3 research outputs found
Canagliflozin and Renal Outcomes in Type 2 Diabetes and Nephropathy
- Author
- Abdallah J
- Abdul Ghani R
- Abdul Kadir K
- Abdullah R
- Abejuela Z
- Abouglila K
- Abraham O
- Abrahamsen I
- Abraira Munoz Ld
- Abramowitz M
- Abusnana S
- Accini Mendoza Jl
- Acosta I
- Agafyina A
- Agarwal R
- Agarwal Rajiv
- Aggarwal N
- Agra J
- Ahmed F
- Ahuad Guerrero Ra
- Aiello J
- Ainsworth P
- Aizenberg D
- Akiyama H
- Akright L
- Akyea-Djamson A
- Al-Karadsheh A
- Alamartine E
- Alappan R
- Albisu Jp
- Ali N
- Ali Z
- Alicic R
- Allison Dc
- Alvarez Garcia P
- Alvarisqueta A
- Alves da Costa Fa
- Amerena J
- Amigo A
- Amodeo C
- Andreeva V
- Andreotti Turatti La
- Angelescu Lm
- Angelova A
- Anghel V
- Antic S
- Aoki H
- Aquitania G
- Arauz-Pacheco C
- Arcos E
- Arfeen S
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- Arutchelvam V
- Arvind M
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- Bentley-Lewis R
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- Bernardo M
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- Bijata-Bronisz R
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- Biscoveanu M
- Bompoint Severine
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- Bordonava A
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- Botsyurko V
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- Brusco O
- Buganova I
- Bull Scott
- Burst V
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- Busegeanu Mm
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- Capuano George
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- Cartasegna Lr
- Castellino P
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- Chae Dw
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- Chapin L
- Charytan David M
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- Che Yusof Md
- Cheifetz A
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- Cherney D 3rd
- Chertow G
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- Chilito A
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- Chuang Lm
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- Commendatore V
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- Culak J
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- Cunha Borges Jl
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- Vyasam Rc
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- Wadud K
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- Wakeling J
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- Watanabe T
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- Yao C
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- Yu X
- Yumita W
- Yupanqui H
- Zaidman Cj
- Zalunardo N
- Zamarripa Escobedo R
- Zanata Petry Tb
- Zanella Mt
- Zaniewski-Singh M.
- Zanoli L
- Zaoui P
- Zateyshchikova A
- Zateyshshikov D
- Zaviriukha V
- Zemek S
- Zhang H
- Zhang Hong
- Zhao M
- Zheng H
- Zinman B
- Zinman Bernard
- Zlova T
- Zsom M
- Zub L
- Zykova T
- Publication venue
- 'Massachusetts Medical Society'
- Publication date
- 01/01/2019
- Field of study
BACKGROUND Type 2 diabetes mellitus is the leading cause of kidney failure worldwide, but few effective long-term treatments are available. In cardiovascular trials of inhibitors of sodiumâglucose cotransporter 2 (SGLT2), exploratory results have suggested that such drugs may improve renal outcomes in patients with type 2 diabetes. METHODS In this double-blind, randomized trial, we assigned patients with type 2 diabetes and albuminuric chronic kidney disease to receive canagliflozin, an oral SGLT2 inhibitor, at a dose of 100 mg daily or placebo. All the patients had an estimated glomerular filtration rate (GFR) of 30 to 300 to 5000) and were treated with reninâangiotensin system blockade. The primary outcome was a composite of end-stage kidney disease (dialysis, transplantation, or a sustained estimated GFR of <15 ml per minute per 1.73 m 2), a doubling of the serum creatinine level, or death from renal or cardiovascular causes. Prespecified secondary outcomes were tested hierarchically. RESULTS The trial was stopped early after a planned interim analysis on the recommendation of the data and safety monitoring committee. At that time, 4401 patients had undergone randomization, with a median follow-up of 2.62 years. The relative risk of the primary outcome was 30% lower in the canagliflozin group than in the placebo group, with event rates of 43.2 and 61.2 per 1000 patient-years, respectively (hazard ratio, 0.70; 95% confidence interval [CI], 0.59 to 0.82; P=0.00001). The relative risk of the renal-specific composite of end-stage kidney disease, a doubling of the creatinine level, or death from renal causes was lower by 34% (hazard ratio, 0.66; 95% CI, 0.53 to 0.81; P<0.001), and the relative risk of end-stage kidney disease was lower by 32% (hazard ratio, 0.68; 95% CI, 0.54 to 0.86; P=0.002). The canagliflozin group also had a lower risk of cardiovascular death, myocardial infarction, or stroke (hazard ratio, 0.80; 95% CI, 0.67 to 0.95; P=0.01) and hospitalization for heart failure (hazard ratio, 0.61; 95% CI, 0.47 to 0.80; P<0.001). There were no significant differences in rates of amputation or fracture. CONCLUSIONS In patients with type 2 diabetes and kidney disease, the risk of kidney failure and cardiovascular events was lower in the canagliflozin group than in the placebo group at a median follow-up of 2.62 years
Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)1.
