40,571 research outputs found

    Magnetism and Mott Transition: A Slave-rotor Study

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    Motivated by the debate of spin-density-wave (SDW) versus local-moment (LM) picture in the iron-based superconducting (FeSC) materials, we consider a two-band orbital-symmetric Hubbard model in which there is robust Fermi surface nesting at (π,0)(\pi,0). We obtain the phase diagram of such system by a mean-field slave-rotor approach, in which the Fermi surface nesting and the SDW order are explicitly taken into account via a natural separation of scale between the Hund's coupling and the Coulomb interaction. We find that for a sizable range of Hund's coupling the Mott transition acquires a strong first-order character, but there also exists a small range of stronger Hund's coupling in which an enhancement of magnetization can be observed on the SDW side. We interpret the former scenario as one in which a sharp distinction can be drawn between LM and the SDW picture, and the latter scenario as one in which signs of LM physics begin to develop in the metallic phase. It is tempting to suggest that some FeSC materials are in the vicinity of the latter scenario.Comment: 7 pages, 5 figures; v2: Added discussions on anisotropy in renormalized hopping, clarifications and discussions with regard to orbital order, new inset in Fig. 3(d), expanded and revised bibliography, plus other minor revisions. Accepted to PR

    Gamma-ray Luminosity and Photon Index Evolution of FSRQ Blazars and Contribution to the Gamma-ray Background

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    We present the redshift evolutions and distributions of the gamma-ray luminosity and photon spectral index of flat spectrum radio quasar (FSRQ) type blazars, using non-parametric methods to obtain the evolutions and distributions directly from the data. The sample we use for analysis consists of almost all FSRQs observed with a greater than approximately 7 sigma detection threshold in the first year catalog of the Fermi Gamma-ray Space Telescope's Large Area Telescope, with redshfits as determined from optical spectroscopy by Shaw et al. We find that FSQRs undergo rapid gamma-ray luminosity evolution, but negligible photon index evolution, with redshift. With these evolutions accounted for we determine the density evolution and luminosity function of FSRQs, and calculate their total contribution to the extragalactic gamma-ray background radiation, resolved and unresolved, which is found to be 16(+10/-4)%, in agreement with previous studies.Comment: 9 pages, 10 figures, Accepted to Ap

    Automatically Designing CNN Architectures for Medical Image Segmentation

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    Deep neural network architectures have traditionally been designed and explored with human expertise in a long-lasting trial-and-error process. This process requires huge amount of time, expertise, and resources. To address this tedious problem, we propose a novel algorithm to optimally find hyperparameters of a deep network architecture automatically. We specifically focus on designing neural architectures for medical image segmentation task. Our proposed method is based on a policy gradient reinforcement learning for which the reward function is assigned a segmentation evaluation utility (i.e., dice index). We show the efficacy of the proposed method with its low computational cost in comparison with the state-of-the-art medical image segmentation networks. We also present a new architecture design, a densely connected encoder-decoder CNN, as a strong baseline architecture to apply the proposed hyperparameter search algorithm. We apply the proposed algorithm to each layer of the baseline architectures. As an application, we train the proposed system on cine cardiac MR images from Automated Cardiac Diagnosis Challenge (ACDC) MICCAI 2017. Starting from a baseline segmentation architecture, the resulting network architecture obtains the state-of-the-art results in accuracy without performing any trial-and-error based architecture design approaches or close supervision of the hyperparameters changes.Comment: Accepted to Machine Learning in Medical Imaging (MLMI 2018

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    How Much Multiuser Diversity is Required for Energy Limited Multiuser Systems?

