190 research outputs found
Cellular mechanisms by which proinsulin C-peptide prevents insulin-induced neointima formation in human saphenous vein
- Author
- AE Kitabchi
- B Huang
- C Indolfi
- CC Liang
- CC Wang
- CE Hills
- D Tanous
- D Walcher
- DE Cutlip
- DM Nathan
- E Foster
- E Lindahl
- EA Forsyth
- EJ Wallitt
- F Ledru
- G Grunberger
- G Zerbini
- H Suma
- IG Joshua
- J Herlitz
- J Wahren
- J Wahren
- J Wahren
- J Wahren
- JP Despres
- KE Porter
- KE Porter
- L Nordquist
- M Hoffman
- MR Gitman
- NA Turner
- NA Turner
- NA Turner
- NA Turner
- NM Al Rasheed
- P Luppi
- PH Stone
- PK Aley
- R Kornowski
- R Lorusso
- R Raghow
- R Rigler
- RH Zhou
- S Sjoberg
- S Sjoberg
- SH Park
- T Hakala
- V Cifarelli
- VH Thourani
- W Duckworth
- Y Kobayashi
- Publication venue
- Springer-Verlag
- Publication date
- 01/01/2010
- Field of study
AIMS/HYPOTHESIS: Endothelial cells (ECs) and smooth muscle cells (SMCs) play key roles in the development of intimal hyperplasia in saphenous vein (SV) bypass grafts. In diabetic patients, insulin administration controls hyperglycaemia but cardiovascular complications remain. Insulin is synthesised as a pro-peptide, from which C-peptide is cleaved and released into the circulation with insulin; exogenous insulin lacks C-peptide. Here we investigate modulation of human SV neointima formation and SV-EC and SV-SMC function by insulin and C-peptide. METHODS: Effects of insulin and C-peptide on neointima formation (organ cultures), EC and SMC proliferation (cell counting), EC migration (scratch wound), SMC migration (Boyden chamber) and signalling (immunoblotting) were examined. A real-time RT-PCR array identified insulin-responsive genes, and results were confirmed by real-time RT-PCR. Targeted gene silencing (siRNA) was used to assess functional relevance. RESULTS: Insulin (100 nmol/l) augmented SV neointimal thickening (70% increase, 14 days), SMC proliferation (55% increase, 7 days) and migration (150% increase, 6 h); effects were abrogated by 10 nmol/l C-peptide. C-peptide did not affect insulin-induced Akt or extracellular signal-regulated kinase signalling (15 min), but array data and gene silencing implicated sterol regulatory element binding transcription factor 1 (SREBF1). Insulin (1-100 nmol/l) did not modify EC proliferation or migration, whereas 10 nmol/l C-peptide stimulated EC proliferation by 40% (5 days). CONCLUSIONS/INTERPRETATION: Our data support a causative role for insulin in human SV neointima formation with a novel counter-regulatory effect of proinsulin C-peptide. Thus, C-peptide can limit the detrimental effects of insulin on SMC function. Co-supplementing insulin therapy with C-peptide could improve therapy in insulin-treated patients
Energy and macronutrient intake and dietary pattern among school children in Bahrain: a cross-sectional study
- Author
- A Must
- Al Sendi AM
- Al Sendi Aneesa M
- American Academy of Pediatrics
- AO Musaiger
- AO Musaiger
- AO Musaiger
- AO Musaiger
- AO Musaiger
- AO Musaiger
- AO Musaiger
- AO Musaiger
- AO Musaiger
- AWM Abdul Wahab
- B Koletzko
- C Chunming
- C Power
- CL Williams
- CL Williams
- Committee on School Health
- DC Nieman
- DC Welten
- Department of Health
- Dietary Reference Values for Food Energy
- F Ahmed
- FE Thompson
- First Data Bank Inc
- Food and Agriculture Organization of the United Nations
- G Samuelson
- GC Frank
- GS Berenson
- H Clavien
- H Crawley
- HC McGill
- JAA Cruz
- KM Flegal
- M De Onis
- MF Rolland-Cachera
- MF Rolland-Cachera
- N Gharib
- Nadia Gharib
- NM Gharib
- Parveen Rasheed
- RD Lee
- Report of a WHO Expert Committee
- RM Krauss
- RM Lauer
- RP Troiano
- S Bowman
- TA Nicklas
- TA Nicklas
- TA Nicklas
- Wh Dietz
- Publication venue
- BioMed Central
- Publication date
- 01/01/2011
- Field of study
Abstract Background Obesity is increasing in Bahrain and there is lack of information on the energy and macronutrient intake of children. The objective of this research was to study the energy and macronutrient intake as well as food frequency pattern of Bahraini school children. Methods This is a cross-sectional descriptive study conducted on Bahraini school boys and girls aged 6-18 years from all the 11 populated regions of the country. Data on food intake consisted of a 24-hour dietary recall and was obtained by interviewing a sub-sample of the study population. Information was also obtained through a self-administered questionnaire for the entire sample on the weekly frequency of food items that were grouped into 7 categories based on similarity of nutrient profiles. Dietary analysis was performed using the Nutritionist 5 (First Data Bank Version 1.6 1998). Results While the average energy intake of students was close to the Estimated Average Requirements of the UK Reference standards, protein