- Author
- Abdel-Aziz AK
- Abdelfatah S
- Abdellatif M
- Abdoli A
- Abel S
- Abeliovich H
- Abildgaard MH
- Abudu YP
- Acevedo-Arozena A
- Adamopoulos IE
- Adeli K
- Adolph TE
- Adornetto A
- Aflaki E
- Agam G
- Agarwal A
- Aggarwal BB
- Agnello M
- Agostinis P
- Agrewala JN
- Agrotis A
- Aguilar PV
- Ahmad ST
- Ahmed ZM
- Ahumada-Castro U
- Aits S
- Aizawa S
- Akkoc Y
- Akoumianaki T
- Akpinar HA
- Al-Abd AM
- Al-Akra L
- Al-Gharaibeh A
- Alaoui-Jamali MA
- Alberti S
- Alcocer-GĂłmez E
- Alessandri C
- Ali M
- Alim Al-Bari MA
- Aliwaini S
- Alizadeh J
- Almacellas E
- Almasan A
- Alonso A
- Alonso GD
- Altan-Bonnet N
- Altieri DC
- Alves da Costa C
- Alves S
- Alzaharna MM
- Amadio M
- Amantini C
- Amaral C
- Ambrosio S
- Amer AO
- Ammanathan V
- An Z
- Andersen SU
- Andrabi SA
- Andrade-Silva M
- Andres AM
- Angelini S
- Ann D
- Anozie UC
- Ansari MY
- Antas P
- Antebi A
- AntĂłn Z
- Anwar T
- Apetoh L
- Apostolova N
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- Araki Y
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- Araya J
- AraĂșjo WL
- Arden C
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- Arimoto H
- Ariosa AR
- Armstrong-James D
- Arnauné-Pelloquin L
- Aroca A
- Arroyo DS
- Arsov I
- Artero R
- Arévalo M-A
- Asaro DML
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- Ashrafizadeh M
- Ashur-Fabian O
- Atanasov AG
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- Auberger P
- Auner HW
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- Avin-Wittenberg T
- Aydin Y
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- Backer JM
- Backues SK
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- Baehrecke EH
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- Vitale I
- Vocadlo DJ
- Voitsekhovskaja OV
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- Vooijs MA
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- Vucicevic L
- Wade-Martins R
- Waguri S
- Waite KA
- Wakatsuki S
- Walker DW
- Walker MJ
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- Wang Q
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- Wheatley SP
- Whitton JL
- Whitworth AJ
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- Wildenberg ME
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- Wilkinson S
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- Winsor NJ
- Witkin SS
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- Yakhine-Diop SMS
- Yamaguchi M
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- Yazdankhah M
- Yen PM
- Yi C
- Yi-Ren Hong C-WH
- Yin X-M
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- Yiu SPT
- Yoo YH
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- Yousefi B
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- Zanella I
- Zang QS
- Zanivan S
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- Zaragoza P
- Zarbalis KS
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- Zarrouk A
- Zeitlin SO
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- Zhang J-P
- Zhang KYB
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- Zhang X
- Zhang X
- Zhang X
- Zhang X
- Zhang X-W
- Zhang XD
- Zhang Y
- Zhang Y
- Zhang Y
- Zhang Y
- Zhang Y
- Zhang Y-D
- Zhang Y-Y
- Zhang Z
- Zhang Z
- Zhang Z
- Zhang Z
- Zhang Z
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- Zhao H
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- Zheng Z-G
- Zhivotovsky B
- Zhong Q
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- Zhou Z
- Zhou Z-Y
- Zhu B
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- Zhu G-Q
- Zhu H
- Zhu H
- Zhu H
- Zhu W-G
- Zhu Y
