Photoanode Engineering Using TiO 2 Nanofibers for Enhancing the Photovoltaic Parameters of Natural Dye Sensitised Solar Cells

Dye Sensitized solar cell (DSSC) have been looked upon as having the potential to modernize photovoltaic as a cost effective technology. Especially nanostructured DSSC is proposed to have the capability to boost the efficiency by limiting charge recombination, thereby increasing the charge transportation which affects the overall conversion efficiency favourably. In the present work we discuss the effect of nanofibers as photo anode for increasing the efficiency of a dye sensitized solar cell. As we know nanostructured metal oxides have paying much attention in the field of photovoltaics due to their physical properties and dimensionality. This type of geometry provides direct and spatially separated charge transport channels for electrons and holes. TiO2 single-crystalline nanofibers of different diameter are prepared by electrospinning process and TiO2 nanoparticles by doctor blade technique are used for fabricating the device using natural sensitizers

Anthraquinones- A probe to enhance the photovoltaic properties of DSSCs

Natural dye sensitized solar cells are a promising class of photovoltaic cells with the capacity of generating green energy at low production cost since no expensive equipment is required in their fabrication. Photovoltaics are a precious technology in the hasty world where energy prices are goes on increasing within seconds. Researchers are focusing to facilitate for producing eco-friendly, low cost and more efficient dye sensitized solar cells. In the present work we discuss the comparative photovoltaic studies of Lawsone, a natural dye from henna plant and Alizarin, a natural dye from the root of madder for fabricating the Dye sensitized solar cells (DSSCs). The absorption spectrum of Lawsone and Alizarin is found to be shifted to the longer wavelength region after the complex formation. As a result there is a significant increase in short circuit current density and conversion efficiency. This result compares with the standard dye i.e. N719 dye

Photoanode Engineering Using TiO 2 Nanofibers for Enhancing the Photovoltaic Parameters of Natural Dye Sensitised Solar Cells

Dye Sensitized solar cell (DSSC) have been looked upon as having the potential to modernize photovoltaic as a cost effective technology. Especially nanostructured DSSC is proposed to have the capability to boost the efficiency by limiting charge recombination, thereby increasing the charge transportation which affects the overall conversion efficiency favourably. In the present work we discuss the effect of nanofibers as photo anode for increasing the efficiency of a dye sensitized solar cell. As we know nanostructured metal oxides have paying much attention in the field of photovoltaics due to their physical properties and dimensionality. This type of geometry provides direct and spatially separated charge transport channels for electrons and holes. TiO2 single-crystalline nanofibers of different diameter are prepared by electrospinning process and TiO2 nanoparticles by doctor blade technique are used for fabricating the device using natural sensitizers

PHOTOVOLTAIC STUDIES OF DYE SENSITIZED SOLAR CELL WITH MODIFIED PEDOT: PSS AS COUNTER ELECTRODE

ABSTRACT A dye sensitized solar cell is one of the solar cells that use organic dyes to achieve photovoltaic properties

Dissecting features of epigenetic variants underlying cardiometabolic risk using full-resolution epigenome profiling in regulatory elements

Sparse profiling of CpG methylation in blood by microarrays has identified epigenetic links to common diseases. Here we apply methylC-capture sequencing (MCC-Seq) in a clinical population of similar to 200 adipose tissue and matched blood samples (N-total similar to 400), providing high- resolution methylation profiling (>1.3 M CpGs) at regulatory elements. We link methylation to cardiometabolic risk through associations to circulating plasma lipid levels and identify lipid-associated CpGs with unique localization patterns in regulatory elements. We show distinct features of tissue-specific versus tissue-independent lipid-linked regulatory regions by contrasting with parallel assessments in similar to 800 independent adipose tissue and blood samples from the general population. We follow-up on adipose-specific regulatory regions under (1) genetic and (2) epigenetic (environmental) regulation via integrational studies. Overall, the comprehensive sequencing of regulatory element methylomes reveals a rich landscape of functional variants linked genetically as well as epigenetically to plasma lipid traits

An atlas of genetic influences on osteoporosis in humans and mice.

Osteoporosis is a common aging-related disease diagnosed primarily using bone mineral density (BMD). We assessed genetic determinants of BMD as estimated by heel quantitative ultrasound in 426,824 individuals, identifying 518 genome-wide significant loci (301 novel), explaining 20% of its variance. We identified 13 bone fracture loci, all associated with estimated BMD (eBMD), in ~1.2 million individuals. We then identified target genes enriched for genes known to influence bone density and strength (maximum odds ratio (OR) = 58, P = 1 × 10-75) from cell-specific features, including chromatin conformation and accessible chromatin sites. We next performed rapid-throughput skeletal phenotyping of 126 knockout mice with disruptions in predicted target genes and found an increased abnormal skeletal phenotype frequency compared to 526 unselected lines (P < 0.0001). In-depth analysis of one gene, DAAM2, showed a disproportionate decrease in bone strength relative to mineralization. This genetic atlas provides evidence linking associated SNPs to causal genes, offers new insight into osteoporosis pathophysiology, and highlights opportunities for drug development

An atlas of genetic influences on osteoporosis in humans and mice.

Osteoporosis is a common aging-related disease diagnosed primarily using bone mineral density (BMD). We assessed genetic determinants of BMD as estimated by heel quantitative ultrasound in 426,824 individuals, identifying 518 genome-wide significant loci (301 novel), explaining 20% of its variance. We identified 13 bone fracture loci, all associated with estimated BMD (eBMD), in ~1.2 million individuals. We then identified target genes enriched for genes known to influence bone density and strength (maximum odds ratio (OR) = 58, P = 1 × 10-75) from cell-specific features, including chromatin conformation and accessible chromatin sites. We next performed rapid-throughput skeletal phenotyping of 126 knockout mice with disruptions in predicted target genes and found an increased abnormal skeletal phenotype frequency compared to 526 unselected lines (P < 0.0001). In-depth analysis of one gene, DAAM2, showed a disproportionate decrease in bone strength relative to mineralization. This genetic atlas provides evidence linking associated SNPs to causal genes, offers new insight into osteoporosis pathophysiology, and highlights opportunities for drug development