7 research outputs found
Calcium fluxes in juvenile tilapia, Oreochromis mossambicus, exposed to sublethal waterborne Cd, Cu or mixtures of these metals
Contains fulltext :
11062.pdf (publisher's version ) (Open Access
Effects of combined waterborne Cd and Cu exposures on ionic composition and plasma cortisol in tilapia, Oreochromis mossambicus
Contains fulltext :
10963.pdf (publisher's version ) (Open Access
Integrated physiological response of tilapia, Oreochromis mossambicus, to sublethal copper exposure
Contains fulltext :
10961.pdf (publisher's version ) (Open Access
Interactions between copper and cadmium during single and combined exposure in juvenile tilapia oreochromis-mossambicus: influence of feeding condition on whole-body metal accumulation and the effect of the metals on tissue water and ion content
Contains fulltext :
10921.pdf (publisher's version ) (Open Access
Interactions between copper and cadmium during single or combined metal exposures in the teleost fish Oreochromis mossambicus : heavy metal accumulation and endocrine events
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The pathology of chronic erosive dermatopathy in Murray cod, Maccullochella peelii peelii (Mitchell)
Chronic erosive dermatopathy (CED) is a disease of intensively farmed Murray cod in Australia that has been reported in association with the use of groundwater (mechanically extracted from shallow boreholes) supplies. CED results in focal ulceration of the skin overlying sensory canals of the head and flanks. Trials were conducted at an affected fish farm to study the development of the condition, both in Murray cod and in goldfish, and also to assess the reported recovery of lesions when affected fish were transferred to river water. Grossly, lesions began after 2-3 weeks with degeneration of tissue at the periphery of pores communicating with the sensory canals. Widening of these pores along the axis of the canals resulted from a loss of tissue covering the canal. Histopathologically, hyperplasia of the canal epithelial lining was seen after 3 weeks in borehole water and subsequent necrosis and sloughing of this tissue resulted in the loss of the canal roof. Canal regeneration occurred when fish were transferred from borehole water into river water. The lack of lesions in other organs and the pattern of lesion development support exposure to waterborne factors as the most likely aetiology