Capacity Constraint, Price Discrimination, and Oligopoly

In the presence of market power in oligopolistic environment, price discrimination is a natural phenomenon. Surprisingly this setting has not been analyzed in depth in the literature. In contrast with existing literature, e.g., Hazledine (2006) and Kutlu (2009), we consider quantity setting games where firms compete in two stages. In the first stage firms decide on the choice of capacity and in the second stage they decide on the structure of price discrimination where the level of price discrimination is exogenous. In contrast to Hazledine (2006) we find that in the Cournot framework the quantity-weighted average price depends on the level of price discrimination. We also find that in the Stackelberg framework both the leader and the follower price discriminate as opposed to Kutlu (2009) which concludes that the leader doesn't price discriminate. Moreover, it is discovered that both the players (even the follower) prefer to be in the Stackelberg framework rather than the Cournot framework when price discrimination exists. Comparing welfare under various settings, it is found that competition is not always good for the total welfare if price discrimination exists. desirable axioms.

Acute brucella melitensis M16 infection model in mice treated with tumor necrosis factor-alpha inhibitors

Introduction: There is limited data in the literature about brucellosis related to an intracellular pathogen and anti-tumor necrosis factor alpha (anti-TNFα) medication. The aim of this study was to evaluate acute Brucella infections in mice receiving anti-TNFα drug treatment. Methodology: Anti-TNFα drugs were injected in mice on the first and fifth days of the study, after which the mice were infected with B. melitensis M16 strain. Mice were sacrificed on the fourteenth day after infection. Bacterial loads in the liver and spleen were defined, and histopathological changes were evaluated. Results: Neither the liver nor the spleen showed an increased bacterial load in all anti-TNFα drug groups when compared to a non-treated, infected group. The most significant histopathological findings were neutrophil infiltrations in the red pulp of the spleen and apoptotic cells with hepatocellular pleomorphism in the liver. There was no significant difference among the groups in terms of previously reported histopathological findings, such as extramedullary hematopoiesis and granuloma formation. Conclusions: There were no differences in hepatic and splenic bacterial load and granuloma formation, which indicate worsening of the acute Brucella infection in mice; in other words, anti-TNFα treatment did not exacerbate the acute Brucella spp. infection in mice. © 2015 Kutlu et al

Ölümünün 27. yıldönümünde:Abdülhak Hamid Tarhan

Taha Toros Arşivi, Dosya No: 56-Abdülhak Hamit Tarhanİstanbul Kalkınma Ajansı (TR10/14/YEN/0033) İstanbul Development Agency (TR10/14/YEN/0033