intake substantially exceeded the Reference Nutrient Intake values as did daily sugar consumption. Dietary fiber fell short of the Dietary Recommended Values (UK) and 36%-50% students exceeded the Energy % limits for total fat, saturated fat and cholesterol. The Polyunsaturated: Saturated fat ratio remained at an unacceptable level of 0.6 for girls and boys. While sweets, snacks and regular soda drinks were popular, milk, fruits and vegetables were not commonly consumed. Conclusions High sugar consumption, low intake of dietary fiber and high energy % of saturated fat and dietary cholesterol by many Bahraini children, is likely to increase their risk of obesity and cardiovascular diseases in later life. Nutrition education programs in schools should emphasize the importance of healthy balanced diets for growth and health maintenance of children as well as dietary prevention of diseases.</p
Megalin/LRP2 Expression Is Induced by Peroxisome Proliferator-Activated Receptor -Alpha and -Gamma: Implications for PPARs' Roles in Renal Function
- Author
- A Cimini
- A Gauthier
- A IJpenberg
- A Knutson
- A Levin
- A Nykjaer
- A Tojo
- A Tojo
- AA Bulhak
- AA Eddy
- AC Calkin
- B Erranz
- BA Stanton
- Ben Ko
- BH Lee
- BM Forman
- C Caruso-Neves
- C Mysiorek
- Carlos Céspedes
- Carlos P. Vio
- CE Fleming
- CL Rokos
- CP Vio
- CW Park
- CW Park
- E Carro
- E Imano
- E Letavernier
- E Vinuesa
- EI Christensen
- EI Christensen
- Felipe Cabezas
- G Carvajal
- G Chinetti
- G Chinetti
- G Chinetti-Gbaguidi
- G Wolf
- G Wolf
- GD Pan
- H Giral
- H Hama
- HL Fang
- J Herz
- JC Ansquer
- JE Toblli
- Jonathan Lagos
- JR Leheste
- K Odera
- LG Ardiles
- M Ambjorn
- M Hosojima
- M Marino
- M Rakhshandehroo
- M Schupp
- M Schupp
- María-Paz Marzolo
- ME Maurer
- ME Thomas
- MI Yuseff
- Miguel Bronfman
- MO Dietrich
- MO Pedersen
- MP Marzolo
- MR Hayden
- NC Inestrosa
- NM Al-Rasheed
- O Braissant
- OS Gardner
- P Kumpers
- P Madeddu
- P Mardones
- Q Yang
- R Gonzalez-Villalobos
- R Gonzalez-Villalobos
- R Li
- R Spoelgen
- RA McCarthy
- RA Orlando
- RP Czekay
- RS Chung
- S Cui
- S Kantarci
- S Zafiriou
- SA Kliewer
- SC Benson
- SE Guggino
- SP Salas
- T Banks
- T Itoh
- T Nakamura
- T Nakamura
- T Nakamura
- T Shibata
- T Watanabe
- T Yang
- TE Willnow
- TJ Roberts
- W Liu
- Y Fujigaki
- Y Guan
- Y Guan
- Y Li
- Y Miyazaki
- Y Oyama
- Z Zou
- Publication venue
- Public Library of Science
- Publication date
- 02/02/2011
- Field of study
BACKGROUND: Megalin is a large endocytic receptor with relevant functions during development and adult life. It is expressed at the apical surface of several epithelial cell types, including proximal tubule cells (PTCs) in the kidney, where it internalizes apolipoproteins, vitamins and hormones with their corresponding carrier proteins and signaling molecules. Despite the important physiological roles of megalin little is known about the regulation of its expression. By analyzing the human megalin promoter, we found three response elements for the peroxisomal proliferator-activated receptor (PPAR). The objective of this study was to test whether megalin expression is regulated by the PPARs. METHODOLOGY/PRINCIPAL FINDINGS: Treatment of epithelial cell lines with PPARα or PPARγ ligands increased megalin mRNA and protein expression. The stimulation of megalin mRNA expression was blocked by the addition of specific PPARα or PPARγ antagonists. Furthermore, PPAR bound to three PPAR response elements located in the megalin promoter, as shown by EMSA, and PPARα and its agonist activated a luciferase construct containing a portion of the megalin promoter and the first response element. Accordingly, the activation of PPARα and PPARγ enhanced megalin expression in mouse kidney. As previously observed, high concentrations of bovine serum albumin (BSA) decreased megalin in PTCs in vitro; however, PTCs pretreated with PPARα and PPARγ agonists avoided this BSA-mediated reduction of megalin expression. Finally, we found that megalin expression was significantly inhibited in the PTCs of rats that were injected with BSA to induce tubulointerstitial damage and proteinuria. Treatment of these rats with PPARγ agonists counteracted the reduction in megalin expression and the proteinuria induced by BSA. CONCLUSIONS: PPARα/γ and their agonists positively control megalin expression. This regulation could have an important impact on several megalin-mediated physiological processes and on pathophysiologies such as chronic kidney disease associated with diabetes and hypertension, in which megalin expression is impaired
Measurement and interpretation of same-sign W boson pair production in association with two jets in pp collisions at s = 13 TeV with the ATLAS detector
- Author
- Aad G
- Abbott B
- Abeling K
- Abicht NJ
- Abidi SH
- Aboulhorma A
