Genetical and physiological studies of the domestic fowl

A. GENETICS OF THE FOWL: 1930 I The inheritance of frizzled plumage. Journ. Genet. 22: 109 -127. • 1933 II A four -gene autosomal linkage group. Genetics 18: 82 -91. • 1934 III Congenital tremor in young chicks. Journ. Hered. 25: 341 -390. (Hutt and Child) • 1936 IV Linkage relations of crest, dominant white and frizzling in the fowl. Amer. Nat. 70: 379 -394. (warren and Hutt) • V The modified frizzle. Journ. Genet. 32: 277 -295. • VI A tentative chromosome map. 3eue Forachungen in Tierzucht und Abstammungslehre (Doerst Festschrift): 105-112. • 1938 VII Breed differences in susceptibility to extreme heat. Poultry Science 17: 454-462. • 1939 VIII Breed differences in resistance to a deficiency of vitamin B1 in the fowl. Journ. Agric. Res. 58: 305-316. (Lamoreux and Hutt) • 1938 IX Naked, a new sex -linked mutation. Journ. Hered. 29: 370 -379. (Hutt and Sturkie) • X A relation between breed characteristics and poor reproduction in White Wyandotte fowls. Amer. Nat. (In press)NOT IN THE REGULAR SERIES: 1929 A note on Lambert =s mosaic in the fowl. Journ. Hered. 20: 323 -324. • Sex dimorphism and vari+bility in the appendiculcr skeleton of the Leghorn fowl. Poultry Science 8: 202- 218. • 1932 Eight new mutations in the domestic fowl. Proc. Sixth Internat. Congress of Genetics, Ithaca, New York, 1932. Vol. 2: 96-97.B. MAMMALIAN GENETICS: 1930 Bovine quadruplets including twins apparently monozygotic. Journ. Hered. 21: 339-348. • 1932 Congenital tailleesness in the rat. Journ. Hered. 23: 363-367. (Hutt and Mydland) • 1934 A hereditary lethal muscle contracture in cattle. Mourn. Hered. 25: 41-46,C. HUMAN GENETICS 1934 Sex differences in the expression of autosomal genes affecting humen dentition. A Decade of Progress in Eugenics, Scientific Papers of the 3rd. International Congress of Eugenics, 1932: 447-452. • 1935 An earlier record of the toothless men of Sind. Journ. Herod. 26: 65-66.B. EMBRYONIC MORTALITY IN THE FOWL: 1929 I The frequencies of various malpositions of the chick embryo and their significance. Proc. Roy Soc. Edin. 49, Pt. 2, No. 10: 118-130. • II Chondrodystrophy in the chick. Proc. Roy. Soc. Edin. 49, Pt. 2, No. 11: 131 -144. (Hutt and Greenwood) • III Chick monsters in relation to embryonic mortality. Proc. Roy. Soc. Edin. 49, Pt. 2, No. 12: 145 - 155. (Hutt and Greenwood) • 1930 IV Comparative rates of mortality in eggs laid at different periods of the day and their bearing on theories of the origin of monsters. Poultry Science 9: 194-203. (Hutt and. Pilkey) • 1934 V Relationships between positions of the egg and frequencies of malpositions. Poultry Science 13: 3 -13. (Hutt and Pilkey) • Vi The relation between abnormal orientation of the 4-day embryo and position of the chick at hatching. Journ. Agric. Res. 48: 517 -531. (Covers and Hutt) • 1938 VII On the relation of malpositions to the size and shape of eggs. Poultry Science 17: 345 -352.NOT IN THE REGULAR SERIES: 1930 On the origin, common types end economic significance of teratological monsters in embryos of the domestic fowl. Froc. 4th World's Poultry Congress, London: 195 -202.E. AVIAN PHYSIOLOGY (mostly endocrinology and physiology of reproduction): 1928 Further experiments in feeding thyroid to fowls. Poultry Science 7: 50-66. (Cole and Hutt) • Potentially fetal fatigue of the cervical muscles of the fowl resulting from an excessively large comb. Vet. Journ. 84: 579 -584. • 1929 On the relation of fertility in fowls to the amount of testicular material and density of sperm suspension. Proc. Roy. Soc. Edin. 49, Pt. 2, No. 9: 102-117. • 1930 A note on the effects of different doses of thyroid on the fowl. Journ. Exper. Biol. 7: 1 -6. • 1933 On the fecundity of partially ovariotomised fowls. Journ. Exp. Zool. 65: 199 -214. (Hutt and Grussendorf) • 1935 Idiopathic hypoparathyroidism and tetany in the fowl. Endocrinology 19: 39& -492. (Hutt and Boyd) • 1938 The influence of estrogens in egg yolk upon avian blood calcium. Endocrinology 23: 793-799. (Altmann and Hutt) • 1939 Variability of body temperature in the normal chick. Poultry Science lge 70-75. (Lamoreax and Hutt) • An intrafollicular ovum laïd by a fowl. Poultry Science (In press)F. ORNITHOLOGY: 1932 Birds observed.from shipboard in crossing the North Atlantic. The Auk 49z 184 -190. • 1938 Humber of feathers and body sire in passerine birds. The Auk 55: 651 -657. (Hutt and Ball)G. REVIEWS AND GENERAL: 1932 Paradoxical terminology in genetics. Amer. Nat. 66: 274 -277. • 1933 Research with a hen. Science 78: 449 -452. 1934 Inherited, lethal characters in domestic animals. The Cornell Veterinarian 24: 1 -25. • 1938 The geneticist's objectives in poultry improvement. Amer. Nat. 72: 268-284