- Zhu Y
- Zhuang H
- Zhuang X
- Zientara-Rytter K
- Zimmermann CM
- Ziviani E
- Zoladek T
- Zong W-X
- Zorov DB
- Zorzano A
- Zou W
- Zou Z
- Zou Z
- Zuryn S
- Zwerschke W
- Ălvarez ĂMC
- Ăvalos Y
- Ănal G
- ĂstĂŒn S
- ÄoliÄ M
- ÄokiÄ J
- Ćœerovnik E
- Publication venue
- 'Informa UK Limited'
- Publication date
- 01/01/2021
- Field of study
In 2008, we published the first set of guidelines for standardizing research in autophagy. Since then, this topic has received increasing attention, and many scientists have entered the field. Our knowledge base and relevant new technologies have also been expanding. Thus, it is important to formulate on a regular basis updated guidelines for monitoring autophagy in different organisms. Despite numerous reviews, there continues to be confusion regarding acceptable methods to evaluate autophagy, especially in multicellular eukaryotes. Here, we present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes. These guidelines are not meant to be a dogmatic set of rules, because the appropriateness of any assay largely depends on the question being asked and the system being used. Moreover, no individual assay is perfect for every situation, calling for the use of multiple techniques to properly monitor autophagy in each experimental setting. Finally, several core components of the autophagy machinery have been implicated in distinct autophagic processes (canonical and noncanonical autophagy), implying that genetic approaches to block autophagy should rely on targeting two or more autophagy-related genes that ideally participate in distinct steps of the pathway. Along similar lines, because multiple proteins involved in autophagy also regulate other cellular pathways including apoptosis, not all of them can be used as a specific marker for bona fide autophagic responses. Here, we critically discuss current methods of assessing autophagy and the information they can, or cannot, provide. Our ultimate goal is to encourage intellectual and technical innovation in the field
Pertussis in Latin America and the Hispanic Caribbean: a systematic review
- Author
- Abarca VK
- Acosta E
- Agosti MR
- Almada C
- Amato Vieira R
- Angela Gentile
- Aquino-Andrade A
- Astudillo M
- Barata RB
- Barrantes T
- Baxter R
- Bellettini CV
- Bosch JJ
- Cardona D
- Cardozo MA
- Cruz LF
- Da Cruz MG
- David A
- de Araujo TM
- de Jesus DM
- De La Hoz F
- de Moraes Jde C
- Delamonica E
- Domenech de Celles M
- Domenech de Celles M
- Elsa Sarti
- Fallo A
- FarĂas Z
- Fernandes AR
- Galindo Santana BM
- Galindo Santana BM
- Galleguillos B
- Garib Z
- Gentile A
- Gentile A
- Gentile Ă
- GonzĂĄlez Arias M
- Guerreiro J
- Gutiérrez-Trujillo G
- Herrera JF
- Juan Pablo Torres
- Katz N
- Leite D
- Lucia Bricks
- Maldonado Briones H
- Mantilla DĂaz T
- Martins RM
- MarĂa L. Ăvila-AgĂŒero
- Mendoza-Flores L
- Moreno L
- MorĂłn Duarte LS
- MorĂłn Duarte LS
- Neyro S
- Olea Normandin AM
- Oliveira E Silva R
- Oliveira JC
- Paiva T
- Pan American Health Organization
- Pan American Health Organization
- Pan American Health Organization
- Paulo S
- Perez-Perez GF
- Pinell-McNamara VA
- Queiroz IT
- Quian Rivero JW
- Renato Avila Kfouri
- Richard E. Glover
- Riva Posse CA
- Rivera A
- Rolando Ulloa-Gutierrez
- Romanin V
- Romanin V
- Romano F
- Ruvinsky S
- Sato APS
- Sevilla ME
- Sosa M
- SuĂĄrez Moreno V
- Tauil M
- Ulloa VirgĂŒe AP
- Vargas Herrera J
- Villareal C
- Villegas D
- von Specht M
- World Health Organization
- World Health Organization
- Zerpa R
- Publication venue
- 'Informa UK Limited'
- Publication date
- Field of study