Why Quantum Theory is Possibly Wrong

Quantum theory is a tremendously successful physical theory, but nevertheless suffers from two serious problems: the measurement problem and the problem of interpretational underdetermination. The latter, however, is largely overlooked as a genuine problem of its own. Both problems concern the doctrine of realism, but pull, quite curiously, into opposite directions. The measurement problem can be captured such that due to scientific realism about quantum theory common sense anti-realism follows, while theory underdetermination usually counts as an argument against scientific realism. I will also consider the more refined distinctions of ontic and epistemic realism and demonstrate that quantum theory in its most viable interpretations conflicts with at least one of the various realism claims. A way out of the conundrum is to come to the bold conclusion that quantum theory is, possibly, wrong (in the realist sense)

Weyl’s gauge argument

The standard U(1) “gauge principle” or “gauge argument” produces an exact potential A=dλ and a vanishing field F=ddλ=0. Weyl has his own gauge argument, which is sketchy, archaic and hard to follow; but at least it produces an inexact potential A and a nonvanishing field F=dA≠0. I attempt a reconstruction

Representation of Quantum Field Theory by Elementary Quantum Information

In this paper is considered relativistic quantum field theory expressed by elementary units of quantum information as they are considered as fundamental entity of nature by Carl Friedrich von Weizsaecker. Through quantization of a Weyl spinor describing an elementary unit of quantum information and consisting of four real components one obtains four pairs of creation and annihilation operators acting in a tensor space of states containing many units of quantum information. There can be constructed position and momentum operators from the creation and annihilation operators and based on these operators the Poincare group can be represented in this abstract tensor space of quantum information. A general state in the tensor space can be mapped to a state in Minkowski space-time by using the position representation of the eigenstates of the occupation number operators which correspond to the eigenstates of the harmonic oscillator. This yields a description of relativistic quantum mechanics. Quantization of the coefficients of a general state in the tensor space leads to many particle theory and thus to quantum field theory.Comment: 7 page

Quantum gauge models without classical Higgs mechanism

We examine the status of massive gauge theories, such as those usually obtained by spontaneous symmetry breakdown, from the viewpoint of causal (Epstein-Glaser) renormalization. The BRS formulation of gauge invariance in this framework, starting from canonical quantization of massive (as well as massless) vector bosons as fundamental entities, and proceeding perturbatively, allows one to rederive the reductive group symmetry of interactions, the need for scalar fields in gauge theory, and the covariant derivative. Thus the presence of higgs particles is explained without recourse to a Higgs(-Englert-Brout-Guralnik-Hagen-Kibble) mechanism. Along the way, we dispel doubts about the compatibility of causal gauge invariance with grand unified theories.Comment: 20 pages in two-column EPJC format, shortened version accepted for publication. For more details, consult version

Association of mast cell-derived VEGF and proteases in dengue shock syndrome

Background: Recent in-vitro studies have suggested that mast cells are involved in Dengue virus infection. To clarify the role of mast cells in the development of clinical Dengue fever, we compared the plasma levels of several mast cell-derived mediators (vascular endothelial cell growth factor [VEGF], soluble VEGF receptors [sVEGFRs], tryptase, and chymase) and -related cytokines (IL-4, -9, and -17) between patients with differing severity of Dengue fever and healthy controls. Methodology/Principal Findings: The study was performed at Children\u27s Hospital No. 2, Ho Chi Minh City, and Vinh Long Province Hospital, Vietnam from 2002 to 2005. Study patients included 103 with Dengue fever (DF), Dengue hemorrhagic fever (DHF), and Dengue shock syndrome (DSS), as diagnosed by the World Health Organization criteria. There were 189 healthy subjects, and 19 febrile illness patients of the same Kinh ethnicity. The levels of mast cell-derived mediators and -related cytokines in plasma were measured by ELISA. VEGF and sVEGFR-1 levels were significantly increased in DHF and DSS compared with those of DF and controls, whereas sVEGFR-2 levels were significantly decreased in DHF and DSS. Significant increases in tryptase and chymase levels, which were accompanied by high IL-9 and -17 concentrations, were detected in DHF and DSS patients. By day 4 of admission, VEGF, sVEGFRs, and proteases levels had returned to similar levels as DF and controls. In-vitro VEGF production by mast cells was examined in KU812 and HMC-1 cells, and was found to be highest when the cells were inoculated with Dengue virus and human Dengue virus-immune serum in the presence of IL-9. Conclusions: As mast cells are an important source of VEGF, tryptase, and chymase, our findings suggest that mast cell activation and mast cell-derived mediators participate in the development of DHF. The two proteases, particularly chymase, might serve as good predictive markers of Dengue disease severity