271 research outputs found
Tracing Changes in Families Who Participated in the Home-Start Parenting Program: Parental Sense of Competence as Mechanism of Change
- Author
- A Bandura
- Alithe L. van den Akker
- B Hess
- B Hoza
- BO Muthén
- BO Muthén
- BO Muthén
- C Leung
- C Webster-Stratton
- C Webster-Stratton
- CL Smith
- DE Stull
- DH Dallaire
- DM Gondoli
- DP MacKinnon
- DP MacKinnon
- DR Maughan
- DS DeGarmo
- DS Shaw
- EB Owens
- Ellen Reitz
- GA Kane
- GR Patterson
- HJA Bakel van
- HT Chen
- J Barnes
- J Belsky
- J Cheong
- J MacLoed
- J Patterson
- JB Willett
- Jessica J. Asscher
- JJ Asscher
- JL Ohan
- JL Schafer
- JM Riksen-Walraven
- Jo Hermanns
- JR Weisz
- JRM Gerris
- LRA Alink
- M Erickson
- M Galama
- M Verhoeven
- M Verhoeven
- MA Slater
- MA Sweet
- Maja Deković
- MR Sanders
- N Frost
- Peter Prinzie
- PJ Curran
- PK Coleman
- PK Coleman
- R Spoth
- RE Tremblay
- RW Thompson
- S Landy
- SP Hinshaw
- TJ Dishion
- TL Jones
- TM Achenbach
- Publication venue
- Springer US
- Publication date
- 01/01/2010
- Field of study
The present study aimed to (1) determine the long-term effectiveness of Home-Start, a preventive parenting program, and (2) test the hypothesis that changes in maternal sense of competence mediate the program’s effects. Participants were 124 mothers (n = 66 intervention, n = 58 comparison). Four assessments took place during a 1-year period. Latent growth modeling showed that Home-Start enhanced growth in maternal sense of competence and supportive parenting, and led to a decrease in the use of inept discipline. Results of mediational and cross-lagged analyses were consistent with the hypothesized model: Participation in Home-Start was related to the changes in maternal sense of competence, which in turn predicted changes in parenting. The results affirm the importance of directly targeting parental sense of competence in the context of prevention work with parents
Sex- and age-dependent association of SLC11A1 polymorphisms with tuberculosis in Chinese: a case control study
- Author
- A Schwenk
- B Vesosky
- BS Zwilling
- C Sikstrom
- CB Holmes
- CH Barton
- Che Kit Lin
- CM Chan
- DW Haas
- E Sin
- EG Hoal
- ES Lander
- Eudora YD Chow
- F Supek
- FJ Kallmann
- G Govoni
- HF Duan
- HT Li
- J Fitness
- JC Delgado
- JM Blackwell
- JM McDermid
- Kam Keung Chan
- Kim Hung Leung
- Kin Sang Chan
- KY Yuen
- L Henn
- Lap San Yiu
- LR Cardon
- M Cellier
- M Cellier
- M Chan-Yeung
- M Chan-Yeung
- M Zevallos
- MK Lam
- N Jabado
- NE Breslow
- NP Juffermans
- P Selvaraj
- PDO Davies
- PM Murphy
- PS Gao
- R Bellamy
- RF Ritchie
- S Gruenheid
- S Marquet
- S Meddows-Taylor
- S Nelson
- S Ryu
- S Vidal
- S Vidal
- SA Miller
- Shea Ping Yip
- SL Kunkel
- SP Yip
- T Abe
- VP Puzyrev
- VS Chan
- W Liu
- Wa Sang Wong
- Wai Man Lai
- WC Yam
- Wing Cheong Yam
- World Health Organisation
- WW Stead
- WW Stead
- X Ma
- X Ma
- Y Benjamini
- Y Benjamini
- Y Ueno
- YS Liaw
- Publication venue
- BioMed Central
- Publication date
- 01/01/2007
- Field of study
BACKGROUND: Host genetic factors are important determinants in tuberculosis (TB). The SLC11A1 (or NRAMP1) gene has been studied extensively for genetic association with TB, but with inconsistent findings. In addition, no study has yet looked into the effect of sex and age on the relationship between SLC11A1 polymorphisms and TB. METHODS: A case-control study was conducted. In total, 278 pulmonary TB patients and 282 sex- and age-matched controls without TB were recruited. All subjects were ethnic Chinese. On the basis of linkage disequilibrium pattern, three genetic markers from SLC11A1 and one from the nearby IL8RB locus were selected and examined for association with TB susceptibility. These markers were genotyped using single strand conformation polymorphism analysis or fragment analysis of amplified products. RESULTS: Statistically significant differences in allele (P = 0.0165, OR = 1.51) and genotype (P = 0.0163, OR = 1.59) frequencies of the linked markers SLC6a/b (classically called D543N and 3'UTR) of the SLC11A1 locus were found between patients and controls. With stratification by sex, positive associations were identified in the female group for both allele (P = 0.0049, OR = 2.54) and genotype (P = 0.0075, OR = 2.74) frequencies. With stratification by age, positive associations were demonstrated in the young age group (age ≤65 years) for both allele (P = 0.0047, OR = 2.52) and genotype (P = 0.0031, OR = 2.92) frequencies. All positive findings remained significant even after correction for multiple comparisons. No significant differences were noted in either the male group or the older age group. No significant differences were found for the other markers (one SLC11A1 marker and one IL8RB marker) either. CONCLUSION: This study confirmed the association between SLC11A1 and TB susceptibility and demonstrated for the first time that the association was restricted to females and the young age group
