8 research outputs found
JS-K, a glutathione/glutathione S-transferase-activated nitric oxide releasing prodrug inhibits androgen receptor and WNT-signaling in prostate cancer cells
Get PDFA reactive oxygen species activation mechanism contributes to JS-K-induced apoptosis in human bladder cancer cells
No full textJS-K, a nitric oxide pro-drug, regulates growth and apoptosis through the ubiquitin-proteasome pathway in prostate cancer cells
No full textProtein phosphatase 2A activation mechanism contributes to JS-K induced caspase-dependent apoptosis in human hepatocellular carcinoma cells
No full textEnhancing the effectiveness of androgen deprivation in prostate cancer by inducing Filamin A nuclear localization
No full textPCA3 long noncoding RNA modulates the expression of key cancer-related genes in LNCaP prostate cancer cells
No full text
