Présentation et discussion de quelques diagrammes de phases dans le système Ca-Fe-Si-C-O des formations ferrifères de la région de Berrahal - Oued El Aneb (massif de l’Edough, NE algérien)

La région de Berrahal-Oued El Aneb (Edough) contient d'anciens gisements et indices de magnétite ± hématite liés à des roches calco-silicatées. Les corps minéralisés sont stratiformes, plus ou moins lenticulaires, et sont encaissés dans des schistes à disthène, staurotide et grenat au voisinage de marbres. Les principales paragenèses relevées dans les corps  minéralisés comprennent essentiellement magnétite, olivine (fayalite), pyroxène (hédenbergite), calcite, micas et grenat (almandin). Ces minéraux ont subi les mêmes effets du métamorphisme que les roches encaissantes. L’interprétation d’analyses chimiques montre que les formations à l'origine des corps minéralisés correspondent à des roches carbonatoferrifères impures, plus ou moins siliceuses. Le fer aurait été exprimé sous forme carbonatée (sidérite). Un processus pétrogénétique, illustré par la discussion de diagrammes de phases, expliquant l'évolution des corps minéralisés dans leur contexte est proposé et nous a conduit à retracer la succession des différentes étapes d'évolution de ces corps et à expliquer la cristallisation des minéraux constituant les paragenèsesentrant dans la composition des faciès de minerai par une décarbonatation (+ oxydoréduction) métamorphique des formations originelles.Mots-clés : Edough, magnétite, formations ferrifères, métamorphisme, décarbonatation

Comparison between the Efficiency of Heterojunction Thin Film InGaP\GaAs\Ge and InGaP\GaAs Solar Cell

This paper presents the design parameters for a thin film 3J InGaP/GaAs/Ge solar cell with a simulated maximum efficiency of 32.11% using Tcad Silvaco. Design parameters include the doping concentration, molar fraction, layers’ thickness and tunnel junction characteristics. An initial dual junction InGaP/GaAs model of a previous published heterojunction cell was simulated in Tcad Silvaco to accurately predict solar cell performance. To improve the solar cell’s performance, we have fixed meshing, material properties, models and numerical methods. However, thickness and layer doping concentration were taken as variables. We, first simulate the InGaP\GaAs dual junction cell by changing the doping concentrations and thicknesses which showed an increase in efficiency. Next, a triple junction InGaP/GaAs/Ge cell was modeled by adding a Ge layer to the previous dual junction InGaP/GaAs model with an InGaP /GaAs tunnel junction

Chiral expansions of the pi0 lifetime

The corrections induced by light quark masses to the current algebra result for the $\pi^0$ lifetime are reexamined. We consider NNLO corrections and we compute all the one-loop and the two-loop diagrams which contribute to the decay amplitude at NNLO in the two-flavour chiral expansion. We show that the result is renormalizable, as Weinberg consistency conditions are satisfied. We find that chiral logarithms are present at this order unlike the case at NLO. The result could be used in conjunction with lattice QCD simulations, the feasibility of which was recently demonstrated. We discuss the matching between the two-flavour and the three-flavour chiral expansions in the anomalous sector at order one-loop and derive the relations between the coupling constants. A modified chiral counting is proposed, in which $m_s$ counts as $O(p)$. We have updated the various inputs needed and used this to make a phenomenological prediction.Comment: 20 pages, 1 figure; v2: comments and references added, accepted for publication in PR

Chronic myeloid leukemia and HIV-infection

The incidence of non-HIV-associated hematologic malignancies, including chronic myeloproliferative disorders, is increasing in HIV-infected (HIVþ) patients. This is thought to be due to prolonged survival in the era of highly active antiretroviral therapy (HAART). Previously, only six cases of chronic myeloid leukemia (CML) have been described in HIVþ individuals and limited information is available regarding the management of patients with concurrent CML and HIV- infection. We report three cases of CML in HIVþ patients who were treated with imatinib and HAART. Treatment was generally well tolerated, and cytogenetic response (complete in two patients) was achieved with follow-up ranging from 3 to 69 months. HIV viral load remained undetectable and CD4 cell counts were stable in all three patients. Concurrent treatment with imatinib and HAART can result in appropriate control of CML and HIV-infection as well as long-term survival

Aggressive presentation of breast implant-associated ALK-1 negative anaplastic large cell lymphoma with bilateral axillary lymph node involvement

We recently encountered a unique case of breast silicone implant-associated ALK-1 negative anaplastic large cell lymphoma (ALCL) in a female with a remote history of breast carcinoma, which presented as a sinusoidal infiltrate in an axillary lymph node (LN) and had a highly complex karyotype

Identification of rare Epstein-Barr virus infected memory B cells and plasma cells in non-monomorphic post-transplant lymphoproliferative disorders and the signature of viral signaling

Background and Objectives. In early and polymorphic post-transplant lymphoprolifera- tive disorders (PTLD) Epstein-Barr virus (EBV), through its latency proteins, drives the proliferation of B lymphocytes, a process which in immunocompetent individuals leads to the establishment of latently infected memory B cells. Design and Methods. We analyzed 11 cases, which included early and polymorphic PTLD, and 12 controls for latency of EBV infection and their antigenic profile. Results. We identified a minority of terminally differentiated EBER+ IRTA1+ memory B cells and EBER+ CD138+ PRDM1+ plasma cells in these samples. These elements were identified both in PTLD and in tumor-free tonsils from post-transplant patients but not in EBV– control tonsils. The expression of EBV latency proteins is heterogeneous, and is associated with activation of the NF-κB pathway. EBV signaling (through EBNA2, LMP1 and LMP2A) and NF-κB activation correlated with upregulation of target proteins: cMYC, JunB, CCL22, TRAF1 and IRF4. EBV-infected lymphocytes in early and polymor- phic PTLDs represent a mixture of latencies II, III and, in at least 1/3 of infected cells, of latency 0. Interpretation and conclusions. EBV infection correlates with NF-κB activation, with EBV-dependent cell signaling, and lastly, with the presence of EBV-infected plasma cells and memory cells. Key words: post-transplant lymphoproliferative disorder, Epstein-Barr virus, viral latency, NF-κB signaling, plasma cell, memory B cell