Cancer stem cell metabolism: A potential target for cancer therapy
- Author
- A Annibaldi
- A Serafino
- A Viale
- Abhijeet Deshmukh
- AE Greijer
- Arun Dharmarajan
- AS Silva
- B Bao
- BA Reynolds
- BC Fuchs
- CC Wykoff
- CH Jung
- CJ Yeom
- CM Fillmore
- CM Martin
- CR Hill
- CT Hensley
- CV Dang
- CY Chang
- D Bonnet
- D Daye
- D Lopez
- D Nakada
- DJ Ceradini
- DR Wise
- E Dazert
- E Fox
- E Monzani
- EA Newsholme
- ED Lagadinou
- EK Rofstad
- EM Kemper
- ES Dyer
- F Tomao
- Frank Arfuso
- G Mattheolabakis
- GL Semenza
- GM Shi
- H Lou
- H Sawayama
- HT Cheong
- I Abraham
- I Samudio
- J Lubelski
- J Zhou
- JL Wike-Hooley
- JM Adams
- JM Heddleston
- JR Griffiths
- K Chen
- K Inoki
- K Michalak
- K Moitra
- Kedar Deshpande
- KM Bailey
- KM Comerford
- KS Sastry
- L Romero-Ramirez
- LC Tu
- LM Phan
- LM Shelton
- M Al-Hajj
- M Bi
- M Janiszewska
- M Konopleva
- M Olempska
- M Peiris-Pages
- M Skrtic
- M Villalba
- M Wu
- MA Carducci
- MG Vander Heiden
- MH Raaijmakers
- MJ Holness
- MM Ho
- MW Dewhirst
- N Hammoudi
- NN Danial
- NY Frank
- P Kakarla
- P Kannan
- P Krishnamurthy
- P Sancho
- P Sancho
- Philip Newsholme
- PP Hsu
- PP Liu
- PS Winter
- R Kim
- R Moreno-Sánchez
- R Palorini
- R Siegel
- R Tang
- RB Hamanaka
- RE Moellering
- RJ Deberardinis
- RJ Gillies
- RJ Kathawala
- RJ Shaw
- RS Hundal
- RW Robey
- S Godar
- S Ma
- S Warrier
- S Yuan
- SC Dolfi
- SH Sahlberg
- SK Singh
- SV Ambudkar
- T Brabletz
- T Moriyama
- T Schatton
- U Karsten
- U Tidefelt
- W Lu
- W Zhou
- WH Wilson
- X-C Huang
- XQ Ye
- Y Liu
- Y Matsuda
- Y Zhao
- YH Wang
- Z Madjd
- Z Shi
- Publication venue
- 'Springer Science and Business Media LLC'
- Publication date
- 01/01/2016
- Field of study
© 2016 The Author(s). Cancer Stem cells (CSCs) are a unipotent cell population present within the tumour cell mass. CSCs are known to be highly chemo-resistant, and in recent years, they have gained intense interest as key tumour initiating cells that may also play an integral role in tumour recurrence following chemotherapy. Cancer cells have the ability to alter their metabolism in order to fulfil bio-energetic and biosynthetic requirements. They are largely dependent on aerobic glycolysis for their energy production and also are associated with increased fatty acid synthesis and increased rates of glutamine utilisation. Emerging evidence has shown that therapeutic resistance to cancer treatment may arise due to dysregulation in glucose metabolism, fatty acid synthesis, and glutaminolysis. To propagate their lethal effects and maintain survival, tumour cells alter their metabolic requirements to ensure optimal nutrient use for their survival, evasion from host immune attack, and proliferation. It is now evident that cancer cells metabolise glutamine to grow rapidly because it provides the metabolic stimulus for required energy and precursors for synthesis of proteins, lipids, and nucleic acids. It can also regulate the activities of some of the signalling pathways that control the proliferation of cancer cells. This review describes the key metabolic pathways required by CSCs to maintain a survival advantage and highlights how a combined approach of targeting cellular metabolism in conjunction with the use of chemotherapeutic drugs may provide a promising strategy to overcome therapeutic resistance and therefore aid in cancer therapy
Cancer Biomarker Discovery: The Entropic Hallmark
- Author
- A Alcaraz
- A Arabzadeh
- A Bagri
- A Balbin
- A Bantis
- A Ben-Naim
- A Bolander
- A Buhmeida
- A Burykin
- A Chetcuti
- A de Baey
- A Efeyan
- A Eroglu
- A Fabarius
- A Forget
- A Fujita
- A Geirsson
- A Geirsson
- A Geirsson
- A Guffanti
- A Hugel
- A Inoue
- A Isidoro
- A Jang
- A Kollara
- A Kuzmanov
- A Lundqvist
- A Maria McCrohan
- A Marreiros
- A Mendes
- A Mitra
