Transnational NGOs between Popular Uprising and Authoritarian Regime: Developments in Egypt

Scholars differentiate the concepts of internationalism and transnationalism. While the first refers to the connectivity between macro institutions such as states, multinational corporations and other institutionalized actors within and beyond national boundaries, the second term emphasizes public movements, organizations and communities engaged in de-territorialized socio‐cultural, political and economic activities. This paper focuses on the role of transnational NGOs (TNGOs) in recent developments in Egypt. The current scholarly debate on the so‐called “Arab spring” considers the mobilization of disempowered youth, intense media-tech application and sustained international pressure as crucial to ousting authoritarian regimes in North Africa. Delineating the role of TNGOs complements such findings. TNGO activities and responses to the Egyptian uprising in 2011 and to the ensuing coup in 2013 reveal the capability of such organizations to balance civic transformational oriented mobilizations with state centred institutional considerations. Furthermore though TNGOs cannot directly change the current political stalemate in Egypt, the power elite might misinterpret the changing and sometimes contradictory positions of these organizations and might eventually encourage the return to authoritarianism. After introductory remarks on the background of the uprising, the paper proceeds to theoretical discussion of transnational engagement followed by recent historical and current empirical developments

Relative commutants of strongly self-absorbing C*-algebras

The relative commutant $A'\cap A^{\mathcal{U}}$ of a strongly self-absorbing algebra $A$ is indistinguishable from its ultrapower $A^{\mathcal{U}}$. This applies both to the case when $A$ is the hyperfinite II$_1$ factor and to the case when it is a strongly self-absorbing C*-algebra. In the latter case we prove analogous results for $\ell_\infty(A)/c_0(A)$ and reduced powers corresponding to other filters on $\bf N$. Examples of algebras with approximately inner flip and approximately inner half-flip are provided, showing the optimality of our results. We also prove that strongly self-absorbing algebras are smoothly classifiable, unlike the algebras with approximately inner half-flip.Comment: Some minor correction

The Auditory Nerve Overlapped Waveform (ANOW) detects small endolymphatic manipulations that may go undetected by conventional measurements

Electrocochleography (ECochG) has been used to assess Ménière's disease, a pathology associated with endolymphatic hydrops and low-frequency sensorineural hearing loss. However, the current ECochG techniques are limited for use at high-frequencies only (≥1 kHz) and cannot be used to assess and understand the low-frequency sensorineural hearing loss in ears with Ménière's disease. In the current study, we use a relatively new ECochG technique to make measurements that originate from afferent auditory nerve fibers in the apical half of the cochlear spiral to assess effects of endolymphatic hydrops in guinea pig ears. These measurements are made from the Auditory Nerve Overlapped Waveform (ANOW). Hydrops was induced with artificial endolymph injections, iontophoretically applied Ca2+ to endolymph, and exposure to 200 Hz tones. The manipulations used in this study were far smaller than those used in previous investigations on hydrops. In response to all hydropic manipulations, ANOW amplitude to moderate level stimuli was markedly reduced but conventional ECochG measurements of compound action potential thresholds were unaffected (i.e., a less than 2 dB threshold shift). Given the origin of the ANOW, changes in ANOW amplitude likely reflect acute volume disturbances accumulate in the distensible cochlear apex. These results suggest that the ANOW could be used to advance our ability to identify initial stages of dysfunction in ears with Ménière's disease before the pathology progresses to an extent that can be detected with conventional measures

Energy-Efficient Low-Complexity Algorithm in 5G Massive MIMO Systems

Energy efficiency (EE) is a critical design when taking into account circuit power consumption (CPC) in fifth-generation cellular networks. These problems arise because of the increasing number of antennas in massive multiple-input multiple-output (MIMO) systems, attributable to inter-cell interference for channel state information. Apart from that, a higher number of radio frequency (RF) chains at the base station and active users consume more power due to the processing activities in digital-to-analogue converters and power amplifiers. Therefore, antenna selection, user selection, optimal transmission power, and pilot reuse power are important aspects in improving energy efficiency in massive MIMO systems. This work aims to investigate joint antenna selection, optimal transmit power and joint user selection based on deriving the closed-form of the maximal EE, with complete knowledge of large-scale fading with maximum ratio transmission. It also accounts for channel estimation and eliminating pilot contamination as antennasM→∞. This formulates the optimization problem of joint optimal antenna selection, transmits power allocation and joint user selection to mitigate inter-cellinterference in downlink multi-cell massiveMIMO systems under minimized reuse of pilot sequences based on a novel iterative low-complexity algorithm (LCA) for Newton’s methods and Lagrange multipliers. To analyze the precise power consumption, a novel power consumption scheme is proposed for each individual antenna, based on the transmit power amplifier and CPC. Simulation results demonstrate that the maximal EE was achieved using the iterative LCA based on reasonable maximum transmit power, in the case the noise power is less than the received power pilot. The maximum EE was achieved with the desired maximum transmit power threshold by minimizing pilot reuse, in the case the transmit power allocation ρd = 40 dBm, and the optimal EE=71.232 Mb/j

