Multidisciplinary Study of Biological Parameters and Fatigue Evolution in Quay Crane Operators

AbstractIn intermodal terminals the handling of containers and the number of accidents still depends on a wide range of human errors due to fatigue despite the automation level reached nowadays. For this reason it is very important to increase knowledge about the factors affecting the propensity of operators to make errors, increasing the chance of accidents happening. The aim of this work is to propose a novel approach to assess fatigue and performance levels in quay crane operators as a function of physiological parameters and of the many varying boundary conditions encountered in daily work. During their work, quay crane operators have to deal with variable environmental conditions, such as task type, wind speed and direction, lighting conditions that reduce visibility that can require an exacting level of attention. In the trial eight operators have been examined in a session lasting four hours. All actual conditions are reproduced through a fully immersive quay crane simulator. The operator completes the assigned task (the same for each one) and can see through four wide monitors a high quality virtual reality view of the simulation. Most biological parameters are acquired using different devices including a Holter ECG monitor, electromyographic monitoring the four trunk muscles most involved in the test, eye tracker and seat-body pressure interface for both seat pan and backrest. Changes in physiological parameters have been monitored during the trial and interesting correlations with performance levels and boundary conditions have been found for each operator, in accordance with their age and skills. The present study can form the basis for further investigations aimed at developing a cost effective, reliable and robust system for monitoring increasing fatigue and for predicting the critical conditions that may result in an accident

A Measurement of the Branching Ratio of KL→e+e−γγK_L \to e^+e^-\gamma\gamma

We report on a study of the decay $K_L \to e^+e^-\gamma\gamma$ carried out as a part of the KTeV/E799 experiment at Fermilab. The 1997 data yielded a sample of 1543 events, including an expected background of $56 \pm 8$ events. An effective form factor was determined from the observed distribution of the $e^+e^-$ invariant mass. Using this form factor in the calculation of the detector acceptance, the branching ratio was measured to be ${\mathcal B}(K_L \to e^+ e^- \gamma \gamma, E^*_\gamma > 5 {MeV}) = (5.84 \pm 0.15 {\rm ~(stat)} \pm 0.32 {\rm ~(sys)})\times 10^{-7}$.Comment: 5 pages, 4 figure

A measurement of the transverse polarization of Lambda-hyperons produced in inelastic pN-reactions at 450 GeV proton energy

A study of the polarization of Λ hyperons produced in inelastic pN reactions induced by the 450 GeV proton beam from the CERN SPS has been performed with the NA48 detector. The Λ hyperons were detected at a fixed angle of 4.2 mrad in the momentum range from 50 GeV/c to 200 GeV/c. The polarization changes from −0.053 ± 0.034 to −0.298 ± 0.074 for a transverse momentum range of the Λ between 0.28 GeV/c and 0.86 GeV/c. The antiΛ polarization is consistent with zero

Neurodegeneration progresses despite complete elimination of clinical relapses in a mouse model of multiple sclerosis.

BACKGROUND: [corrected] Multiple Sclerosis has two clinical phases reflecting distinct but inter-related pathological processes: focal inflammation drives the relapse-remitting stage and neurodegeneration represents the principal substrate of secondary progression. In contrast to the increasing number of effective anti-inflammatory disease modifying treatments for relapse-remitting disease, the absence of therapies for progressive disease represents a major unmet clinical need. This raises the unanswered question of whether elimination of clinical relapses will prevent subsequent progression and if so how early in the disease course should treatment be initiated. Experimental autoimmune encephalomyelitis in the Biozzi ABH mouse recapitulates the clinical and pathological features of multiple sclerosis including relapse-remitting episodes with inflammatory mediated demyelination and progressive disability with neurodegeneration. To address the relationship between inflammation and neurodegeneration we used an auto-immune tolerance strategy to eliminate clinical relapses in EAE in a manner analogous to the clinical effect of disease modifying treatments. RESULTS: By arresting clinical relapses in EAE at two distinct stages, early and late disease, we demonstrate that halting immune driven demyelination even after the first major clinical event is insufficient to prevent long-term neurodegeneration and associated gliosis. Nonetheless, early intervention is partially neuroprotective, whereas later interventions are not. Furthermore early tolerisation is also associated with increased remyelination. CONCLUSIONS: These findings are consistent with both a partial uncoupling of inflammation and neurodegeneration and that the regenerative response of remyelination is negatively correlated with inflammation. These findings strongly support the need for early combinatorial treatment of immunomodulatory therapies and neuroprotective treatments to prevent long-term neurodegeneration in multiple sclerosis

Futur dels teixits intel·ligents

Els teixits intel·ligents del futur es desenvolupen amb nous materials i amb tecnologies innovadores, però també amb clau d'eficiència ambiental. Es revisen tendències de la indústria tèxtil relacionades, entre d'altres, amb: Teixits funcionals i materials amb propietats singulars. e-Textil, wearables i gestió 4.0. Automatització, robòtica i impressió 3D. Ecologia industrial, impacte ambiental i valorització energètic