- A Nassar
- A Petitjean
- A Petitjean
- A Pinzon-Charry
- A Richmond
- A Roesch
- A Santinelli
- A Schmidt
- A Shapiro
- A Sreekumar
- A Takeno
- A Urbanucci
- A Winter
- A Yemelyanov
- AA Babiker
- AA Babiker
- AA Tarhini
- AC Dudley
- AD Judge
- AD Ortega
- AG Puebla-Mora
- AH Fox
- AH Fox
- AI Riker
- AJ Liu
- AK Meeker
- AK Panigrahi
- AK Witkiewicz
- AL Cheung
- AL Fachin
- AL Gartel
- AL Okorokov
- AM Craig
- AM Lena
- AM Levin
- AM Tokes
- AP South
- AS Payne
- AS Verkman
- AS Verkman
- AS Verkman
- AS Wierzbicki
- AV Skamrov
- AW Maniccia
- B Cinar
- B Gao
- B Ouyang
- B Ren
- B Ryu
- B Tycko
- B Vukovic
- B Wu
- B Wu
- BD Dimitrov
- BF Gilmore
- BF Goncalves
- BF Skinnider
- BK Zehentner
- BL Adamsen
- BL de Groot
- BR Johnson
- BR Middleman
- BS Strauss
- C Adami
- C Bastide
- C Cao
- C Cybulski
- C Haqq
- C Hayashi
- C Hornig
- C Ivorra
- C Kumar-Sinha
- C Langner
- C Magi-Galluzzi
- C Magi-Galluzzi
- C Muller-Tidow
- C Nessler-Menardi
- C Nunez
- C Pequeux
- C Rajesh
- C Xu
- C Yuli
- CD Sturgis
- CD Truong
- CG Pham
- CG Rogers
- CG Rogers
- CJ Hutchison
- CJ Pronk
- CK Mavis
- CL Stewart
- CL Wu
- CM Caves
- CM Caves
- CM Clemson
- CM Perou
- CP Gray
- CP Gray
- CP Gray
- CR Mangahas
- CS Bond
- D Acehan
- D Bonn
- D Denhardt
- D Fu
- D Gilley
- D Gisselsson
- D Hanahan
- D Javelaud
- D Kroepfl
- D Lodygin
- D Maraldo
- D Ouazia
- D Scadden
- D Wang
- D Wang
- D Wang
- D Weckermann
- D Wodarz
- DA Shoskes
- DA Tennant
- DE Hansel
- DF Gleason
- DG Albertson
- DG Tenen
- DH Palmer
- DJ Darley
- DJ Grignon
- DJ Smit
- DJ Son
- DJ Wong
- DJC MacKay
- DK Koynova
- DK Lee
- DM Kube
- DM Peehl
- DR Wonsey
- DS Perez
- DV Makarov
- DV Makarov
- E Beitz
- E Beitz
- E Cabuy
- E Fagiani
- E Flannery
- E Geissinger
- E Kikuchi
- E Kruse
- E Sbisa
- E Sprecher
- E Tajkhorshid
- EA Ostrander
- EH Feng
- EH Feng
- EM Rosen
- EM Rosen
- EM Schaeffer
- EO Kehinde
- ET Canepa
- EV Vykhovanets
- EY Lin
- EY Tan
- F Bai
- F Bonetto
- F Boralevi
- F Buback
- F Colotta
- F Langer
- F Saad
- F Tas
- F Yang
- FS Krause
- G Benga
- G Bhanot
- G Blandino
- G Buchanan
- G Kaic
- G Kristiansen
- G Kristiansen
- G Kroemer
- G Murugaiyan
- G Petrovics
- G Pirtskhalaishvili
- G Russo
- G Sardana
- G Stirling
- G Tscheudschilsuren
- G Untergasser
- G Venkataraman
- G von Figura
- GA Jarvis
- GE Crooks
- GF Azzone
- GF Ding
- GL Shen-Ong
- GP Bienert
- GQ Daley
- GR Ribeiro
- GS Palapattu
- GV Glinsky
- GV Glinsky
- H Chang-zheng
- H Chen
- H Dzojic
- H Gronberg
- H Haghnegahdar
- H Harant
- H Harlin
- H Kida
- H Li
- H Liu
- H Miyake
- H Nemoto
- H Ostrer
- H Pilch
- H Rangaswami
- H Rangaswami
- H Rangaswami
- H Ren
- H Roca
- H Roca
- H Samaratunga
- H Savli
- H Schlechte
- H Schoder
- H Sheng
- H Sunwoo
- H Then
- H Wang
- H Wang
- HC Lee
- HG Thomas
- HJ Worman
- HL Winfield
- HS Leff
- HT Quan
- HY Vu
- HZ Zhang
- I Agalliu
- I Bae
- I Ben-Porath
- I Grosse
- I Haritoglou
- I Leav
- I Marinescu
- I Moll
- I Sakai
- IE Wertz
- IK Lim
- IL Tourkova
- IM Bachmann
- IM Shih
- IW Reiniger
- IY Dodin
- J Bartkova
- J Edwards
- J Fellenberg
- J Gille
- J Graells
- J Jaeger
- J Johnson
- J Lammerding
- J Laoukili
- J Lapointe
- J Linden
- J Luo
- J Luo
- J Luo
- J Mandelin
- J Melamed
- J Moser
- J Rangel
- J Rapley
- J Reimand
- J Ruiz-Ederra
- J Schmitz
- J Schultz
- J Soikkeli
- J Stewart
- J Suh
- J Walter-Yohrling
- J Wang
- J Wang
- J Weiske
- J Xu
- J Yang
- J Yang
- J Zheng
- JA Camargo
- JA Douglas
- JA Hackett
- JA Karam
- JA McGrath
- JA McGrath
- JA McGrath
- JA McGrath
- JA Mobley
- JA Pettus
- JB Roche
- JC Cheville
- JD French
- JD Licchesi
- JD Owen
- JE Lai-Cheong
- JE Sleeman
- JE Wilusz
- JG Wen
- JH Lee
- JH Luo
- JJ Brey
- JJ Park
- JJ Raffoul
- JJ Tseng
- JJ Van den Oord
- JK Wahl 3rd
- JL Broers
- JL Herrmann
- JL Phillips
- JM Brandner
- JM Bridger
- JM Cummins
- JM Gonzalez
- JM Lizcano
- JM Muller
- JM Muller
- JM Parrondo
- JM Saffin
- JN Hutchinson
- JN Mubiru
- JN Mubiru
- JN Mubiru
- JP Arbitrario
- JP Clark
- JP Ehlers
- JP Fruehauf
- JP Struewing
- JR Cansino Alcaide
- JR McMillan
- JS Butler
- JS Butler
- JS Fang
- JS Hub
- JS Lee
- JS Mattick
- JS Mattick
- JS Mattick
- JT Bishoff
- JT Chang
- JW Denham
- K Armstrong
- K Fujita
- K Ishibashi
- K Ishitani
- K Kami
- K Kasuno
- K Kumagai
- K Mizutani
- K Namiki
- K Ruan
- K Ruan
- K Shilo
- K Sircar
- K Suzuki
- K Swisshelm
- K Takahara
- K Tani