- Abramowicz H
- Abreu H
- Abulaiti Y
- Abusleme Hoffman AC
- Acharya BS
- Adam Bourdarios C
- Adamczyk L
- Adamek L
- Addepalli SV
- Addison MJ
- Adelman J
- Adiguzel A
- Adye T
- Affolder AA
- Afik Y
- Agaras MN
- Agarwala J
- Aggarwal A
- Agheorghiesei C
- Ahmad A
- Ahmadov F
- Ahmed WS
- Ahuja S
- Ai X
- Aielli G
- Aikot A
- Ait Tamlihat M
- Aitbenchikh B
- Aizenberg I
- Akbiyik M
- Akimov AV
- Akiyama D
- Akolkar NN
- Al Khoury K
- Alberghi GL
- Albert J
- Albicocco P
- Albouy GL
- Alderweireldt S
- Aleksa M
- Aleksandrov IN
- Alexa C
- Alexopoulos T
- Alfonsi F
- Algren M
- Alhroob M
- Ali B
- Ali HMJ
- Ali S
- Alibocus SW
- Aliev M
- Alimonti G
- Alkakhi W
- Allaire C
- Allbrooke BMM
- Allen JF
- Allendes Flores CA
- Allport PP
- Aloisio A
- Alonso F
- Alpigiani C
- Alvarez Estevez M
- Alvarez Fernandez A
- Alves Cardoso M
- Alviggi MG
- Aly M
- Amaral Coutinho Y
- Ambler A
- Amelung C
- Amerl M
- Ames CG
- Amidei D
- Amor Dos Santos SP
- Amos KR
- Ananiev V
- Anastopoulos C
- Andeen T
- Anders JK
- Andrean SY
- Andreazza A
- Angelidakis S
- Angerami A
- Anisenkov AV
- Annovi A
- Antel C
- Anthony MT
- Antipov E
- Antonelli M
- Anulli F
- Aoki M
- Aoki T
- Aparisi Pozo JA
- Aparo MA
- Aperio Bella L
- Appelt C
- Apyan A
- Aranzabal N
- Arcangeletti C
- Arce ATH
- Arena E
- Arguin J-F
- Argyropoulos S
- Arling J-H
- Arnaez O
- Arnold H
- Artoni G
- Asada H
- Asai K
- Asai S
- Asbah NA
- Assamagan K
- Astalos R
- Atashi S
- Atkin RJ
- Atkinson M
- Atmani H
- Atmasiddha PA
- Augsten K
- Auricchio S
- Auriol AD
- Austrup VA
- Avolio G
- Axiotis K
- Azuelos G
- Babal D
- Bachacou H
- Bachas K
- Bachiu A
- Backman F
- Badea A
- Bagnaia P
- Bahmani M
- Bailey AJ
- Bailey VR
- Baines JT
- Baines L
- Bakalis C
- Baker OK
- Bakos E
- Bakshi Gupta D
- Balakrishnan V
- Balasubramanian R
- Baldin EM
- Balek P
- Ballabene E
- Balli F
- Baltes LM
- Balunas WK
- Balz J
- Banas E
- Bandieramonte M
- Bandyopadhyay A
- Bansal S
- Barak L
- Barakat M
- Barberio EL
- Barberis D
- Barbero M
- Barel MZ
- Barends KN
- Barillari T
- Barisits M-S
- Barklow T
- Baron Moreno DA
- Baron P
- Baroncelli A
- Barone G
- Barr AJ
- Barr JD
- Barranco Navarro L
- Barreiro Guimarães da Costa J
- Barreiro F
- Barron U
- Barros Teixeira MG
- Barsov S
- Bartels F
- Bartoldus R
- Barton AE
- Bartos P
- Basan A
- Baselga M
- Bassalat A
- Basso MJ
- Basson CR
- Bates RL
- Batlamous S
- Batley JR
- Batool B
- Battaglia M
- Battulga D
- Bauce M
- Bauer M
- Bauer P
- Bazzano Hurrell LT
- Beacham JB
- Beau T
- Beauchemin PH
- Becherer F
- Bechtle P
- Beck HP
- Becker K
- Becot C
- Beddall AJ
- Bednyakov VA
- Bee CP
- Beemster LJ
- Beermann TA
- Begalli M
- Begel M
- Behera A
- Behr JK
- Beirer JF
- Beisiegel F
- Belfkir M
- Bella G
- Bellagamba L
- Bellerive A
- Bellos P
- Beloborodov K
- Belyaev NL
- Benchekroun D
- Bendebba F
- Benhammou Y
- Benoit M
- Bensinger JR
- Bentvelsen S
- Beresford L
- Beretta M
- Bergeaas Kuutmann E
- Berger N
- Bergmann B
- Beringer J
- Bernardi G
- Bernius C
- Bernlochner FU
- Bernon F
- Berry T
- Berta P
- Berthold A
- Bertram IA
- Bethke S
- Betti A
- Bevan AJ
- Bhamjee M
- Bhatta S
- Bhattacharya DS
- Bhattarai P
- Bhopatkar VS
- Bi R
- Bianchi RM
- Bianco G
- Biebel O
- Bielski R
- Biglietti M
- Billoud TRV
- Bindi M
- Bingul A
- Bini C
- Biondini A
- Birch-sykes CJ
- Bird GA
- Birman M
- Biros M
- Biryukov S
- Bisanz T
- Bisceglie E
- Biswal JP
- Biswas D
- Bitadze A
- Bjørke K
- Bloch I
- Blocker C
- Blue A
- Blumenschein U
- Blumenthal J
- Bobbink GJ
- Bobrovnikov VS
- Boehler M
- Boehm B
- Bogavac D
- Bogdanchikov AG
- Bohm C
- Boisvert V
- Bokan P
- Bold T
- Bomben M
- Bona M
- Boonekamp M
- Booth CD
- Borbély AG
- Bordulev IS
- Borecka-Bielska HM
- Borgna LS
- Borissov G
- Bortoletto D
- Boscherini D
- Bosman M
- Bossio Sola JD
- Bouaouda K
- Bouchhar N
- Boudreau J
- Bouhova-Thacker EV
- Boumediene D
- Bouquet R
- Boveia A
- Boyd J
- Boye D
- Boyko IR
- Bracinik J
- Brahimi N
- Brandt G
- Brandt O
- Braren F
- Brau B
- Brau JE
- Brener R
- Brenner L
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- Bressler S
- Britton D
- Britzger D
- Brock I
- Brooijmans G
- Brooks WK
- Brost E
- Brown LM
- Bruce LE
- Bruckler TL
- Bruckman de Renstrom PA
- Bruni A
- Bruni G
- Bruschi M
- Bruscino N
- Brüers B
- Buanes T
- Buat Q
- Buchin D
- Buckley AG
- Bulekov O
- Bullard BA
- Burdin S
- Burgard CD
- Burger AM
- Burghgrave B
- Burlayenko O
- Burr JTP
- Burton CD
- Burzynski JC
- Busch EL
- Bussey PJ
- Butler JM
- Buttar CM
- Butterworth JM
- Buttinger W
- Buxo Vazquez CJ
- Buzykaev AR
- Büscher V
- Cabrera Urbán S
- Cadamuro L
- Caforio D
- Cai H
- Cai Y
- Cairo VMM
- Cakir O
- Calace N
- Calafiura P
- Calderini G
- Calfayan P
- Callea G
- Caloba LP
- Calvet D
- Calvet S
- Calvet TP
- Calvetti M
- Camacho Toro R
- Camarda S
