2,038 research outputs found
A post-merger enhancement only in star-forming Type 2 Seyfert galaxies:the deep learning view
Supermassive black holes require a reservoir of cold gas at the centre of their host galaxy in order to accrete and shine as active galactic nuclei (AGN). Major mergers have the ability to drive gas rapidly inwards, but observations trying to link mergers with AGN have found mixed results due to the difficulty of consistently identifying galaxy mergers in surveys. This study applies deep learning to this problem, using convolutional neural networks trained to identify simulated post-merger galaxies from survey-realistic imaging. This provides a fast and repeatable alternative to human visual inspection. Using this tool, we examine a sample of ~8500 Seyfert 2 galaxies (L[OIII] ~ erg/s) at z < 0.3 in the Sloan Digital Sky Survey and find a merger fraction of % compared with inactive control galaxies, in which we find a merger fraction of %, indicating an overall lack of mergers among AGN hosts compared with controls. However, matching the controls to the AGN hosts in stellar mass and star formation rate reveals that AGN hosts in the star-forming blue cloud exhibit a ~ merger enhancement over controls, while those in the quiescent red sequence have significantly lower relative merger fractions, leading to the observed overall deficit due to the differing SFR distributions. We conclude that while mergers are not the dominant trigger of all low-luminosity, obscured AGN activity in the nearby Universe, they are more important to AGN fuelling in galaxies with higher cold gas mass fractions as traced through star formation
The ALHAMBRA Survey: Bayesian Photometric Redshifts with 23 bands for 3 squared degrees
The ALHAMBRA (Advance Large Homogeneous Area Medium Band Redshift
Astronomical) survey has observed 8 different regions of the sky, including
sections of the COSMOS, DEEP2, ELAIS, GOODS-N, SDSS and Groth fields using a
new photometric system with 20 contiguous ~ filters covering the
optical range, combining them with deep imaging. The observations,
carried out with the Calar Alto 3.5m telescope using the wide field (0.25 sq.
deg FOV) optical camera LAICA and the NIR instrument Omega-2000, correspond to
~700hrs on-target science images. The photometric system was designed to
maximize the effective depth of the survey in terms of accurate spectral-type
and photo-zs estimation along with the capability of identification of
relatively faint emission lines. Here we present multicolor photometry and
photo-zs for ~438k galaxies, detected in synthetic F814W images, complete down
to I~24.5 AB, taking into account realistic noise estimates, and correcting by
PSF and aperture effects with the ColorPro software. The photometric ZP have
been calibrated using stellar transformation equations and refined internally,
using a new technique based on the highly robust photometric redshifts measured
for emission line galaxies. We calculate photometric redshifts with the BPZ2
code, which includes new empirically calibrated templates and priors. Our
photo-zs have a precision of for I<22.5 and 1.4% for
22.5<I<24.5. Precisions of less than 0.5% are reached for the brighter
spectroscopic sample, showing the potential of medium-band photometric surveys.
The global shows a mean redshift =0.56 for I=0.86 for
I<24.5 AB. The data presented here covers an effective area of 2.79 sq. deg,
split into 14 strips of 58.5'x15.5' and represents ~32 hrs of on-target.Comment: The catalog data and a full resolution version of this paper is
available at https://cloud.iaa.csic.es/alhambra
Polo-like kinase 3 regulates CtIP during DNA double-strand break repair in G1
DNA double-strand breaks (DSBs) are repaired by nonhomologous end joining (NHEJ) or homologous recombination (HR). The C terminal binding proteinâinteracting protein (CtIP) is phosphorylated in G2 by cyclin-dependent kinases to initiate resection and promote HR. CtIP also exerts functions during NHEJ, although the mechanism phosphorylating CtIP in G1 is unknown. In this paper, we identify Plk3 (Polo-like kinase 3) as a novel DSB response factor that phosphorylates CtIP in G1 in a damage-inducible manner and impacts on various cellular processes in G1. First, Plk3 and CtIP enhance the formation of ionizing radiation-induced translocations; second, they promote large-scale genomic deletions from restriction enzyme-induced DSBs; third, they are required for resection and repair of complex DSBs; and finally, they regulate alternative NHEJ processes in Kuâ/â mutants. We show that mutating CtIP at S327 or T847 to nonphosphorylatable alanine phenocopies Plk3 or CtIP loss. Plk3 binds to CtIP phosphorylated at S327 via its Polo box domains, which is necessary for robust damage-induced CtIP phosphorylation at S327 and subsequent CtIP phosphorylation at T847
N7-methylguanosine methylation of tRNAs regulates survival to stress in cancer
Tumour progression and therapy tolerance are highly regulated and complex processes largely dependent on the plasticity of cancer cells and their capacity to respond to stress. The higher plasticity of cancer cells highlights the need for identifying targetable molecular pathways that challenge cancer cell survival. Here, we show that
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