29 research outputs found

    SARNET2 benchmark on air ingress experiments QUENCH-10, -16

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    International audienceThe QUENCH-10 (Q-10) and QUENCH-16 (Q-16) experiments were chosen as a SARNET2 code benchmark (SARNET2-COOL-D5.4) exercise to assess the status of modelling air ingress sequences and to compare the capabilities of the various codes used for accident analyses, specifically ATHLET-CD (GRS and RUB), ICARE-CATHARE (IRSN), MAAP (EDF), MELCOR (INRNE and PSI), SOCRAT (IBRAE), and RELAP/SCDAPSim (PSI). Both experiments addressed air ingress into an overheated core following earlier partial oxidation in steam. Q-10 was performed with extensive preoxidation, moderate/high air flow rate and high temperatures at onset of reflood (max Tpct = 2200 K), while Q-16 was performed with limited preoxidation, low air flow rate and relative low temperatures at reflood initiation (max Tpct = 1870 K). Variables relating to the major signatures (thermal response, hydrogen generation, oxide layer development, oxygen and nitrogen consumption and reflood behaviour) were compared globally and/or at selected locations. In each simulation, the same input models and assumptions are used for both experiments, differing only in respect of the boundary conditions. However, some slight idealisations were made to the assumed boundary conditions in order to avoid ambiguities in the code-to-code comparisons; in this way, it was possible to focus more easily on the key phenomena and hence make the results of the exercise more transparent. Remarks are made concerning the capability of physical modelling within the codes, description of the experiment facility and test conduct as specified in the code input, and code limitations that might warrant additional research to support model improvements, especially the modelling of nitride formation and melt oxidation. © 2014 Elsevier Ltd. All rights reserved

    Modeling technology diffusion of beef cattle in Brazil using a cellular automata.

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    We introduce a cellular automata model representing the diffusion of technological intensification of beef cattle considering farmer's preferences. We also analyze the theoretical properties of the model and the sensitivity of key parameters.Organizadores: Maria Fernanda Moura, Jayme Garcia Arnal Barbedo, Alaine Margarete GuimarĂŁes, Valter Castelhano de Oliveira. SBIAgro 2019

    Protective effects of green tea against hepatic injury induced by high-cholesterol diet in rats: histopathological analysis, oxidative DNA damage and COX-2 expression

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    Conselho Nacional de Desenvolvimento CientĂ­fico e TecnolĂłgico (CNPq)Purpose The goal of this study was to investigate whether daily administration of green tea is able to protect the liver injury induced by cholesterol. Methods Male Wistar rats (n = 24) were distributed into four groups: group 1, negative control; group 2, cholesterol at 1% (w/w) in the diet treated for 5 weeks; group 3, cholesterol at 1% treated for 5 weeks and green tea at 1% (w/v) in drinking water in the last week only and group 4, cholesterol and green tea at 1% in drinking water for 5 weeks. Results The results pointed out that treatment with green tea in the last week (group 3) showed mild degenerative changes of liver tissue in cholesterol exposed group when compared to group 2. Green tea aqueous extract was not able to reduce cholesterol levels, that is, no significant statistical differences (p > 0.05) were noticed when compared to positive control group. Nevertheless, green tea was able to decrease oxidative deoxyribonucleic acid (DNA) damage either to peripheral blood or to liver cells as depicted by significant statistical differences (p < 0.05) in the mean tail moment between groups treated with green tea and cholesterol and cholesterol only. Furthermore, histomorphometric analysis of COX-2 expression revealed that in groups exposed to green tea they were significantly decreased (p < 0.05), regardless of time exposure adopted. Conclusion Taken together, our results suggest that daily administration of green tea for at least 7 days displays some preventive properties as indicated by COX-2 downregulation and decreased oxidative DNA damage.54965974Conselho Nacional de Desenvolvimento CientĂ­fico e TecnolĂłgico (CNPq)Conselho Nacional de Desenvolvimento CientĂ­fico e TecnolĂłgico (CNPq
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