- K Tokunaga
- K Tomkova
- K Trpkov
- K Yamada
- KA Tasken
- KA Zuhlke
- KH Hoffmann
- KH Tsui
- KL Nastiuk
- KL Simpson
- KL van Golen
- KN Ekdahl
- KP Porkka
- KR Ghani
- KV Prasanth
- L Capparuccia
- L Carlsson
- L Cheng
- L de Meis
- L Galluzzi
- L Kneisel
- L Lessard
- L Li
- L Moro
- L Moro
- L O'Regan
- L Packer
- L Pasini
- L True
- L Verdoucq
- L Verschooten
- L Xu
- L Zheng
- LA Diaz-Martinez
- LB Mora
- LE Clarke
- LE Finlan
- LF Brown
- LF Zerbini
- LL Chen
- LL Smith
- LL Smith
- LM Godsel
- LP Kunju
- M Aalamian-Matheis
- M Aghaei
- M Bakhanashvili
- M Boury-Jamot
- M Boury-Jamot
- M Buxade
- M Chellaiah
- M Cimitan
- M Craig
- M Dumas
- M Dundr
- M Esteller
- M Estler
- M Fabbri
- M Fukunaga-Kalabis
- M Fukunaga-Kalabis
- M Furuno
- M Gilbert
- M Hara-Chikuma
- M Hara-Chikuma
- M Hara-Chikuma
- M Hara-Chikuma
- M Hatzfeld
- M Hatzfeld
- M Heenen
- M Ismail
- M Karaivanov
- M Kashani-Sabet
- M Kato
- M Katoh
- M Kinoshita
- M Korabiowska
- M Kulesz-Martin
- M Lei
- M Lisovsky
- M Marko
- M Mellado
- M Mishima
- M Mockenhaupt
- M Moghaddami
- M Moghaddami
- M Nessling
- M Olsson
- M Onaitis
- M Parisi
- M Schostak
- M Tischkowitz
- M Traka
- M Tribus
- M Varma
- M Venoux
- M Watanabe
- M Yang
- M Yasui
- M Zhou
- M Zhou
- M Zhou
- MA Castro
- MA Chekmareva
- MA Chellaiah
- MA Ficazzola
- MA Halsey
- MA Roder
- MA Rubin
- MA Rubin
- MA Rubin
- MB Martens
- MB Rettig
- MC Caino
- MC Schumacher
- MD Desrosiers
- MD Duff
- MD Lloyd
- ME Blank
- ME Grossmann
- ME Grossmann
- ME Laski
- ME Morrison
- MF Fraga
- MH Vaarala
- MK Thomsen
- MK Thomsen
- MM Petrovic
- MM Rashid
- MR Rosner
- MS O'Connor
- MS Tretiakova
- MT Kalcioglu
- MT Ling
- MT Martin
- MT Sung
- MV Blagosklonny
- MY Lee
- N Bahamontes-Rosa
- N Donin
- N Konishi
- N Lapteva
- N Maeda
- N Nadiminty
- N Rabiau
- N Singh
- NG Hussein
- NI Andreyev
- NJ Fisch
- NV Whittock
- NV Whittock
- O Bettendorf
- O Canbolat
- O Itzhaki
- O Kwon
- OA Rosso
- OW Rokhlin
- P Agre
- P Agre
- P Agre
- P Agre
- P Allavena
- P Berthon
- P Boukamp
- P Cossu-Rocca
- P Dhawan
- P Dhawan
- P Duesberg
- P Duesberg
- P Duesberg
- P Hainaut
- P Ji
- P Karakiewicz
- P Koivisto
- P Roy-Burman
- P Rustin
- P Sini
- P Slijepcevic
- P Sotomayor
- P Sotomayor
- P Steigemann
- P Zhang
- Pablo Moscato
- PD Walden
- PG Gallagher
- PH Gumerlock
- PH Konig
- PI Nedvetsky
- PJ Gray Jr
- PJ Zielie
- PL Chang
- PM Lacal
- PM Lacal
- PM Mordaka
- PM Steijlen
- PT Landsberg
- PT Went
- PU Malmstrom
- PW Kuchel
- PW Lamberti
- Q Lu
- Q Zhou
- R Agarwal
- R Berretta
- R Birnie
- R Bordoni
- R Casalini
- R Das
- R Dobson
- R Halaban
- R Horuk
- R Klimek
- R Klimek
- R Kuefer
- R Lin
- R Mazzucchelli
- R Mukherjee
- R Radosavljevic
- R Rai
- R Sougrat
- RA Craven
- RA Hegele
- RA Sturm
- RA Weinberg
- RC Lee
- RD Ballal
- RD Loberg
- RD Loberg
- RD Loberg
- RD Loberg
- Regina Berretta
- RG Uzzo
- RJ Jin
- RJ Urban
- RJ Webster
- RL Boguslavsky
- RL Shattuck
- RMH Lopez-Ruiz
- RP Ciavarra
- RP Ciavarra
- RP Singh
- RS Weinstein
- RT Nitta
- RW DeVere White
- S Ashida
- S Brychtova
- S Cardinale
- S Das
- S Das
- S Dettwiler
- S Di Cesare
- S Doratiotto
- S Ersoy-Evans
- S Fan
- S Gagos
- S Ghosh
- S Gunia
- S Hanada
- S Hauptmann
- S Hiratsuka
- S Horsburgh
- S Huerta-Yepez
- S Jhavar
- S Jhavar
- S Kuwahara
- S Kyle
- S Landreville
- S Makovets
- S Morohashi
- S Olgac
- S Philip
- S Philip
- S Prislei
- S Rankin
- S Rankin
- S Satoh
- S Sur
- S Takahashi
- S Takahashi
- S Tancharoen
- S Verdier-Sevrain
- S Yeh
- S Zha
- S Zha
- S Zha
- SA Narod
- SH Lin
- SJ Yeung
- SK Kang
- SL Kong
- SL Zheng
- SM Edwards
- SM Ho
- SM Saparov
- SQ Xie
- SR Alonso
- SS Kadkol
- SV Setzer
- T Dalmay
- T Drewa
- T Hoevel
- T Ishiyama
- T Jamaspishvili
- T Kusumi
- T Litman
- T Ma
- T Mirtti
- T Murakami
- T Oyama
- T Schlomm
- T Schlomm
- T Sheridan
- T Shimizu
- T Sobolik-Delmaire
- T Sun
- T Takahashi
- T Takeda
- T Thornburg
- T Watanabe
- T Yu
- TB Lewis
- TD Prickett
- TH Ecke
- TJ McDonnell
- TJ Park
- TL Schmit
- TL Schmit
- TO Nielsen
- TR Mercer
- U Brinck
- U Graeven
- U Gruneberg
- UV Wesley
- V Ambros
- V Barak
- V Fradet
- V Molinie
- V Molinie
- V Molinie
- V Molinie
- V Molinie
- V Molinie
- V Pekovic
- V Rajaram
- V Samanna
- V Wessagowit
- V Winnepenninckx
- VD Acevedo
- VK Vanguri
- VL Verstraeten
- W Chen
- W Cosme-Blanco
- W Gade
- W Ritchie
- W Spirkl
- W Tettelbach
- W Wang