- Camarero Munoz D
- Camarri P
- Camerlingo MT
- Cameron D
- Camincher C
- Campanelli M
- Camplani A
- Canale V
- Canesse A
- Cantero J
- Cao Y
- Capocasa F
- Capua M
- Carbone A
- Cardarelli R
- Cardenas JCJ
- Cardillo F
- Carli T
- Carlino G
- Carlotto JI
- Carlson BT
- Carlson EM
- Carminati L
- Carnelli A
- Carnesale M
- Caron S
- Carquin E
- Carratta G
- Carrio Argos F
- Carrá S
- Carter JWS
- Carter TM
- Casado MP
- Caspar M
- Castiglia EG
- Castillo Garcia L
- Castillo Gimenez V
- Castillo FL
- Castro NF
- Catinaccio A
- Catmore JR
- Cavaliere V
- Cavalli N
- Cavasinni V
- Cekmecelioglu YC
- Celebi E
- Celli F
- Centonze MS
- Cepaitis V
- Cerny K
- Cerqueira AS
- Cerri A
- Cerrito L
- Cerutti F
- Cervato B
- Cervelli A
- Cesarini G
- Cetin SA
- Chadi Z
- Chakraborty D
- Chan J
- Chan WY
- Chapman JD
- Chapon E
- Chargeishvili B
- Charlton DG
- Charman TP
- Chatterjee M
- Chauhan C
- Chekanov S
- Chekulaev SV
- Chelkov GA
- Chen A
- Chen B
- Chen B
- Chen H
- Chen H
- Chen J
- Chen J
- Chen M
- Chen S
- Chen SJ
- Chen X
- Chen X
- Chen Y
- Cheng CL
- Cheng HC
- Cheong S
- Cheplakov A
- Cheremushkina E
- Cherepanova E
- Cherkaoui El Moursli R
- Cheu E
- Cheung K
- Chevalier L
- Chiarella V
- Chiarelli G
- Chiedde N
- Chiodini G
- Chisholm AS
- Chitan A
- Chitishvili M
- Chizhov MV
- Choi K
- Chomont AR
- Chou Y
- Chow EYS
- Chowdhury T
- Chu KL
- Chu MC
- Chu X
- Chudoba J
- Chwastowski JJ
- Cieri D
- Ciesla KM
- Cindro V
- Ciocio A
- Cirotto F
- Citron ZH
- Citterio M
- Ciubotaru DA
- Ciungu BM
- Clark A
- Clark PJ
- Clavijo Columbie JM
- Clawson SE
- Clement C
- Clercx J
- Coadou Y
- Cobal M
- Coccaro A
- Coelho Barrue RF
- Coelho Lopes De Sa R
- Coelli S
- Cohen H
- Coimbra AEC
- Cole B
- Collot J
- Conde Muiño P
- Connell MP
- Connell SH
- Connelly IA
- Conroy EI
- Conventi F
- Cooke HG
- Cooper-Sarkar AM
- Cordeiro Oudot Choi A
- Cormier F
- Corpe LD
- Corradi M
- Corriveau F
- Cortes-Gonzalez A
- Costa MJ
- Costanza F
- Costanzo D
- Cote BM
- Cowan G
- Cranmer K
- Cremonini D
- Crescioli F
- Cristinziani M
- Cristoforetti M
- Croft V
- Crosby JE
- Crosetti G
- Crépé-Renaudin S
- Cueto A
- Cuhadar Donszelmann T
- Cui H
- Cui Z
- Cunningham WR
- Curcio F
- Czodrowski P
- Czurylo MM
- Da Cunha Sargedas De Sousa MJ
- Da Fonseca Pinto JV
- Da Via C
- Dabrowski W
- Dado T
- Dahbi S
- Dai T
- Dal Santo D
- Dallapiccola C
- Dam M
- Damp J
- Dandoy JR
- Daneri MF
- Danninger M
- Dao V
- Darbo G
- Darmora S
- Das SJ
- David C
- Davidek T
- Davis-Purcell B
- Dawson I
- Day-hall HA
- De Asmundis R
- De Biase N
- De Castro S
- De Groot N
- de Jong P
- De la Torre H
- De Maria A
- De Salvo A
- De Sanctis U
- De Santo A
- De Vivie De Regie JB
- De K
- Dedovich DV
- Degens J
- Deiana AM
- Del Corso F
- Del Peso J
- Del Rio F
- Deliot F
- Delitzsch CM
- Della Pietra M
- Della Volpe D
- Dell’Acqua A
- Dell’Asta L
- Delmastro M
- Delsart PA
- Demers S
- Demichev M
- Denisov SP
- Derendarz D
- Derue F
- Dervan P
- Desch K
- Deutsch C
- Di Bello FA
- Di Ciaccio A
- Di Ciaccio L
- Di Domenico A
- Di Donato C
- Di Girolamo A
- Di Gregorio G
- Di Luca A
- Di Micco B
- Di Nardo R
- Diaconu C
- Diamantopoulou M
- Dias Do Vale T
- Dias FA
- Diaz Capriles FG
- Diaz MA
- Didenko M
- Diehl EB
- Diehl L
- Diez Pardos C
- Dimitriadi C
- Dimitrievska A
- Dingfelder J
- Dinu I-M
- Dittmeier SJ
- Dittus F
- Djama F
- Djobava T
- Djuvsland JI
- Doglioni C
- Dohnalova A
- Dolejsi J
- Dolezal Z
- Dona KM
- Donadelli M
- Dong B
- Donini J
- Dopke J
- Doria A
- Dos Santos Fernandes N
- Dougan P
- Dova MT
- Doyle AT
- Draguet MA
- Dreyer E
- Drivas-koulouris I
- Drobac AS
- Drozdova M
- du Pree TA
- Du D
- Dubinin F
- Dubovsky M
- Duchovni E
- Duckeck G
- Ducu OA
- Duda D
- Dudarev A
- Duden ER
- Duflot L
- Dumitriu AE
- Dunford M
- Dungs S
- Dunne K
- Duperrin A
- Duran Yildiz H
- Durglishvili A
- Dwyer BL
- Dyckes GI
- Dyndal M
- Dysch S
- Dziedzic BS
- Díez Cornell S
- Dührssen M
- Dülsen C
- Düren M
- D’amen G
- D’Amico V
- D’Auria S
- D’Eramo L
- D’Onofrio A
- D’Onofrio M
- D’uffizi M
- Earnshaw ZO
- Eberwein GH
- Eckerova B
- Eggebrecht S
- Egidio Purcino De Souza E
- Ehrke LF
- Eigen G
- Einsweiler K
- Ekelof T
- Ekman PA
- El Farkh S
- El Ghazali Y
- El Jarrari H
- El Moussaouy A
- Ellajosyula V
- Ellert M
- Ellinghaus F
- Elliot AA
- Ellis N
- Elmsheuser J
- Elsing M
- Emeliyanov D
- Enari Y
- Ene I
- Epari S
- Erdmann J
- Erland PA
- Errenst M
- Escalier M
- Escobar C
- Etzion E
- Evans G
- Evans H
- Evans LS
- Evans MO
- Ezhilov A
- Ezzarqtouni S
- Fabbri F
- Fabbri L
- Facini G
- Fadeyev V
- Fakhrutdinov RM
- Falciano S
- Falda Ulhoa Coelho LF
- Falke PJ
- Faltova J
- Fan C
- Fan Y
- Fang Y
- Fanti M
- Faraj M
- Farazpay Z
- Farbin A
- Farilla A
- Farooque T
- Farrington SM
- Fassi F
- Fassouliotis D
- Faucci Giannelli M
- Fawcett WJ
