Dynamic stability investigation of two right circular cylinders in axial free-flight at Mach numbers from 0.4 to 1.7 : fineness-ratio-2.56 cylinder and fineness-ratio-4.0 cylinder with flared afterbody

Dynamic longitudinal stability of right circular cylinders in axial free fligh

New Easter Tide Prayers of the Mass in the Typal Edition of the Roman Missal of 2002.

Autor obrađuje četrnaest novih molitava vazmenoga vremena, koje se nalaze u novom tipskom izdanju Rimskoga misala iz 2002. godine. U prvom dijelu istražio je i naznačio izvore tih molitava, od kojih su tri novosastavljene, a ostalih jedanaest preuzeto je iz drevnih sakramentara. U drugom dijelu razlaže se nauk o Kristovom vazmenom otajstvu po kojem se vjernici spašavaju. Naglašava se "pozitivni" vid vazmenog otajstva: Kristovo uskrsnuće i proslava, a osobito se ističe Kristova velikosvećenička uloga, što u odnosu na prethodni misal predstavlja novinu. Vjernici se spašavaju po tome da uzimaju udjela u Kristovu vazmenom otajstvu. Oni su već zajedno s njime proslavljeni, što će se u potpunosti očitovati u blaženoj vječnosti. Ističe se također otajstvena snaga bogoslužnog slavlja. Ono ima tu moć da Kristovo spasenje učini stvarnim i djelatnim među vjernicima koji u tome slavlju sudjeluju.The author elaborates on fourteen new prayers of the Easter tide that can be found in the new typal edition of the Roman missal from 2002. In the first section the author explores and discovers the sources of the prayers. Three of them have been recently composed, while thirteen have been taken from the old books of sacraments. The second section of the paper explains the doctrine of Christ’s Easter Mystery by which the faithful are saved. The “positive aspect” of the Mystery is emphasised: Christ’s resurrection and His role of the Great High Priest. This is a novelty compared to the previous edition of the missal. The salvation of the faithful is presented in their participation in Christ’s Easter Mystery. His glory is their glory too, which becomes completely obvious in the blessed eternity. The emphasis is also given to the sacramental power of the Eucharist where there is the power which makes Christ’s salvation real and efficient for the faithful who participate in this celebration

Platinum-group minerals in the Skouries Cu-Au (Pd, Pt, Te) porphyry deposit

The Skouries deposit is a platinum-group element (PGE) enriched Cu-Au porphyry system located in the Chalkidiki peninsula, Greece, with associated Ag, Bi and Te enrichment. The deposit is hosted by multiple porphyritic monzonite and syenite intrusions, which originated from a magma chamber at depth. An initial quartz monzonite porphyritic intrusion contains a quartz–magnetite ± chalcopyrite–pyrite vein stockwork with intense potassic alteration. The quartz monzonite intrusion is cross cut by a set of syenite and mafic porphyry dykes and quartz–chalcopyrite–bornite ± magnetite veins which host the majority of the Cu and Au mineralisation. Late stage quartz–pyrite veins, with associated phyllic alteration crosscut all previous vein generations. Electron microprobe and scanning electron microscopy shows that the PGE are hosted by platinum-group minerals (PGM) in the quartz-chalcopyrite–bornite ± magnetite veins and within potassic alteration assemblages. The PGE mineralisation in Skouries is therefore part of the main high temperature hypogene mineralisation event. Platinum-group minerals at Skouries include: sopcheite [Ag4Pd3Te4], merenskyite [(Pd,Pt)(Te,Bi)2] and kotulskite [Pd(Te,Bi)], with rare telargpalite [(Pd,Ag)3Te], isomertieite [Pd11Sb2As2], naldrettite [Pd2Sb], testibiopalladite [PdTe(Sb,Te)] and sobolevskite [PdBi]. The most common platinum-group mineral is sopcheite. The PGM in Skouries are small, 52 µm2 on average, and occur as spherical grains on the boundaries between sulphides and silicates, and as inclusions within hydrothermal quartz and sulphides. These observations support a “semi-metal collector model” whereby an immiscible Bi-Te melt acts as a collector for PGE and other precious metals in high temperature hydrothermal fluids. This mechanism would allow the formation of PGM in porphyries without Pt and Pd fluid saturation

Altered extracellular vesicle microrna expression in ischemic stroke and small vessel disease

Active transport of microRNAs (miRNA) in extracellular vesicles (EV) occurs in disease. Circulating EV-packaged miRNAs in the serum of stroke patients were compared to stroke mimics with matched cardio- and cerebrovascular risk factors, with corroboration of results in a pre-clinical model. An unbiased miRNA microarray was performed in stroke vs stroke mimic patients (n=39). Results were validated (n=173 patients) by real-time quantitative polymerase chain reaction. miRNA expression was quantified in total serum/EV (n=5-7) of naïve adult spontaneously hypertensive stroke-prone rats (SHRSP), their normotensive reference strain (Wistar Kyoto, WKY) and in circulating EV (n=3), peri-infarct brain (n=6) or EV derived from this region (n=3) in SHRSP following transient middle cerebral artery occlusion (tMCAO). Circulating EV concentration did not differ between stroke and stroke mimic patients. The microarray identified many altered EV-packaged miRNAs: levels of miRNA-17-5p, -20b-5p and -93-5p (miRNA-17 family members) and miRNA-27b-3p were significantly (p≤0.05) increased in stroke vs stroke mimic patients. Patients with small vessel disease (SVD) consistently had the highest miRNA levels. Circulating EV concentration was unaltered between naïve SHRSP and WKY but levels of miRNA-17-5p and -93-5p were significantly increased in SHRSP. tMCAO in SHRSP did not further alter circulating EV miRNA-17 family member expression and nor did it change total miRNA-17 family levels in peri-infarct brain tissue or in EV isolated from this region at 24hrs post-tMCAO. Changes in EV packaged miRNA expression was validated in patients with stroke, particularly those with SVD, and corroborated pre-clinically. Together, altered circulating EV levels of miRNA-17 family members may reflect the chronic sequelae underlying cerebrovascular SVD rather than the acute ischemic stroke itself