2,969 research outputs found
Ionization probability of iron particles at meteoritic velocities
Ion production for ablation of micron size iron particles moving at supersonic speed in air and argo
Techniques for the observation of micrometeorite craters in metal substrates utilizing electron micrographic replica methods
Thin film replica technique for obtaining high quality electron micrographs of craters in metal substrates formed by microscopic hypervelocity particle impac
Ionization probability of iron particles at meteoric velocities
Ion pairs produced by total ablation of iron particles in air and argon measured as function of particle velocit
High voltage breakdown initiated by particle impact
High voltage breakdown initiated by particle impact across electrode ga
Micrometeorite bombardment initiating discharges and breakdown in ion thrustors
Micrometeorite bombardment initiating discharges and breakdown in ion thrustor
Slip or not slip? A methodical examination of the interface formation model using two-dimensional droplet spreading on a horizontal planar substrate as a prototype system
We consider the spreading of a thin two-dimensional droplet on a planar
substrate as a prototype system to compare the contemporary model for contact
line motion based on interface formation of Shikhmurzaev [Int. J. Multiphas.
Flow 19, 589 (1993)], to the more commonly used continuum fluid dynamical
equations augmented with the Navier-slip condition. Considering quasistatic
droplet evolution and using the method of matched asymptotics, we find that the
evolution of the droplet radius using the interface formation model reduces to
an equivalent expression for a slip model, where the prescribed microscopic
dynamic contact angle has a velocity dependent correction to its static value.
This result is found for both the original interface formation model
formulation and for a more recent version, where mass transfer from bulk to
surface layers is accounted for through the boundary conditions. Various
features of the model, such as the pressure behaviour and rolling motion at the
contact line, and their relevance, are also considered in the prototype system
we adopt.Comment: 45 pages, 18 figure
A laboratory measurement of meteor luminous efficiency
Laboratory measurement of meteor luminous efficienc
Drag coefficients of microscopic spheres in free-molecule flow
Drag coefficients of microscopic spheres in free molecule flo
From Service to Experience: Understanding and Defining the Hospitality Business
Failure adequately to define or understand hospitality as a commercial phenomenon has created a fragmented academic environment and a schizophrenia in the industry that has the potential to limit its development as a global industry. This article suggests that, by redefining hospitality as behaviour and experience, a new perspective emerges that has exciting implications for the management of hospitality businesses. A framework to describe hospitality in the commercial domain is proposed. This framework suggests a focus on the host–guest relationship, generosity, theatre and performance, ‘lots of little surprises’, and the security of strangers – a focus that provides guests with experiences that are personal, memorable and add value to their lives
Genetic Variation in Selenoprotein Genes, Lifestyle, and Risk of Colon and Rectal Cancer
BACKGROUND: Associations between selenium and cancer have directed attention to role of selenoproteins in the carcinogenic process. METHODS: We used data from two population-based case-control studies of colon (n = 1555 cases, 1956 controls) and rectal (n = 754 cases, 959 controls) cancer. We evaluated the association between genetic variation in TXNRD1, TXNRD2, TXNRD3, C11orf31 (SelH), SelW, SelN1, SelS, SepX, and SeP15 with colorectal cancer risk. RESULTS: After adjustment for multiple comparisons, several associations were observed. Two SNPs in TXNRD3 were associated with rectal cancer (rs11718498 dominant OR 1.42 95% CI 1.16,1.74 pACT 0.0036 and rs9637365 recessive 0.70 95% CI 0.55,0.90 pACT 0.0208). Four SNPs in SepN1 were associated with rectal cancer (rs11247735 recessive OR 1.30 95% CI 1.04,1.63 pACT 0.0410; rs2072749 GGvsAA OR 0.53 95% CI 0.36,0.80 pACT 0.0159; rs4659382 recessive OR 0.58 95% CI 0.39,0.86 pACT 0.0247; rs718391 dominant OR 0.76 95% CI 0.62,0.94 pACT 0.0300). Interaction between these genes and exposures that could influence these genes showed numerous significant associations after adjustment for multiple comparisons. Two SNPs in TXNRD1 and four SNPs in TXNRD2 interacted with aspirin/NSAID to influence colon cancer; one SNP in TXNRD1, two SNPs in TXNRD2, and one SNP in TXNRD3 interacted with aspirin/NSAIDs to influence rectal cancer. Five SNPs in TXNRD2 and one in SelS, SeP15, and SelW1 interacted with estrogen to modify colon cancer risk; one SNP in SelW1 interacted with estrogen to alter rectal cancer risk. Several SNPs in this candidate pathway influenced survival after diagnosis with colon cancer (SeP15 and SepX1 increased HRR) and rectal cancer (SepX1 increased HRR). CONCLUSIONS: Findings support an association between selenoprotein genes and colon and rectal cancer development and survival after diagnosis. Given the interactions observed, it is likely that the impact of cancer susceptibility from genotype is modified by lifestyle
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