- Fayard L
- Federic P
- Federicova P
- Fedin OL
- Fedotov G
- Feickert M
- Feligioni L
- Fellers DE
- Feng C
- Feng M
- Feng Z
- Fenton MJ
- Fenyuk AB
- Ferencz L
- Ferguson RAM
- Fernandez Luengo SI
- Fernoux MJV
- Ferrando J
- Ferrari A
- Ferrari P
- Ferrari R
- Ferrere D
- Ferretti C
- Fiedler F
- Filipčič A
- Filmer EK
- Filthaut F
- Fiolhais MCN
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- Fleck I
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- Zhemchugov A
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- Zhulanov V
- Zimine NI
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- Zwalinski L
- Åkesson TPA
- Öncel ÖO
- Ženiš T
- Živković L
- Publication venue
- Springer Nature
- Publication date
- 01/04/2024
- Field of study
This paper presents the measurement of fducial and diferential cross sections for both the inclusive and electroweak production of a same-sign W-boson pair in association with two jets (W±W±jj) using 139 fb−1 of proton-proton collision data recorded at a centre-of-mass energy of √s = 13 TeV by the ATLAS detector at the Large Hadron Collider. The analysis is performed by selecting two same-charge leptons, electron or muon, and at least two jets with large invariant mass and a large rapidity diference. The measured fducial cross sections for electroweak and inclusive W±W±jj production are 2.92 ± 0.22 (stat.) ± 0.19 (syst.)fb and 3.38±0.22 (stat.)±0.19 (syst.)fb, respectively, in agreement with Standard Model predictions. The measurements are used to constrain anomalous quartic gauge couplings by extracting 95% confdence level intervals on dimension-8 operators. A search for doubly charged Higgs bosons H±± that are produced in vector-boson fusion processes and decay into a same-sign W boson pair is performed. The largest deviation from the Standard Model occurs for an H±± mass near 450 GeV, with a global signifcance of 2.5 standard deviations
Combination of searches for heavy spin-1 resonances using 139 fb−1 of proton-proton collision data at s = 13 TeV with the ATLAS detector
- Author
- Aad G
- Aakvaag E
- Abbott B
- Abeling K
- Abicht NJ
- Abidi SH
- Aboelela M
- Aboulhorma A
- Abramowicz H
- Abreu H
- Abulaiti Y
- Acharya BS
- Ackermann A
- Adam Bourdarios C
- Adamczyk L
- Addepalli SV
- Addison MJ
- Adelman J
- Adiguzel A
- Adye T
- Affolder AA
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- Agaras MN
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- Aggarwal A
- Agheorghiesei C
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- Akolkar NN
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- Aleksa M
- Aleksandrov IN
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- Zeng H
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- Zenger DT
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- Zerradi S
- Zerwas D
- Zhai M
- Zhang DF
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- Zhang X
- Zhang X
- Zhang Y
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- Zhang Y
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- Zhang Z
- Zhao H
- Zhao T
- Zhao Y
- Zhao Z
- Zhao Z
- Zhemchugov A
- Zheng J
- Zheng K
- Zheng X
- Zheng Z
- Zhong D
- Zhou B
- Zhou H
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- Zhou Y
- Zhu CG
- Zhu J
- Zhu Y
- Zhu Y
- Zhuang X
- Zhukov K
- Zimine NI
- Zinsser J
- Ziolkowski M
- Zoccoli A
- Zoch K
- Zorbas TG
- Zormpa O
- Zou W
- Zwalinski L
- Åkesson TPA
- Öncel ÖO
- Ženiš T
- Živković L
- Publication venue
- Springer Nature
- Publication date
- 19/04/2024
- Field of study
A combination of searches for new heavy spin-1 resonances decaying into different pairings of W, Z, or Higgs bosons, as well as directly into leptons or quarks, is presented. The data sample used corresponds to 139 fb−1 of proton-proton collisions at
= 13 TeV collected during 2015–2018 with the ATLAS detector at the CERN Large Hadron Collider. Analyses selecting quark pairs (qq, bb,
, and tb) or third-generation leptons (τν and ττ) are included in this kind of combination for the first time. A simplified model predicting a spin-1 heavy vector-boson triplet is used. Cross-section limits are set at the 95% confidence level and are compared with predictions for the benchmark model. These limits are also expressed in terms of constraints on couplings of the heavy vector-boson triplet to quarks, leptons, and the Higgs boson. The complementarity of the various analyses increases the sensitivity to new physics, and the resulting constraints are stronger than those from any individual analysis considered. The data exclude a heavy vector-boson triplet with mass below 5.8 TeV in a weakly coupled scenario, below 4.4 TeV in a strongly coupled scenario, and up to 1.5 TeV in the case of production via vector-boson fusion
Thrombocytopenia in malaria: who cares?