Conservation of epigenetic regulation by the MLL3/4 tumour suppressor in planarian pluripotent stem cells

Currently, little is known about the evolution of epigenetic regulation in animal stem cells. Here we demonstrate, using the planarian stem cell system to investigate the role of the COMPASS family of MLL3/4 histone methyltransferases that their function as tumor suppressors in mammalian stem cells is conserved over a long evolutionary distance. To investigate the potential conservation of a genome-wide epigenetic regulatory program in animal stem cells, we assess the effects of Mll3/4 loss of function by performing RNA-seq and ChIP-seq on the G2/M planarian stem cell population, part of which contributes to the formation of outgrowths. We find many oncogenes and tumor suppressors among the affected genes that are likely candidates for mediating MLL3/4 tumor suppression function. Our work demonstrates conservation of an important epigenetic regulatory program in animals and highlights the utility of the planarian model system for studying epigenetic regulation

Pendidikan Rohani Dalam Tradisi Amaliyah Di Pondok Pesantren Salafiyah Kota Kediri

In this study, the authors used a descriptive qualitative approach which departed from two questions, namely; 1.) "What is Amaliyah doing at the Salafiyah Islamic Boarding School in Kediri?", 2.) How is the relationship between Amaliyah at the Salafiyah Islamic Boarding School and spiritual education? " With these several questions, the results of the research conducted found that spiritual education in the amaliyah tradition at the Salafiyah Islamic Boarding School includes: grave pilgrimages, prayer beads, and recitation of 1000 sholawat. With this amaliyah tradition, we are able to; 1.) overcoming anxiety, fear, and uneasiness of the hearts of the students, 2.) calming the soul, because in it there are things that bring calm, such as dzikrullah in reading tahlil, tahmid, tasbih, and sholawat., 3.) giving direction, motivation and finally grow full awareness to obey, submit to carry out orders and know the prohibitions

Inflammatory myopathy occurring shortly after severe acute respiratory syndrome coronavirus 2 vaccination: two case reports

Background: Vaccination remains the cornerstone approach to exiting the current global coronavirus disease 2019 pandemic caused by severe acute respiratory syndrome coronavirus 2. The novel messenger ribonucleic acid vaccines offer a high level of protection and are widely used throughout the world. With more people receiving the vaccines, better understanding of their relative safety can be reached. In this report, we describe two patients who developed inflammatory myopathy within 48 hours of receiving the Pfizer BNT162b2 vaccine. Case presentation: Patient A, a 55-year-old South East Asian woman, presented with a 6-week history of pruritic facial and torso rash and a 1-week history of worsening proximal myopathy. Her rash first developed 2 days after receiving the first dose of BNT162b2 vaccine. Patient B, a 72-year-old Caucasian woman, presented with a 2-week history of proximal myopathy a day after receiving the second dose of BNT162b2 vaccine. Both patients had elevated creatine kinase on admission. Patient A tested positive for anti-Mi-2a antibody and anti-Ro-52 antibody, while Patient B was positive for anti-fibrillarin antibody. Magnetic resonance imaging subsequently confirmed generalized acute muscle inflammation and subcutaneous inflammation consistent with inflammatory myositis. Both patients did not have a previous history or family history of autoimmune disease. Patients A and B were diagnosed with dermatomyositis and inflammatory myositis, respectively. They were initially treated with pulsed intravenous methylprednisolone followed by oral prednisolone. However, as their conditions were resistant to corticosteroids, both eventually received and responded well to intravenous immunoglobulin therapy. Conclusion: There are previously reported cases of severe acute respiratory syndrome coronavirus 2-induced and other vaccine-related inflammatory myopathies. However, the precise mechanisms are not elucidated. Without more evidence and convincing pathophysiology, it is not possible to conclude that our patients developed inflammatory myopathy because of the vaccine. However, the timing of the disease onset and the lack of previous history raise an important question of this novel messenger ribonucleic acid therapy