Modeling the Effect of Oil Price on Global Fertilizer Prices

The main purpose of this paper is to evaluate the effect of crude oil price on global fertilizer prices in both the mean and volatility. The endogenous structural breakpoint unit root test, the autoregressive distributed lag (ARDL) model, and alternative volatility models, including the generalized autoregressive conditional heteroskedasticity (GARCH) model, Exponential GARCH (EGARCH) model, and GJR model, are used to investigate the relationship between crude oil price and six global fertilizer prices. Weekly data for 2003-2008 for the seven price series are analyzed. The empirical results from ARDL show that most fertilizer prices are significantly affected by the crude oil price, which explains why global fertilizer prices reached a peak in 2008. We also find that that the volatility of global fertilizer prices and crude oil price from March to December 2008 are higher than in other periods, and that the peak crude oil price caused greater volatility in the crude oil price and global fertilizer prices. As volatility invokes financial risk, the relationship between oil price and global fertilizer prices and their associated volatility is important for public policy relating to the development of optimal energy use, global agricultural production, and financial integration.volatility;crude oil price;global fertilizer price;non-renewable fertilizers;structural breakpoint unit root test

Reduction of mm-Regular Noncrossing Partitions

In this paper, we present a reduction algorithm which transforms $m$-regular partitions of $[n]=\{1, 2, ..., n\}$ to $(m-1)$-regular partitions of $[n-1]$. We show that this algorithm preserves the noncrossing property. This yields a simple explanation of an identity due to Simion-Ullman and Klazar in connection with enumeration problems on noncrossing partitions and RNA secondary structures. For ordinary noncrossing partitions, the reduction algorithm leads to a representation of noncrossing partitions in terms of independent arcs and loops, as well as an identity of Simion and Ullman which expresses the Narayana numbers in terms of the Catalan numbers

Riordan Paths and Derangements

Riordan paths are Motzkin paths without horizontal steps on the x-axis. We establish a correspondence between Riordan paths and $(321,3\bar{1}42)$-avoiding derangements. We also present a combinatorial proof of a recurrence relation for the Riordan numbers in the spirit of the Foata-Zeilberger proof of a recurrence relation on the Schr\"oder numbers.Comment: 9 pages, 2 figure

Fluctuations of Entropy Production in Partially Masked Electric Circuits: Theoretical Analysis

In this work we perform theoretical analysis about a coupled RC circuit with constant driven currents. Starting from stochastic differential equations, where voltages are subject to thermal noises, we derive time-correlation functions, steady-state distributions and transition probabilities of the system. The validity of the fluctuation theorem (FT) is examined for scenarios with complete and incomplete descriptions.Comment: 4 pages, 1 figur

Studies of acoustic emission from point and extended sources

The use of simulated and controlled acoustic emission signals forms the basis of a powerful tool for the detailed study of various deformation and wave interaction processes in materials. The results of experiments and signal analyses of acoustic emission resulting from point sources such as various types of indentation-produced cracks in brittle materials and the growth of fatigue cracks in 7075-T6 aluminum panels are discussed. Recent work dealing with the modeling and subsequent signal processing of an extended source of emission in a material is reviewed. Results of the forward problem and the inverse problem are presented with the example of a source distributed through the interior of a specimen

DLC2 modulates angiogenic responses in vascular endothelial cells by regulating cell attachment and migration.

Deleted in liver cancer 1 (DLC1) is a RhoGTPase activation protein-containing tumor suppressor that associates with various types of cancer. Although DLC2 shares a similar domain structure with that of DLC1, the function of DLC2 is not well characterized. Here, we describe the expression and ablation of DLC2 in mice using a reporter-knockout approach. DLC2 is expressed in several tissues and in endothelial cells (ECs) of blood vessels. Although ECs and blood vessels show no histological abnormalities and mice appear overall healthy, DLC2-mutant mice display enhanced angiogenic responses induced by matrigel and by tumor cells. Silencing of DLC2 in human ECs has reduced cell attachment, increased migration, and tube formation. These changes are rescued by silencing of RhoA, suggesting that the process is RhoA pathway dependent. These results indicate that DLC2 is not required for mouse development and normal vessel formation, but may protect mouse from unwanted angiogenesis induced by, for example, tumor cells