- Author
- Aggarwal A
- Alecrim MGC
- Alexandre MA
- Almeida AJ
- Andrade BB
- Anstey NM
- Araujo CF
- Araújo Filho JA
- Bashawri LA
- Batista D
- Beale PJ
- Becchi C
- Bhatia V
- Blajchman MA
- Brown KM
- Campos FM
- Carvalho BO
- Casals-Pascual C
- Chagas C
- Chotivanich K
- Combes V
- Conte R
- Cummins D
- D'Acremont V
- Daniel-Ribeiro CT
- Daniel-Ribeiro CT
- Davidson RA
- de Paula LV
- del Portillo HA
- Dennis LH
- Doi T
- Echeverri M
- Engwerda CR
- Erel O
- Erel O
- Essien EM
- Fajardo LF
- Franchini M
- George P
- Gerardin P
- Gonzalez B
- Gonzalez LM
- Grau GE
- Greenwood BM
- Grynberg P
- Harish R
- Helena Cristina Cardoso Coelho
- Hill GJ
- Hoffman SL
- Holland BH
- Honda S
- Huang DB
- Inyang AL
- Jadhav UM
- Jaff MS
- João Barberino Santos
- Kakar A
- Karpatkin S
- Karpatkin S
- Katira B
- Kaur D
- Kelton JG
- Khan FY
- Kochar DK
- Kochar DK
- Koltas IS
- Komoda M
- Kortepeter M
- Kreil A
- Kumar A
- Lacerda MV
- Lacerda MV
- Lacerda MVG
- Lacerda MVG
- Lacerda MVG
- Lacerda MVG
- Ladhani S
- Lathia TB
- Lee SH
- Li Z
- Llanos C
- Lou J
- Maina RN
- Makkar RP
- Marcus Vinícius Guimarães Lacerda
- Maria Paula Gomes Mourão
- Marques HO
- Marques HO
- Martin-Jaular L
- Mast Q
- Mast Q
- Menendez C
- Mohanty D
- Mohanty D
- Mohanty S
- Mohapatra MK
- Monath TP
- Moulin F
- Mourão MP
- Mourão MPG
- Muniz-Junqueira MI
- Muniz-Junqueira MI
- Murthy GL
- Nardi MA
- Naveira JB
- Nicodemo AC
- Noronha EF
- Oh MD
- Ohtaka M
- Pain A
- Panasiuk A
- Pankoui Mfonkeu JB
- Parakh A
- Park CHL
- Park JW
- Patel U
- Peck-Radosavljevic M
- Piguet PF
- Poespoprodjo JR
- Prasad R
- Rasheed A
- Rebulla P
- Rebulla P
- Rifakis PM
- Rios-Orrego A
- Robinson P
- Rodriguez-Morales AJ
- Rodriguez-Morales AJ
- Rogier C
- Samuel H
- Santana Filho FS
- Santos MC
- Santos PD
- Scaradavou A
- Schlossberg HR
- Shaikh QH
- Shear HL
- Silva IBA
- Silva SBR
- Silva SL
- Skudowitz RB
- Song JY
- Srichaikul T
- Srichaikul T
- Srivastava S
- Suarez-Mutis MC
- Sun G
- Takaki K
- Tan SO
- Taylor WR
- Terrazzano G
- Thapa R
- Tjitra E
- Touze JE
- Tribulatti MV
- Tyagi P
- Urban BC
- van der Heyde HC
- Victoria MB
- Vij AS
- Warrell DA
- Wassmer SC
- Wassmer SC
- Wassmer SC
- Watier H
- Wickramasinghe SN
- Yamaguchi S
- Publication venue
- 'FapUNIFESP (SciELO)'
- Publication date
- Field of study
A saturated map of common genetic variants associated with human height
- Author
- Abecasis GR
- Adair LS
- Adams HHH
- Aguilar-Salinas CA
- Ahluwalia TS
- Akiyama M
- Al-Mulla F
- Allison MA
- Alvarez M
- Andersen MK
- Ani A
- Appadurai V
- Arbeeva L
- Arias JD
- Arnett DK
- Asselbergs FW
- Assimes TL
- Asvold BO
- Attia J
- Auton A
- Banas B
- Bandinelli S
- Bartell E
- Bennett DA
- Bergler T
- Berndt SI
- Bharadwaj D
- Bhaskar S
- Bielak LF
- Biino G
- Bisgaard H
- Boehnke M
- Boerwinkle E
- Boger CA
- Bollepalli S
- Bonnelykke K
- Bonnycastle LL
- Boomsma DI
- Borglum AD
- Borja JB
- Bork-Jensen J
- Bouchard C
- Bowden DW
- Bradfield JP
- Bradford Y
- Brandslund I
- Braund PS
- Brody JA
- Brumpton B
- Burgdorf KS
- Buring JE
- Cade BE
- Cai H
- Cai QY
- Campbell A
- Canadas-Garre M
- Catamo E
- Caulfield MJ
- Chai JF
- Chai XR
- Chambers JC
- Chandak GR
- Chang LC
- Chang YC
- Chanock SJ
- Chasman DI
- Chaturvedi N
- Chen CH
- Chen SH
- Chen YDI
- Chen ZM
- Cheng CY
- Chesi A
- Cho YS
- Choi SH
- Christofidou P
- Christophersen IE
- Chung RH
- Ciullo M
- Cocca M
- Cole JW
- Collins FS
- Concas MP
- Cooper RS
- Couture C
- Cruz M
- Cucca F
- Cuellar-Partida G
- Cupples LA
- Cutler MJ
- Damrauer SM
- Danning R
- Dantoft TM
- Daw EW
- de Borst GJ
- de Groot LCPGM
- De Jager PL
- de Kleijn DPV
- de Silva HJ
- Dedoussis GV
- Degenhard F
- Delgado GE
- Delitala A
- Deloukas P
- Demirkan A
- den Hollander AI
- Deng X
- Devineni P
- Dietl A
- Dimitriou M
- Dimitrov L
- Dorajoo R
- Du SF
- Easton DF
- Ekici AB
- Elders PJM
- Eliasen AU
- Eliassen AH
- Ellinor PT
- Elmstahl S
- Engmann JE
- Erdmann J
- Esko T
- Evans MK
- Fairhurst-Hunter Z
- Farmaki AE
- Fatkin D
- Faul JD
- Feenstra B
- Feitosa MF
- Fernandez-Lopez JC
- Ferreira T
- Ferrucci L
- Ford I
- Forer L
- Fornage M
- Francescatto M
- Franke A
- Franks PW
- Frayling TM
- Freedman BI
- Freitag-Wolf S
- Fuchsberger C
- Galesloot TE
- Gao Y
- Gao ZS
- Gasparini P
- Geller F
- Giannakopoulou O
- Gieger C
- Girotto G
- Giulianini F
- Gjesing AP
- Goddard ME
- Goel A
- Golightly YM
- Gonzalez-Villalpando C
- Gordon SD
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- Gorski M
- Graff M
- Graham SE
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- Grant SFA
- Grarup N
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- Grove J
- Gudnason V
- Guo XQ
- Gustafsson S
- Haessler J
- Haiman C
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- Hartman CA
- Hattersley AT
- Havulinna AS
- Haworth SJ
- Hayward C
- He J
- Heard-Costa N
- Hebbar P
- Heckbert SR
- Heid IM
- Heng CK
- Hengstenberg C
- Hewitt AW
- Highland HH
- Hindy G
- Hirschhorn JN
- Hishigaki H
- Ho YLA
- Hofer E
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- Horn K
- Hornsby WE
- Hottenga JJ
- Hoyng CB
- Huang HY
- Huang J
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- Huang W
- Huerta-Chagoya A
- Huffman JE
- Hung YJ
- Hunt SC
- Huo SF