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    Multiuser diversity (MUDiv) is one of the central concepts in multiuser (MU) systems. In particular, MUDiv allows for scheduling among users in order to eliminate the negative effects of unfavorable channel fading conditions of some users on the system performance. Scheduling, however, consumes energy (e.g., for making users' channel state information available to the scheduler). This extra usage of energy, which could potentially be used for data transmission, can be very wasteful, especially if the number of users is large. In this paper, we answer the question of how much MUDiv is required for energy limited MU systems. Focusing on uplink MU wireless systems, we develop MU scheduling algorithms which aim at maximizing the MUDiv gain. Toward this end, we introduce a new realistic energy model which accounts for scheduling energy and describes the distribution of the total energy between scheduling and data transmission stages. Using the fact that such energy distribution can be controlled by varying the number of active users, we optimize this number by either (i) minimizing the overall system bit error rate (BER) for a fixed total energy of all users in the system or (ii) minimizing the total energy of all users for fixed BER requirements. We find that for a fixed number of available users, the achievable MUDiv gain can be improved by activating only a subset of users. Using asymptotic analysis and numerical simulations, we show that our approach benefits from MUDiv gains higher than that achievable by generic greedy access algorithm, which is the optimal scheduling method for energy unlimited systems.Comment: 28 pages, 9 figures, submitted to IEEE Trans. Signal Processing in Oct. 200

    Contribution of bsggb \to sgg through the QCD anomaly in exclusive decays B±(η,η)(K±,K±)B^{\pm}\to (\eta^{\prime},\eta)(K^{\pm}, K^{*\pm}) and B0(η,η)(K0,K0)B^{0}\to (\eta^{\prime},\eta)(K^{0},K^{*0})

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    We compute the decay rates for the exclusive decays B±(η,η)(K±,K±)B^{\pm} \to (\eta^{\prime},\eta) (K^{\pm}, K^{*\pm}) and B0(η,η)(K0,K0)B^{0}\to (\eta^{\prime},\eta) (K^{0}, K^{*0}) in a QCD-improved factorization framework by including the contribution from the process bsggs(η,η)b\to sgg \to s (\eta^{\prime}, \eta) through the QCD anomaly. This method provides an alternative estimate of the contribution bsccˉs(η,η)b \to s c\bar{c} \to s(\eta,\eta^\prime) to these decays as compared to the one using the intrinsic charm content of the η\eta^{\prime} and η\eta mesons determined through the decays J/ψ(η,η,ηc)γJ/\psi \to (\eta,\eta^\prime ,\eta_c) \gamma. The resulting branching ratios are compared with the CLEO data on B±ηK±B^{\pm} \to \eta^{\prime} K^{\pm} and B0ηK0B^{0} \to \eta^{\prime} K^{0} and predictions are made for the rest.Comment: 16 pages including 4 postscript figures; uses epsfig. The most recent branching ratios from CLEO, ref. [5], are taken into account. The theory part is unchange

    Methylation of CpG island is not a ubiquitous mechanism for the loss of oestrogen receptor in breast cancer cells.

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    Methylation has been shown to play an important role in the down-regulation of oestrogen receptors (ER) in breast cancer cells. One critical question that remains unclear is whether methylation can account for the loss of ER expression in cells derived from an ER-positive cell line. This laboratory has established an in vitro cell system using long-term growth of human ER-positive breast cancer cell line T47D in oestrogen-free medium. A clonal cell line, T47D:C4:2 (C4:2), has been characterized. Unlike T47D:A18 (A18), which is a T47D line maintained in oestrogen medium, C4:2 has lost the expression of ER and hormone responsiveness. DNA fingerprinting and restriction fragment length polymorphism (RFLP) analysis results confirmed that C4:2 was of the same lineage as A18. These cell lines provide an invaluable system to study the mechanism of ER expression and regulatory pathways leading to hormone-independent growth. The results here clearly demonstrate that the ER CpG island in C4:2 cells remains unmethylated. The loss of ER in the cell line must be due to mechanisms other than methylation. We also evaluated the ER CpG island in the MDA-MB-231:10A (10A) cell line, which is a clone from the MDA-MB-231 line obtained from ATCC and the DNA from the MDA-MB-231 cell line used in the original report. Unlike the cell line from the report, which showed a full methylation pattern in the island, the 10A line only showed a partial methylation pattern in the CpG island. Possible mechanisms pertaining to the heterogeneous methylation pattern of the ER CpG island in the breast cancer cells are discussed
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