Mode Coupling in Quantized High Quality Films

The effect of coupling of quantized modes on transport and localization in ultrathin films with quantum size effect (QSE) is discussed. The emphasis is on comparison of films with Gaussian, exponential, and power-law long-range behavior of the correlation function of surface, thickness, or bulk fluctuations. For small-size inhomogeneities, the mode coupling is the same for inhomogeneities of all types and the transport coefficients behave in the same way. The mode coupling becomes extremely sensitive to the correlators for large-size inhomogeneities leading to the drastically distinct behavior of the transport coefficients. In high-quality films there is a noticeable difference between the QSE patterns for films with bulk and surface inhomogeneities which explains why the recently predicted new type of QSE with large oscillations of the transport coefficients can be observed mostly in films with surface-driven relaxation. In such films with surface-dominated scattering the higher modes contribute to the transport only as a result of opening of the corresponding mode coupling channels and appear one by one. Mode coupling also explains a much higher transport contribution from the higher modes than it is commonly believed. Possible correlations between the inhomogeneities from the opposite walls provide, because of their oscillating response to the mode quantum numbers, a unique insight into the mode coupling. The presence of inhomogeneities of several sizes leads not to a mechanical mixture of QSE patterns, but to the overall shifting and smoothing of the oscillations. The results can lead to new, non-destructive ways of analysis of the buried interfaces and to study of inhomogeneities on the scales which are inaccessible for scanning techniques

Low-Temperature Spin Diffusion in a Spin-Polarized Fermi Gas

We present a finite temperature calculation of the transverse spin-diffusion coefficient, $D_\bot$, in a dilute degenerate Fermi gas in the presence of a small external magnetic field, $H$. While the longitudinal diffusion coefficient displays the conventional low-temperature Fermi-liquid behavior, $D_\parallel \propto T^{-2}$, the corresponding results for $D_\bot$ show three separate regimes: (a) $D_\bot \sim H^{-2}$ for $T \ll H$; (b) $D_\bot \sim T^{-2}$, $D_\bot /D_\parallel \neq 1$ for $T \gg H$ and large spin-rotation parameter $\xi \gg 1$, and (c) $D_\bot = D_\parallel \propto T^{-2}$ for $T \gg H$ and $\xi \ll 1$. Our results are qualitatively consistent with the available experimental data in weakly spin-polarized $^3{\rm He}$ and $^3{\rm He} - ^4{\rm He}$ mixtures.Comment: 13 pages, REVTEX, 3 figures available upon request, RU-94-4

Transverse Dynamics and Relaxation in Spin-Polarized or Two-Level Fermi Systems

A microscopic theory is proposed for transverse dynamics and zero-temperature attenuation in polarized Fermi liquids. The transport equations are a set of two coupled equations in two ‘‘partial transverse densities,’’ which do not reduce to a single equation in a mixed component of a single-particle distribution. The effective interaction is linked to an irreducible vertex by an integral equation, and cannot be given as a limit of a full vertex. A framework for a generalized nonlocal Landau theory is established. The spectrum of attenuating spin waves is calculated at arbitrary polarizations and densities

Anomalous Spin Dynamics and Relaxation in Fermi Liquids

We explain the anomalous temperature dependence of spin diffusion in liquid 3↑ and 3--4He mixtures. The anomaly is an experimental manifestation of a unique zero-temperature attenuation in the Fermi liquid theory. We extended our microscopic theory of spin dynamics in spin-polarized Fermi liquids to finite temperatures. The zero-temperature attenuation changes the behavior of the spectrum near the singular point. The data indicate that the superfluid transition temperature for 3He in 3--4He mixtures is much lower than the current estimates

A20 deficiency sensitizes pancreatic beta cells to cytokine-induced apoptosis in vitro but does not influence type 1 diabetes development in vivo

SCOPUS: ar.jinfo:eu-repo/semantics/publishe

Transverse spin dynamics in a spin-polarized Fermi liquid

The linear equations for transverse spin dynamics in weakly polarised degenerate Fermi liquid with arbitrary relationship between temperature and polarization are derived from Landau-Silin phenomenological kinetic equation with general form of two-particle collision integral. The temperature and polarization dependence of the spin current relaxation time is established. It is found in particular that at finite polarization transverse spin wave damping has a finite value at T=0. The analogy between temperature dependences of spin waves attenuation and ultrasound absorption in degenerate Fermi liquid at arbitrary temperature is presented. We also discuss spin-polarized Fermi liquid in the general context of the Fermi-liquid theory and compare it with "Fermi liquid" with spontaneous magnetization.Comment: 10 page

Relaxation of Spin Polarized 3^3He in Mixtures of 3^3He and 4^4He Below the 4^4He Lambda Point

We report the first study of the depolarization behavior of spin polarized 3He in a mixture of 3He-4He at a temperature below the 4He Lambda point in a deuterated TetraPhenyl Butadiene-doped deuterated PolyStyrene (dTPB-dPS) coated acrylic cell. In our experiment the measured 3He relaxation time is due to the convolution of the 3He longitudinal relaxation time, T1, and the diffusion time constant of 3He in superfluid 4He since depolarization takes place on the walls. We have obtained a 3He relaxation time ~3000 seconds at a temperature around 1.9K. We have shown that it's possible to achieve values of wall depolarization probability on the order of (1-2)x10^-7 for polarized 3He in the superfluid 4He from a dTPB-dPS coated acrylic surface.Comment: The Model used to interpret the data has been change

Internal state conversion in ultracold gases

We consider an ultracold gas of (non-condensed) bosons or fermions with two internal states, and study the effect of a gradient of the transition frequency between these states. When a $\pi/2$ RF pulse is applied to the sample, exchange effects during collisions transfer the atoms into internal states which depend on the direction of their velocity. This results, after a short time, in a spatial separation between the two states. A kinetic equation is solved analytically and numerically; the results agree well with the recent observations of Lewandowski et al.Comment: Accepted version, to appear in PR

Dysfunctional autophagy following exposure to pro-inflammatory cytokines contributes to pancreatic β-cell apoptosis.

Type 1 diabetes (T1D) results from β-cell destruction due to concerted action of both innate and adaptive immune responses. Pro-inflammatory cytokines, such as interleukin-1β and interferon-γ, secreted by the immune cells invading islets of Langerhans, contribute to pancreatic β-cell death in T1D. Cytokine-induced endoplasmic reticulum (ER) stress plays a central role in β-cell demise. ER stress can modulate autophagic response; however, no study addressed the regulation of autophagy during the pathophysiology of T1D. In this study, we document that cytokines activate the AMPK-ULK-1 pathway while inhibiting mTORC1, which stimulates autophagy activity in an ER stress-dependent manner. On the other hand, time-course analysis of LC3-II accumulation in autophagosomes revealed that cytokines block the autophagy flux in an ER stress independent manner, leading to the formation of large dysfunctional autophagosomes and worsening of ER stress. Cytokines rapidly impair lysosome function, leading to lysosome membrane permeabilization, Cathepsin B leakage and lysosomal cell death. Blocking cathepsin activity partially protects against cytokine-induced or torin1-induced apoptosis, whereas blocking autophagy aggravates cytokine-induced CHOP overexpression and β-cell apoptosis. In conclusion, cytokines stimulate the early steps of autophagy while blocking the autophagic flux, which aggravate ER stress and trigger lysosomal cell death. Restoration of autophagy/lysosomal function may represent a novel strategy to improve β-cell resistance in the context of T1D