Waveform simulator synthesizes complex functions

Multichannel apparatus produces or simulates a complex curve which can be viewed on an oscilloscope display surface and can be adjusted to match an original complex experimentally produced curve

Experimental investigation of the fundamental modes of a collisionless plasma quarterly report no. 1, mar. 10 - jun. 10, 1964

Plasma probe manipulators, electromagnetic shielding, receiver signal to noise ratio, and plasma parameters for experiment and measurements on plasma propertie

Waveform simulator Patent

Sign wave generation simulator for variable amplitude, frequency, damping, and phase pulses for oscilloscope displa

Cyclotron waves in a collisionless plasma

Plasma waves with resonances near electron cyclotron frequency investigated in long collisionless plasma column - wavelength dispersion curves and relation

Experimental investigation of the fundamental modes of a collisionless plasma quarterly report no. 3, sep. 11 - dec. 10, 1964

Plasma waves between electron cyclotron frequency and upper hybrid frequency in collisionless hydrogen plasm

Experimental investigation of the fundamental modes of a collisionless plasma quarterly report no. 2, jun. 11 - sep. 10, 1964

Dispersion relation for waves near electron cyclotron frequenc

The role of cluster evolution in disrupting planetary systems and disks: the Kozai mechanism

We examine the effects of dynamical evolution in clusters on planetary systems or protoplanetary disks orbiting the components of binary stars. In particular, we look for evidence that the companions of host stars of planetary systems or disks could have their inclination angles raised from zero to between the threshold angles (39.23 degrees and 140.77 degrees) that can induce the Kozai mechanism. We find that up to 20 per cent of binary systems have their inclination angles increased to within the threshold range. Given that half of all extrasolar planets could be in binary systems, we suggest that up to 10 per cent of extrasolar planets could be affected by this mechanism.Comment: 5 pages, 4 figures, accepted for publication in MNRAS. References update

Migrant Youths and YouTube Entertainment : Media Participation in Post-Migrant Finland

Recent media studies in Europe have stressed the importance of studying societies’ negotiations on migration and the ability of migrants and other ethnic minorities to participate in these processes. Social media platforms have been widely praised for their openness to culturally diverse voices and representations. For minorities who have often been ignored and misrepresented in traditional media, these platforms arguably provide an empowering space where they can self-represent their identities, provide counter-representations to large and diverse audiences, and enhance their careers as media professionals. The video streaming social media platform, YouTube, is at the forefront of media participation. However, YouTube also has been criticized for promoting a highly commercialized culture of self-commodification and entertainment that maintains the status quo instead of enabling progressive social change. This study presents the results of an examination of the YouTube scene in Finland, a country with the lowest percentage of foreign-born inhabitants in Northern and Western Europe, where few YouTubers with migrant backgrounds have become increasingly visible within the last few years. Drawing on interviews with YouTubers, the study presents new insights into ethnic minority participation on YouTube and challenges binary oppositions between commercialism, entertainment and social change.Peer reviewe

Creative clusters and innovation (C.Chapain, P.Cooke, L.De Propris, S.MacNeill, J.Mateos-Garcia, NESTA)

Copyright @ 2012 Intellec

Preventing a shock to the system. Two-pore channel 1 negatively regulates anaphylaxis

The mammalian two-pore channels TPC1 and TPC2 are patho-physiologically relevant endo-lysosomal cation channels regulated by the Ca2+ mobilising messenger NAADP and the phosphoinositide PI(3,5)P2. Recent work by Arlt et al shows that genetic or chemical inhibition of TPC1 in mice promotes anaphylaxis in vivo through a mechanism involving enhanced endoplasmic reticulum Ca2+ release and secretion in mast cells