton-electron fluid.Comment: uses RevTeX 4; 20 pages. To appear in PRD. v2: removed discussion of
charged fluids and coupling to electromagnetic fields, which are submitted as
a separate paper for a clearer presentation v3: fixed typo in Eq.(9), updated
some reference
Hypersurface-Invariant Approach to Cosmological Perturbations
Using Hamilton-Jacobi theory, we develop a formalism for solving
semi-classical cosmological perturbations which does not require an explicit
choice of time-hypersurface. The Hamilton-Jacobi equation for gravity
interacting with matter (either a scalar or dust field) is solved by making an
Ansatz which includes all terms quadratic in the spatial curvature.
Gravitational radiation and scalar perturbations are treated on an equal
footing. Our technique encompasses linear perturbation theory and it also
describes some mild nonlinear effects. As a concrete example of the method, we
compute the galaxy-galaxy correlation function as well as large-angle microwave
background fluctuations for power-law inflation, and we compare with recent
observations.Comment: 51 pages, Latex 2.09 ALBERTA THY/20-94, DAMTP R94/25 To appear in
Phys. Rev.
Wrinkle and Collapsing Process of Inflatable Tubes Under Bending Loads by Finite Element Analyses
The histamine H4 receptor is a potent inhibitor of adhesion-dependent degranulation in human neutrophils
The histamine H4 receptor regulates the inflammatory response. However, it is not known whether this receptor has a functional role in human neutrophils. We found that fMLP (1 μM), but not histamine (0.1–1 μM), induced Mac-1-dependent adhesion, polarization, and degranulation (release of lactoferrin). A pretreatment of neutrophils with histamine (0.001–1 μM) or JNJ 28610244 (0.1–10 μM), a specific H4 receptor agonist, led to inhibition of degranulation. Total inhibition of degranulation was obtained with 0.1 μM histamine and 10 μM JNJ 28610244. Furthermore, such inhibition by histamine of degranulation was reversed by JNJ 7777120 and JNJ 28307474, two selective H4 receptor antagonists. However, neither histamine nor the H4 receptor agonist JNJ 28610244 prevented fMLP-induced, Mac-1-dependent adhesion, indicating that the H4 receptor may block signals emanating from Mac-1-controlling degranulation. Likewise, engagement of the H4 receptor by the selective agonist JNJ 28610244 blocked Mac-1-dependent activation of p38 MAPK, the kinase that controls neutrophil degranulation. We also show expression of the H4 receptor at the mRNA level in ultrapure human neutrophils and myeloid leukemia PLB-985 cells. We concluded that engagement of this receptor by selective H4 receptor agonists may represent a good, therapeutic approach to accelerate resolution of inflammation