340 research outputs found

    Cooling of Quark Stars in the Color Superconductive Phase: Effect of Photons from Glueball decay

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    The cooling history of a quark star in the color superconductive phase is investigated. Here we specifically focus on the 2-flavour color (2SC) phase where novel process of photon generation via glueball (GLB) decay have been already investigated (Ouyed & Sannino 2001). The picture we present here can in principle be generalized to quark stars entering a superconductive phase where similar photon generation mechanisms are at play. As much as 10^{45}-10^{47} erg of energy is provided by the GLB decay in the 2SC phase. The generated photons slowly diffuse out of the quark star keeping it hot and radiating as a black-body (with possibly a Wien spectrum in gamma-rays) for millions of years. We discuss hot radio-quiet isolated neutron stars in our picture (such as RX J185635-3754 and RX J0720.4-3125) and argue that their nearly blackbody spectra (with a few broad features) and their remarkably tiny hydrogen atmosphere are indications that these might be quark stars in the color superconductive phase where some sort of photon generation mechanism (reminiscent of the GLB decay) has taken place. Fits to observed data of cooling compact stars favor models with superconductive gaps of Delta_2SC = 15-35 MeV and densities rho_2SC=(2.5-3.0)rho_N (rho_N being the nuclear matter saturation density) for quark matter in the 2SC phase. If correct, our model combined with more observations of isolated compact stars could provide vital information to studies of quark matter and its exotic phases.Comment: 7 journal pages, 4 figures, accepted for publication in MNRAS (more discussions on photon cooling versus neutrino cooling before and after pairing of quarks

    Quantifying Four Decades of Arid-region Agricultural Development in Arequipa, Peru Using Landsat

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    The Arequipa Nexus Institute for Food, Energy and the Environment (Nexus Institute) is located in Southwestern Peru, generally bounded by the city of Arequipa to the east, the Majes River to the west, the Pacific Ocean to the south, and the Andes mountains to the north. Though agriculture has been practiced in parts of this cool desert region (MAT~15Ā°C, MA

    Processfolio: uniting Academic Literacies and Critical Emancipatory Action Research for practitioner-led inquiry into EAP writing assessment

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    This paper reports on the design and implementation of an alternative form of writing assessment on a UK English for Academic Purposes (EAP) presessional course. The assessment, termed processfolio, was a response to research inquiry into how writing assessment in a local context negated student agency and inculcated disempowering models of teaching and learning academic writing. The project merged an Academic Literacies approach to writing (Lea and Street, 1998) with a Critical Emancipatory Action Research (Carr and Kemmis, 1986) framework and a Critical Realist(Bhaskar, 1989) perspective. Data collected from the folios and interviews with students and teachers on their experiences of the processfolio found that a small scale intervention has potential for agency to be exercised within the highly constrained context of a UK EAP pre-sessional. New directions in research are proposed which can engage students and teachers to work for change in UK EAP assessment within their internal and external constraints

    ADHD and brain anatomy:What do academic textbooks used in the Netherlands tell students?

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    Studies of brain size of children classified with ADHD appear to reveal smaller brains when compared to ā€˜normalā€™ children. Yet, what does this mean? Even with the use of rigorously screened case and control groups, these studies show only small, average group differences between children with and without an ADHD classification. However, academic textbooks used in the Netherlands often portray individual children with an ADHD classification as having a different, malfunctioning brain that necessitates medical intervention. This conceptualisation of ADHD might serve professional interests, but not necessarily the interests of children

    Interferon-Ī³-Producing CD4+ T Cells Drive Monocyte Activation in the Bone Marrow During Experimental Leishmania donovani Infection

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    Ly6Chi inflammatory monocytes develop in the bone marrow and migrate to the site of infection during inflammation. Upon recruitment, Ly6Chi monocytes can differentiate into dendritic cells or macrophages. According to the tissue environment they can also acquire different functions. Several studies have described pre-activation of Ly6Chi monocytes in the bone marrow during parasitic infection, but whether this process occurs during experimental visceral leishmaniasis and, if so, the mechanisms contributing to their activation are yet to be established. In wild type C57BL/6 (B6) mice infected with Leishmania donovani, the number of bone marrow Ly6Chi monocytes increased over time. Ly6Chi monocytes displayed a highly activated phenotype from 28 days to 5 months post infection (p.i), with >90% expressing MHCII and >20% expressing iNOS. In comparison, in B6.Rag2 -/- mice <10% of bone marrow monocytes were MHCII+ at day 28 p.i., an activation deficiency that was reversed by adoptive transfer of CD4+ T cells. Depletion of CD4+ T cells in B6 mice and the use of mixed bone marrow chimeras further indicated that monocyte activation was driven by IFNĪ³ produced by CD4+ T cells. In B6.Il10 -/- mice, L. donovani infection induced a faster but transient activation of bone marrow monocytes, which correlated with the magnitude of CD4+ T cell production of IFNĪ³ and resolution of the infection. Under all of the above conditions, monocyte activation was associated with greater control of parasite load in the bone marrow. Through reinfection studies in B6.Il10 -/- mice and drug (AmBisomeĀ®) treatment of B6 mice, we also show the dependence of monocyte activation on parasite load. In summary, these data demonstrate that during L. donovani infection, Ly6Chi monocytes are primed in the bone marrow in a process driven by CD4+ T cells and whereby IFNĪ³ promotes and IL-10 limits monocyte activation and that the presence of parasites/parasite antigen plays a crucial role in maintaining bone marrow monocyte activation
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