- Hveem K
- Hwang MY
- Hypponen E
- Iacono WG
- Ichihara S
- Iha H
- Ikeda DD
- Ikram MA
- Isasi CR
- Isono M
- Jackson AU
- Jackson RD
- Jager S
- Jansen IE
- Jarvelin MR
- Ji YJ
- Jiang YX
- Jin ZB
- Jockel KH
- Johansson I
- Jonas JB
- Jonsson A
- Jorgensen T
- Joshi PK
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- Jukema JW
- Justice AE
- Kahonen M
- Kalafati IP
- Kamatani Y
- Kanai M
- Kang KD
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- Kaprio J
- Karaderi T
- Kardia SLR
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- Karpe F
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- Katsuya T
- Kawaguchi T
- Kee F
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- Kentistou KA
- Kessler T
- Khera AV
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- Kiemeney LALM
- Kim BJ
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- Kirchhof P
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- Kleber ME
- Knol MJ
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- Schulze MB
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- Scott RJ
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- Shin JH
- Shiroma EJ
- Shoemaker MB
- Shu XO
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- Simonsick EM
- Sims M
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- Singleton AB
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- Sitlani CM
- Slieker RKC
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- Smith JG
- Smyth LJ
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- Southam LE
- Spector TD
- Spracklen CN
- Stampfer MJ
- Stark KJ
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- Steinthorsdottir V
- Strachan DP
- Sun L
- Sun YV
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- Takeuchi F
- Tallapragada D
- Tang H
- Tardif JC
- Taylor KD
- Tayo BO
- Tcheandjieu C
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- Teumer A
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- Thorleifsson G
- Thorsteinsdottir U
- Timmers PRHJ
- Timpson NJ
- Tonjes A
- Tremblay A
- Trompet S
- Tuomi T
- Tuomilehto J
- Turman C
- Tusie-Luna MT
- Uitterlinden AG
- Vaccargiu S
- van Dam RM
- van der Harst P
- van der Laan SW
- van der Most PJ
- Van der Velde N
- van Duijn CM
- van Klinken JB
- van Schoor NM
- van Setten J
- Vedantam S
- Verma SS
- Verweij N
- Veturi Y
- Vidal PM
- Visscher PM
- Vitart V
- Volker U
- Vollenweider P
- Volzke H
- Vrieze S
- Wacher-Rodarte NH
- Walker M
- Walters RG
- Wang CA
- Wang CL
- Wang LH
- Wang YX
- Wang Z
- Wareham NJ
- Warren HR
- Watanabe RM
- Watkins H
- Wei WB
- Weir DR
- Werge TM
- Wickremasinghe AR
- Widen E
- Wielscher M
- Wiggins KL
- Wilkens LR
- Willemsen G
- Willer CJ
- Willett WC
- Wilson JF
- Winkler TW
- Winsvold BS
- Wong A
- Wong TY
- Woo JT
- Wood AR
- Wright AF
- Wu JY
- Wu Y
- Wuttke M
- Xia R
- Xie T
- Xu HC
- Yajnik CS
- Yamamoto K
- Yang J
- Yang JY
- Yao J
- Yengo L
- Yin XY
- Yokota M
- Young H
- Young KL
- Yousri NA
- Yu L
- Yuan JM
- Zeggini E
- Zemel BS
- Zeng LY
- Zhang WH
- Zhang XY
- Zhao JH
- Zhao W
- Zheng W
- Zhou W
- Zhu XF
- Zimmermann ME
- Zmuda JM
- Zoledziewska M
- Zonderman AB
- Zwart JA
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 29/11/2022
- Field of study
Common single-nucleotide polymorphisms (SNPs) are predicted to collectively explain 40-50% of phenotypic variation in human height, but identifying the specific variants and associated regions requires huge sample sizes(1). Here, using data from a genome-wide association study of 5.4 million individuals of diverse ancestries, we show that 12,111 independent SNPs that are significantly associated with height account for nearly all of the common SNP-based heritability. These SNPs are clustered within 7,209 non-overlapping genomic segments with a mean size of around 90 kb, covering about 21% of the genome. The density of independent associations varies across the genome and the regions of increased density are enriched for biologically relevant genes. In out-of-sample estimation and prediction, the 12,111 SNPs (or all SNPs in the HapMap 3 panel(2)) account for 40% (45%) of phenotypic variance in populations of European ancestry but only around 10-20% (14-24%) in populations of other ancestries. Effect sizes, associated regions and gene prioritization are similar across ancestries, indicating that reduced prediction accuracy is likely to be explained by linkage disequilibrium and differences in allele frequency within associated regions. Finally, we show that the relevant biological pathways are detectable with smaller sample sizes than are needed to implicate causal genes and variants. Overall, this study provides a comprehensive map of specific genomic regions that contain the vast majority of common height-associated variants. Although this map is saturated for populations of European ancestry, further research is needed to achieve equivalent saturation in other ancestries.A large genome-wide association study of more than 5 million individuals reveals that 12,111 single-nucleotide polymorphisms account for nearly all the heritability of height attributable to common genetic variants
A saturated map of common genetic variants associated with human height.