- W Weichert
- W Weichert
- W Weichert
- WB Isaacs
- WD Dunsmuir
- WE Naugler
- William C. S. Cho
- WK Kaufmann
- WK Lee
- X Chen
- X Cheng
- X Dong
- X Dong
- X Huang
- X Li
- X Liu
- X Liu
- X Meng
- X Ouyang
- X Tang
- X Wu
- X Zhang
- XJ Yang
- XQ Wang
- Y Bu
- Y Cai
- Y Deng
- Y Ito
- Y Katagiri
- Y Kozuka
- Y Lai
- Y Liu
- Y Lu
- Y Lu
- Y Ohyama
- Y Shav-Tal
- Y Sugiyama
- Y Sun
- Y Wang
- Y Wang
- Y Wu
- Y Yamamoto
- Y Yang
- Y Zhou
- YB Lim
- YC Chao
- YJ Ko
- YL Liu
- YN Liu
- YT Sasaki
- YT Sasaki
- YU Katagiri
- Z Jiang
- Z Jiang
- Z Jiang
- Z Jiang
- Z Jiang
- Z Rudzki
- ZA Dotan
- ZR Yurkovetsky
- Publication venue
- Public Library of Science
- Publication date
- 01/01/2010
- Field of study
Background: It is a commonly accepted belief that cancer cells modify their transcriptional state during the progression of the disease. We propose that the progression of cancer cells towards malignant phenotypes can be efficiently tracked using high-throughput technologies that follow the gradual changes observed in the gene expression profiles by employing Shannon's mathematical theory of communication. Methods based on Information Theory can then quantify the divergence of cancer cells' transcriptional profiles from those of normally appearing cells of the originating tissues. The relevance of the proposed methods can be evaluated using microarray datasets available in the public domain but the method is in principle applicable to other high-throughput methods. Methodology/Principal Findings: Using melanoma and prostate cancer datasets we illustrate how it is possible to employ Shannon Entropy and the Jensen-Shannon divergence to trace the transcriptional changes progression of the disease. We establish how the variations of these two measures correlate with established biomarkers of cancer progression. The Information Theory measures allow us to identify novel biomarkers for both progressive and relatively more sudden transcriptional changes leading to malignant phenotypes. At the same time, the methodology was able to validate a large number of genes and processes that seem to be implicated in the progression of melanoma and prostate cancer. Conclusions/Significance: We thus present a quantitative guiding rule, a new unifying hallmark of cancer: the cancer cell's transcriptome changes lead to measurable observed transitions of Normalized Shannon Entropy values (as measured by high-throughput technologies). At the same time, tumor cells increment their divergence from the normal tissue profile increasing their disorder via creation of states that we might not directly measure. This unifying hallmark allows, via the the Jensen-Shannon divergence, to identify the arrow of time of the processes from the gene expression profiles, and helps to map the phenotypical and molecular hallmarks of specific cancer subtypes. The deep mathematical basis of the approach allows us to suggest that this principle is, hopefully, of general applicability for other diseases
Measurement and interpretation of same-sign W boson pair production in association with two jets in pp collisions at s = 13 TeV with the ATLAS detector
- Author
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- Åkesson TPA
- Öncel ÖO
- Ženiš T
- Živković L
- Publication venue
- Springer Nature
- Publication date
- 01/04/2024
- Field of study
This paper presents the measurement of fducial and diferential cross sections for both the inclusive and electroweak production of a same-sign W-boson pair in association with two jets (W±W±jj) using 139 fb−1 of proton-proton collision data recorded at a centre-of-mass energy of √s = 13 TeV by the ATLAS detector at the Large Hadron Collider. The analysis is performed by selecting two same-charge leptons, electron or muon, and at least two jets with large invariant mass and a large rapidity diference. The measured fducial cross sections for electroweak and inclusive W±W±jj production are 2.92 ± 0.22 (stat.) ± 0.19 (syst.)fb and 3.38±0.22 (stat.)±0.19 (syst.)fb, respectively, in agreement with Standard Model predictions. The measurements are used to constrain anomalous quartic gauge couplings by extracting 95% confdence level intervals on dimension-8 operators. A search for doubly charged Higgs bosons H±± that are produced in vector-boson fusion processes and decay into a same-sign W boson pair is performed. The largest deviation from the Standard Model occurs for an H±± mass near 450 GeV, with a global signifcance of 2.5 standard deviations
Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)1.
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- Abdel-Aziz AK
- Abdelfatah S
- Abdellatif M
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- Zorzano A
- Zou W
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- Zou Z
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- Zwerschke W
- Álvarez ÉMC
- Ávalos Y
- Önal G
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- Čolić M
- Đokić J
- Žerovnik E
- Publication venue
- 'Informa UK Limited'
- Publication date
- 01/01/2021
- Field of study
In 2008, we published the first set of guidelines for standardizing research in autophagy. Since then, this topic has received increasing attention, and many scientists have entered the field. Our knowledge base and relevant new technologies have also been expanding. Thus, it is important to formulate on a regular basis updated guidelines for monitoring autophagy in different organisms. Despite numerous reviews, there continues to be confusion regarding acceptable methods to evaluate autophagy, especially in multicellular eukaryotes. Here, we present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes. These guidelines are not meant to be a dogmatic set of rules, because the appropriateness of any assay largely depends on the question being asked and the system being used. Moreover, no individual assay is perfect for every situation, calling for the use of multiple techniques to properly monitor autophagy in each experimental setting. Finally, several core components of the autophagy machinery have been implicated in distinct autophagic processes (canonical and noncanonical autophagy), implying that genetic approaches to block autophagy should rely on targeting two or more autophagy-related genes that ideally participate in distinct steps of the pathway. Along similar lines, because multiple proteins involved in autophagy also regulate other cellular pathways including apoptosis, not all of them can be used as a specific marker for bona fide autophagic responses. Here, we critically discuss current methods of assessing autophagy and the information they can, or cannot, provide. Our ultimate goal is to encourage intellectual and technical innovation in the field
Combination of searches for heavy spin-1 resonances using 139 fb−1 of proton-proton collision data at s = 13 TeV with the ATLAS detector
- Author
- Aad G
- Aakvaag E
- Abbott B
- Abeling K
- Abicht NJ
- Abidi SH
- Aboelela M
- Aboulhorma A
- Abramowicz H
- Abreu H
- Abulaiti Y
- Acharya BS
- Ackermann A
- Adam Bourdarios C
- Adamczyk L
- Addepalli SV
- Addison MJ
- Adelman J
- Adiguzel A
- Adye T
- Affolder AA
- Afik Y
- Agaras MN
- Agarwala J
- Aggarwal A
- Agheorghiesei C
- Ahmad A
- Ahmadov F
- Ahmed WS
- Ahuja S
- Ai X
- Aielli G
- Aikot A
- Ait Tamlihat M
- Aitbenchikh B
- Aizenberg I
- Akbiyik M
- Akimov AV
- Akiyama D
- Akolkar NN
- Aktas S
- Al Khoury K
- Alberghi GL
- Albert J
- Albicocco P
- Albouy GL
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- Zhemchugov A
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- Zou W
- Zwalinski L
- Åkesson TPA
- Öncel ÖO
- Ženiš T
- Živković L
- Publication venue
- Springer Nature
- Publication date
- 19/04/2024
- Field of study
A combination of searches for new heavy spin-1 resonances decaying into different pairings of W, Z, or Higgs bosons, as well as directly into leptons or quarks, is presented. The data sample used corresponds to 139 fb−1 of proton-proton collisions at
= 13 TeV collected during 2015–2018 with the ATLAS detector at the CERN Large Hadron Collider. Analyses selecting quark pairs (qq, bb,
, and tb) or third-generation leptons (τν and ττ) are included in this kind of combination for the first time. A simplified model predicting a spin-1 heavy vector-boson triplet is used. Cross-section limits are set at the 95% confidence level and are compared with predictions for the benchmark model. These limits are also expressed in terms of constraints on couplings of the heavy vector-boson triplet to quarks, leptons, and the Higgs boson. The complementarity of the various analyses increases the sensitivity to new physics, and the resulting constraints are stronger than those from any individual analysis considered. The data exclude a heavy vector-boson triplet with mass below 5.8 TeV in a weakly coupled scenario, below 4.4 TeV in a strongly coupled scenario, and up to 1.5 TeV in the case of production via vector-boson fusion
