76 research outputs found

    UD_Japanese-CEJC: Dependency Relation Annotation on Corpus of Everyday Japanese Conversation

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    Conference name: the 24th Meeting of the Special Interest Group on Discourse and Dialogue, Conference place: Prague, Czechia, Session period: 2023/09/11-15, Organizer: Association for Computational Linguisticsapplication/pdfNational Institute for Japanese Language and LinguisticsTohoku UniversityMegagon Labs, Tokyo, Recruit Co., LtdNational Institute for Japanese Language and LinguisticsIn this study, we have developed Universal Dependencies (UD) resources for spoken Japanese in the Corpus of Everyday Japanese Conversation (CEJC). The CEJC is a large corpus of spoken language that encompasses various everyday conversations in Japanese, and includes word delimitation and part-of-speech annotation. We have newly annotated Long Word Unit delimitation and Bunsetsu (Japanese phrase)-based dependencies, including Bunsetsu boundaries, for CEJC. The UD of Japanese resources was constructed in accordance with hand-maintained conversion rules from the CEJC with two types of word delimitation, part-of-speech tags and Bunsetsu-based syntactic dependency relations. Furthermore, we examined various issues pertaining to the construction of UD in the CEJC by comparing it with the written Japanese corpus and evaluating UD parsing accuracy.conference pape

    現代日本語書き言葉均衡コーパスのUniversal Dependencies

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    会議名: 言語資源活用ワークショップ2017, 開催地: 国立国語研究所, 会期: 2017年9月5日-6日, 主催: 国立国語研究所 コーパス開発センター自然言語処理の分野では多言語かつ言語横断的な言語研究が盛んに取り組まれている。その言語横断的な言語研究の取り組みとしてUniversal Dependencies(UD)がある。UDでは品詞や係り受け構造の標準・スキーマを定め,多言語のコーパスを提供している。本論文では,日本語コーパスである現代日本語書き言葉均衡コーパス(BCCWJ)をUDのスキーマへと変換したコーパスについて紹介をする。BCCWJでは日本語における文節単位の係り受け情報がすでに付与されている。この係り受け構造を基にしてUDへと変換するプログラムの開発を行った。しかし,文節単位はUDの単語単位には沿っていない。そのため,BCCWJで提供されている短単位と長単位というふたつの言語単位を単語の単位をして認定したコーパスを構築する。短単位と長単位についてUDのスキーマに当てはめた場合,どのような係り受け構造ができるのかを示す

    UD Japanese-BCCWJの構築と分析

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    National Institute for Japanese Language and LinguisticsNational Institute for Japanese Language and Linguistics会議名: 言語資源活用ワークショップ2018, 開催地: 国立国語研究所, 会期: 2018年9月4日-5日, 主催: 国立国語研究所 コーパス開発センター自然言語処理の分野では多言語かつ言語横断的な言語研究が盛んに取り組まれている。その言語横断的な言語研究の取り組みとしてUniversal Dependencies(UD)がある。本論文では、日本語のコーパスであるUD Japanese-BCCWJについて紹介をする。UD Japanese-BCCWJは現代日本語書き言葉均衡コーパス(BCCWJ)に付随する係り受け情報などを組み合わせて、UDへと変換、構築したBCCWJのUniversal Dependencieである。これは日本語のUDの中でも1980文章、57,256文、約126万単語を含む最大規模また複数のレジスターを内包したデータセットである。UD Japanese-BCCWJの特徴について説明する。またUD Japanese-BCCWJの構築手順について説明し、現状における問題点について議論する

    Ring Gap Structure around Class I Protostar WL 17

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    WL 17 is a Class I object and was considered to have a ring-hole structure. We analyzed the structure around WL 17 to investigate the detailed properties of WL 17. We used ALMA archival data, which have a higher angular resolution than previous observations. We investigated the WL 17 system with the 1.3 mm dust continuum and 12CO and C18O (J = 2-1) line emissions. The dust continuum emission showed a clear ring structure with inner and outer edges of ~11 and ~21 au, respectively. In addition, we detected an inner disk of < 5 au radius enclosing the central star within the ring, the first observation of this structure. Thus, WL 17 has a ring-gap structure, not a ring-hole structure. We did not detect any marked emission in either the gap or inner disk, indicating that there is no sign of a planet, circumplanetary disk, or binary companion. We identified the base of both blue-shifted and red-shifted outflows based on the 12CO emission, which is clearly associated with the disk around WL 17. The outflow mass ejection rate is ~3.6x10^-7 Msun yr-1 and the dynamical timescale is as short as ~ 10^4 yr. The C18O emission showed that an inhomogeneous infalling envelope, which can induce episodic mass accretion, is distributed in the region within ~1000 au from the central protostar. With these new findings, we can constrain the planet formation and dust growth scenarios in the accretion phase of star formation.Comment: 22 pages, 9 figures, Accepted for publication in the Astrophysical Journa

    Relatório de estágio em farmácia comunitária

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    Relatório de estágio realizado no âmbito do Mestrado Integrado em Ciências Farmacêuticas, apresentado à Faculdade de Farmácia da Universidade de Coimbr

    Word Delimitation Issues in UD Japanese

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    A different pathway in the endoplasmic reticulum stress-induced expression of human HRD1 and SEL1 genes

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    Human HRD1 and SEL1 are components of endoplasmic reticulum-associated degradation (ERAD), which is a retrograde transport mechanism from the ER to the cytosol for removing unfolded proteins. The expression of HRD1 and SEL1 was induced by ER stress-inducing agents and overexpression of both ER stress-responsive transcription factors, ATF6 and XBP1. Inhibition of IRE1 and ATF6 revealed that ER stress-induced HRD1 and SEL1 expressions are mediated by IRE1-XBP1- and ATF6-dependent pathways, respectively. These results suggest that the ER stress-induced ERAD gene expressions are mediated by different pathways, which are attributed to the differences in the promoter regions

    Endoplasmic Reticulum Stress and Parkinson’s Disease: The Role of HRD1 in Averting Apoptosis in Neurodegenerative Disease

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    Endoplasmic reticulum (ER) stress has been known to be involved in the pathogenesis of various diseases, particularly neurodegenerative disorders such as Parkinson’s disease (PD). We previously identified the human ubiquitin ligase HRD1 that is associated with protection against ER stress and its associated apoptosis. HRD1 promotes the ubiquitination and degradation of Parkin-associated endothelin receptor-like receptor (Pael-R), an ER stress inducer and causative factor of familial PD, thereby preventing Pael-R-induced neuronal cell death. Moreover, upregulation of HRD1 by the antiepileptic drug zonisamide suppresses 6-hydroxydopamine-induced neuronal cell death. We review recent progress in the studies on the mechanism of ER stress-induced neuronal death related to PD, particularly focusing on the involvement of HRD1 in the prevention of neuronal death as well as a potential therapeutic approach for PD based on the upregulation of HRD1
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