- Author
- 't Hart LM
- 23andMe Research Team
- Abecasis GR
- Adair LS
- Adams HHH
- Aguilar-Salinas CA
- Ahluwalia TS
- Akiyama M
- Al-Mulla F
- Allison MA
- Alvarez M
- Andersen MK
- Ani A
- Appadurai V
- Arbeeva L
- Arias JD
- Arnett DK
- Asselbergs FW
- Assimes TL
- Attia J
- Auton A
- Banas B
- Bandinelli S
- Bartell E
- Bennett DA
- Bergler T
- Berndt SI
- Bharadwaj D
- Bhaskar S
- Bielak LF
- Biino G
- Bin Wei W
- Bisgaard H
- Boehnke M
- Boerwinkle E
- Bollepalli S
- Bonnycastle LL
- Boomsma DI
- Borja JB
- Bork-Jensen J
- Bouchard C
- Bowden DW
- Bradfield JP
- Bradford Y
- Brandslund I
- Braund PS
- Brody JA
- Brumpton B
- Burgdorf KS
- Buring JE
- Böger CA
- Bønnelykke K
- Børglum AD
- Cade BE
- Cai H
- Cai Q
- Campbell A
- Catamo E
- Caulfield MJ
- Cañadas-Garre M
- Chai J-F
- Chai X
- Chambers JC
- Chandak GR
- Chang L-C
- Chang Y-C
- Chanock SJ
- Chasman DI
- Chaturvedi N
- Chen C-H
- Chen S-H
- Chen Y-DI
- Chen Z
- Cheng C-Y
- Chesi A
- Cho YS
- Choi SH
- Christofidou P
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- Zimmermann ME
- Zmuda JM
- Zoledziewska M
- Zonderman AB
- Zwart J-A
- Åsvold BO
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 12/10/2022
- Field of study
Common single-nucleotide polymorphisms (SNPs) are predicted to collectively explain 40-50% of phenotypic variation in human height, but identifying the specific variants and associated regions requires huge sample sizes1. Here, using data from a genome-wide association study of 5.4 million individuals of diverse ancestries, we show that 12,111 independent SNPs that are significantly associated with height account for nearly all of the common SNP-based heritability. These SNPs are clustered within 7,209 non-overlapping genomic segments with a mean size of around 90 kb, covering about 21% of the genome. The density of independent associations varies across the genome and the regions of increased density are enriched for biologically relevant genes. In out-of-sample estimation and prediction, the 12,111 SNPs (or all SNPs in the HapMap 3 panel2) account for 40% (45%) of phenotypic variance in populations of European ancestry but only around 10-20% (14-24%) in populations of other ancestries. Effect sizes, associated regions and gene prioritization are similar across ancestries, indicating that reduced prediction accuracy is likely to be explained by linkage disequilibrium and differences in allele frequency within associated regions. Finally, we show that the relevant biological pathways are detectable with smaller sample sizes than are needed to implicate causal genes and variants. Overall, this study provides a comprehensive map of specific genomic regions that contain the vast majority of common height-associated variants. Although this map is saturated for populations of European ancestry, further research is needed to achieve equivalent saturation in other ancestries
Combined measurement of the Higgs boson mass from the H → γγ and H → ZZ∗ → 4ℓ decay channels with the ATLAS detector using √s = 7, 8, and 13 TeV pp collision data
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- Abbott B
- Abeling K
- Abicht NJ
- Abidi SH
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- Acharya BS
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- Adamczyk L
- Addepalli SV
- Addison MJ
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- Flores Sanz De Acedo L
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- Zenger DT
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- Zhang X
- Zhang X
- Zhang Y
- Zhang Y
- Zhang Y
- Zhang Z
- Zhang Z
- Zhao H
- Zhao P
- Zhao T
- Zhao Y
- Zhao Z
- Zhemchugov A
- Zheng J
- Zheng K
- Zheng X
- Zheng Z
- Zhong D
- Zhou B
- Zhou H
- Zhou N
- Zhou Y
- Zhu CG
- Zhu J
- Zhu Y
- Zhu Y
- Zhuang X
- Zhukov K
- Zhulanov V
- Zimine NI
- Zinsser J
- Ziolkowski M
- Zoccoli A
- Zoch K
- Zorbas TG
- Zormpa O
- Zou W
- Zwalinski L
- Åkesson TPA
- Öncel ÖO
- Ženiš T
- Živković L
- Publication venue
- American Physical Society
- Publication date
- 01/01/2023
- Field of study
A measurement of the mass of the Higgs boson combining the
H
→
Z
Z
∗
→
4
ℓ
and
H
→
γ
γ
decay channels is presented. The result is based on
140
fb
−
1
of proton-proton collision data collected by the ATLAS detector during LHC run 2 at a center-of-mass energy of 13 TeV combined with the run 1 ATLAS mass measurement, performed at center-of-mass energies of 7 and 8 TeV, yielding a Higgs boson mass of
125.11
±
0.09
(
stat
)
±
0.06
(
syst
)
=
125.11
±
0.11
GeV
. This corresponds to a 0.09% precision achieved on this fundamental parameter of the Standard Model of particle physics
Search for dark photons in rare Z boson decays with the ATLAS detector
- Author
- Aad G
- Abbott B
- Abeling K
- Abicht NJ
- Abidi SH
- Aboulhorma A
- Abramowicz H
- Abreu H
- Abulaiti Y
- Abusleme Hoffman AC
- Acharya BS
- Adam Bourdarios C
- Adamczyk L
- Adamek L
- Addepalli SV
- Addison MJ
- Adelman J
- Adiguzel A
- Adye T
- Affolder AA
- Afik Y
- Agaras MN
- Agarwala J
- Aggarwal A
- Agheorghiesei C
- Ahmad A
- Ahmadov F
- Ahmed WS
- Ahuja S
- Ai X
- Aielli G
- Ait Tamlihat M
- Aitbenchikh B
- Aizenberg I
- Akbiyik M
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- Zenger DT
- Zenin O
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- Zerwas D
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- Zhang Z
- Zhang Z
- Zhao H
- Zhao P
- Zhao T
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- Zhemchugov A
- Zheng K
- Zheng X
- Zheng Z
- Zhong D
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- Zhu Y
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- Zhukov K
- Zhulanov V
- Zimine NI
- Zinsser J
- Ziolkowski M
- Zoccoli A
- Zoch K
- Zorbas TG
- Zormpa O
- Zou W
- Zwalinski L
- Åkesson TPA
- Öncel ÖO
- Ženiš T
- Živković L
- Publication venue
- American Physical Society
- Publication date
- 01/01/2023
- Field of study
A search for events with a dark photon produced in association with a dark Higgs boson via rare decays of the standard model
Z
boson is presented, using
139
fb
−
1
of
√
s
=
13
TeV
proton-proton collision data recorded by the ATLAS detector at the Large Hadron Collider. The dark boson decays into a pair of dark photons, and at least two of the three dark photons must each decay into a pair of electrons or muons, resulting in at least two same-flavor opposite-charge lepton pairs in the final state. The data are found to be consistent with the background prediction, and upper limits are set on the dark photon’s coupling to the dark Higgs boson times the kinetic mixing between the standard model photon and the dark photon,
α
D
ϵ
2
, in the dark photon mass range of [5, 40] GeV except for the
Υ
mass window [8.8, 11.1] GeV. This search explores new parameter space not previously excluded by other experiments
- …