Detection of fixed points in spatiotemporal signals by clustering method

We present a method to determine fixed points in spatiotemporal signals. A 144-dimensioanl simulated signal, similar to a Kueppers-Lortz instability, is analyzed and its fixed points are reconstructed.Comment: 3 pages, 3 figure

Alice I\u27m In Wonderland : Since The Day That I First Met You

https://digitalcommons.library.umaine.edu/mmb-vp/2830/thumbnail.jp

The environmental toxicant 2,3,7,8-tetrachlorodibenzo-p-dioxin disrupts morphogenesis of the rat pre-implantation embryo

© 2008 Hutt et al. This is an Open Access article distributed under the terms of the Creative Commons Attribution License 2.0, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. The definitive version was published in BMC Developmental Biology 8 (2008): 1, doi:10.1186/1471-213X-8-1.Environmental toxicants, whose actions are often mediated through the aryl hydrocarbon receptor (AhR) pathway, pose risks to the health and well-being of exposed species, including humans. Of particular concern are exposures during the earliest stages of development that while failing to abrogate embryogenesis, may have long term effects on newborns or adults. The purpose of this study was to evaluate the effect of maternal exposure to the AhR-specific ligand 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) on the development of rat pre-implantation embryos with respect to nuclear and cytoskeletal architecture and cell lineage allocation. We performed a systematic 3 dimensional (3D) confocal microscopy analysis of rat pre-implantation embryos following maternal exposure to environmentally relevant doses of TCDD. Both chronic (50 ng/kg/wk for 3 months) and acute (50 ng/kg and 1 μg/kg at proestrus) maternal TCDD exposure disrupted morphogenesis at the compaction stage (8–16 cell), with defects including monopolar spindle formation, f-actin capping and fragmentation due to aberrant cytokinesis. Additionally, the size, shape and position of nuclei were modified in compaction stage pre-implantation embryos collected from treated animals. Notably, maternal TCDD exposure did not compromise survival to blastocyst, which with the exception of nuclear shape, were morphologically similar to control blastocysts. We have identified the compaction stage of pre-implantation embryogenesis as critically sensitive to the effects of TCDD, while survival to the blastocyst stage is not compromised. To the best of our knowledge this is the first in vivo study to demonstrate a critical window of pre-implantation mammalian development that is vulnerable to disruption by an AhR ligand at environmentally relevant doses.This research was supported by NIH/NIEHS-012916 (BKP), ESHE Fund (DFA), Hall Family Foundation (DFA and KJH) and Biomedical Research Training Grant KUMC (KJH)

Modeling of the Power Cycling Performance of a Si on Si Flip Chip Assembly

Flip Chip (FC) technology offers many advantages over conventional surface mount technology, including a smaller device footprint and higher interconnection density. Low power but complex consumer items, such as mobile telecommunications devices, utilise this packaging technology and it is likely to spread to other electronics sectors where components have higher power dissipations and/or they have to operate in a hostile environment. As the scope for FC packaging broadens, a reliable means of establishing the long term performance of a particular package is necessary. Traditionally thermal cycling has been a primary reliability test for electronic assemblies including FC, however this fails to capture the behaviour of assemblies where the component thermal expansion is well matched to that of the substrate due to the isothermal heating and cooling of the assembly. In this situation power cycling offers an alternative means of determining the module performance. This paper describes the use of Finite Element Modeling (FEM) to explore the effects of power cycling on a silicon on silicon Multi-Chip Module (MCM) constructed with a low solder joint standoff height of 30-35µm. Particular attention was given to the boundary conditions that are inevitably atypical of those used in traditional thermal cycling. The paper presents results of the temperature distributions throughout the assembly, which were found to depend upon the substrate base material (FR4 or copper) that the MCM was attached to. The results of the FEM analysis were verified by assembling test devices and measuring their temperature distribution under steady state and power cycling conditions. The predicted temperatures may then be used as boundary conditions in FEM of thermal stresses and fatigue in the assembly