First Measurement of the Hubble Constant from a Dark Standard Siren using the Dark Energy Survey Galaxies and the LIGO/Virgo Binary-Black-hole Merger GW170814
- Author
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- Publication venue
- IOP PUBLISHING LTD
- Publication date
- 01/05/2019
- Field of study
We present a multi-messenger measurement of the Hubble constant H 0 using the binary–black-hole merger GW170814 as a standard siren, combined with a photometric redshift catalog from the Dark Energy Survey (DES). The luminosity distance is obtained from the gravitational wave signal detected by the Laser Interferometer Gravitational-Wave Observatory (LIGO)/Virgo Collaboration (LVC) on 2017 August 14, and the redshift information is provided by the DES Year 3 data. Black hole mergers such as GW170814 are expected to lack bright electromagnetic emission to uniquely identify their host galaxies and build an object-by-object Hubble diagram. However, they are suitable for a statistical measurement, provided that a galaxy catalog of adequate depth and redshift completion is available. Here we present the first Hubble parameter measurement using a black hole merger. Our analysis results in H0=75−32+40kms−1Mpc−1, which is consistent with both SN Ia and cosmic microwave background measurements of the Hubble constant. The quoted 68% credible region comprises 60% of the uniform prior range [20, 140] km s−1 Mpc−1, and it depends on the assumed prior range. If we take a broader prior of [10, 220] km s−1 Mpc−1, we find {H}_{0 {78}_{-24}^{+96}\,\mathrm{km}\,{{\rm{s}}}^{-1}\,{\mathrm{Mpc}}^{-1} (57% of the prior range). Although a weak constraint on the Hubble constant from a single event is expected using the dark siren method, a multifold increase in the LVC event rate is anticipated in the coming years and combinations of many sirens will lead to improved constraints on H 0
Combination and summary of ATLAS dark matter searches interpreted in a 2HDM with a pseudo-scalar mediator using 139 fb−1 of <mml:math xmlns:mml="http://www.w3.org/1998/Math/MathML" altimg="si157.svg"><mml:mrow><mml:msqrt><mml:mrow><mml:mi>s</mml:mi></mml:mrow></mml:msqrt><mml:mo linebreak="goodbreak" linebreakstyle="after">=</mml:mo><mml:mn>13</mml:mn></mml:mrow></mml:math> TeV pp collision data
- Author
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- Abbott B
- Abeling K
- Abicht NJ
- Abidi SH
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- Abreu H
- Abulaiti Y
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- Yamaguchi Y
- Yamashita E
- Yamauchi H
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- Yao W-M
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- Yeletskikh I
- Yeo BK
- Yexley MR
- Yin P
- Yorita K
- Younas S
- Young C
- Young CJS
- Yu Y
- Yuan M
- Yuan R
- Yue L
- Zaazoua M
- Zabinski B
- Zaid E
- Zakareishvili T
- Zakharchuk N
- Zambito S
- Zang J
- Zanzi D
- Zaplatilek O
- Zeitnitz C
- Zeng H
- Zeng JC
- Zenger DT
- Zenin O
- Zenz S
- Zerradi S
- Zerwas D
- Zhai M
- Zhang B
- Zhang DF
- Zhang J
- Zhang J
- Zhang K
- Zhang L
- Zhang P
- Zhang R
- Zhang S
- Zhang T
- Zhang X
- Zhang X
- Zhang Y
- Zhang Y
- Zhang Z
- Zhang Z
- Zhao H
- Zhao P
- Zhao T
- Zhao Y
- Zhao Z
- Zhemchugov A
- Zheng J
- Zheng K
- Zheng X
- Zheng Z
- Zhong D
- Zhou B
- Zhou H
- Zhou N
- Zhou Y
- Zhu CG
- Zhu J
- Zhu Y
- Zhu Y
- Zhuang X
- Zhukov K
- Zhulanov V
- Zimine NI
- Zinsser J
- Ziolkowski M
- Zoccoli A
- Zoch K
- Zorbas TG
- Zormpa O
- Zou W
- Zwalinski L
- Åkesson TPA
- Öncel ÖO
- Ženiš T
- Živković L
- Publication venue
- Elsevier BV
- Publication date
- 01/07/2024
- Field of study
Results from a wide range of searches targeting different experimental signatures with and without missing transverse momentum (
) are used to constrain a Two–Higgs-Doublet Model (2HDM) with an additional pseudo-scalar mediating the interaction between ordinary and dark matter (2HDM + a). The analyses use up to 139 fb−1 of proton–proton collision data at a centre-of-mass energy
TeV recorded with the ATLAS detector at the Large Hadron Collider during 2015–2018. The results from three of the most sensitive searches are combined statistically. These searches target signatures with large
and a leptonically decaying Z boson; large
and a Higgs boson decaying to bottom quarks; and production of charged Higgs bosons in final states with top and bottom quarks, respectively. Constraints are derived for several common and new benchmark scenarios in the 2HDM + a
Search for dark photons in rare Z boson decays with the ATLAS detector
- Author
- Aad G
- Abbott B
- Abeling K
- Abicht NJ
- Abidi SH
- Aboulhorma A
- Abramowicz H
- Abreu H
- Abulaiti Y
- Abusleme Hoffman AC
- Acharya BS
- Adam Bourdarios C
- Adamczyk L
- Adamek L
- Addepalli SV
- Addison MJ
- Adelman J
- Adiguzel A
- Adye T
- Affolder AA
- Afik Y
- Agaras MN
- Agarwala J
- Aggarwal A
- Agheorghiesei C
- Ahmad A
- Ahmadov F
- Ahmed WS
- Ahuja S
- Ai X
- Aielli G
- Ait Tamlihat M
- Aitbenchikh B
- Aizenberg I
- Akbiyik M
- Akimov AV
- Akiyama D
- Akolkar NN
- Al Khoury K
- Alberghi GL
- Albert J
- Albicocco P
- Albouy GL
- Alderweireldt S
- Aleksa M
- Aleksandrov IN
- Alexa C
- Alexopoulos T
- Alfonsi A
- Alfonsi F
- Algren M
- Alhroob M
- Ali B
- Ali HMJ
- Ali S
- Alibocus SW
- Aliev M
- Alimonti G
- Alkakhi W
- Allaire C
- Allbrooke BMM
- Allen JF
- Allendes Flores CA
- Allport PP
- Aloisio A
- Alonso F
- Alpigiani C
- Alvarez Estevez M
- Alvarez Fernandez A
- Alves Cardoso M
- Alviggi MG
- Aly M
- Amaral Coutinho Y
- Ambler A
- Amelung C
- Amerl M
- Ames CG
- Amidei D
- Amor Dos Santos SP
- Amos KR
- Ananiev V
- Anastopoulos C
- Andeen T
- Anders JK
- Andrean SY
- Andreazza A
- Angelidakis S
- Angerami A
- Anisenkov AV
- Annovi A
- Antel C
- Anthony MT
- Antipov E
- Antonelli M
- Antrim DJA
- Anulli F
- Aoki M
- Aoki T
- Aparisi Pozo JA
- Aparo MA
- Aperio Bella L
- Appelt C
- Apyan A
- Aranzabal N
- Arcangeletti C
- Arce ATH
- Arena E
- Arguin J-F
- Argyropoulos S
- Arling J-H
- Arnaez O
- Arnold H
- Arrubarrena Tame ZP
- Artoni G
- Asada H
- Asai K
- Asai S
- Asbah NA
- Assahsah J
- Assamagan K
- Astalos R
- Atashi S
- Atkin RJ
- Atkinson M
- ATLAS Collaboration
- Atlay NB
- Atmani H
- Atmasiddha PA
- Augsten K
- Auricchio S
- Auriol AD
- Austrup VA
- Avolio G
- Axiotis K
- Azuelos G
- Babal D
- Bachacou H
- Bachas K
- Bachiu A
- Backman F
- Badea A
- Bagnaia P
- Bahmani M
- Bailey AJ
- Bailey VR
- Baines JT
- Baines L
- Bakalis C
- Baker OK
- Bakos E
- Bakshi Gupta D
- Balasubramanian R
- Baldin EM
- Balek P
- Ballabene E
- Balli F
- Baltes LM
- Balunas WK
- Balz J
- Banas E
- Bandieramonte M
- Bandyopadhyay A
- Bansal S
- Barak L
- Barakat M
- Barberio EL
- Barberis D
- Barbero M
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- Zamora Saa JA
- Zang J
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- Zeitnitz C
- Zeng H
- Zeng JC
- Zenger DT
- Zenin O
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- Zerradi S
- Zerwas D
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- Zhang Z
- Zhang Z
- Zhao H
- Zhao P
- Zhao T
- Zhao Y
- Zhao Z
- Zhemchugov A
- Zheng K
- Zheng X
- Zheng Z
- Zhong D
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- Zhou Y
- Zhu CG
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- Zhu Y
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- Zhuang X
- Zhukov K
- Zhulanov V
- Zimine NI
- Zinsser J
- Ziolkowski M
- Zoccoli A
- Zoch K
- Zorbas TG
- Zormpa O
- Zou W
- Zwalinski L
- Åkesson TPA
- Öncel ÖO
- Ženiš T
- Živković L
- Publication venue
- American Physical Society
- Publication date
- 01/01/2023
- Field of study
A search for events with a dark photon produced in association with a dark Higgs boson via rare decays of the standard model
Z
boson is presented, using
139
fb
−
1
of
√
s
=
13
TeV
proton-proton collision data recorded by the ATLAS detector at the Large Hadron Collider. The dark boson decays into a pair of dark photons, and at least two of the three dark photons must each decay into a pair of electrons or muons, resulting in at least two same-flavor opposite-charge lepton pairs in the final state. The data are found to be consistent with the background prediction, and upper limits are set on the dark photon’s coupling to the dark Higgs boson times the kinetic mixing between the standard model photon and the dark photon,
α
D
ϵ
2
, in the dark photon mass range of [5, 40] GeV except for the
Υ
mass window [8.8, 11.1] GeV. This search explores new parameter space not previously excluded by other experiments
- …