80 research outputs found

    Running into New Territory in SUSY Parameter Space

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    The LEP-II bound on the light Higgs mass rules out the vast majority of parameter space left to the Minimal Supersymmetric Standard Model (MSSM) with weak-scale soft-masses. This suggests the importance of exploring extensions of the MSSM with non-minimal Higgs physics. In this article, we explore a theory with an additional singlet superfield and an extended gauge sector. The theory has a number of novel features compared to both the MSSM and Next-to-MSSM, including easily realizing a light CP-even Higgs mass consistent with LEP-II limits, tan(beta) < 1, and a lightest Higgs which is charged. These features are achieved while remaining consistent with perturbative unification and without large stop-masses. Discovery modes at the Tevatron and LHC are discussed.Comment: 15 pages, 5 figures; Typo in equation (4.5) corrected; submitted to JHE

    Higgs as a pseudo-Goldstone boson, the mu problem and gauge-mediated supersymmetry breaking

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    We study the interplay between the spontaneous breaking of a global symmetry of the Higgs sector and gauge-mediated supersymmetry breaking, in the framework of a supersymmetric model with global SU(3) symmetry. In addition to solving the supersymmetric flavour problem and alleviating the little hierarchy problem, this scenario automatically triggers the breaking of the global symmetry and provides an elegant solution to the mu/Bmu problem of gauge mediation. We study in detail the processes of global symmetry and electroweak symmetry breaking, including the contributions of the top/stop and gauge-Higgs sectors to the one-loop effective potential of the pseudo-Goldstone Higgs boson. While the joint effect of supersymmetry and of the global symmetry allows in principle the electroweak symmetry to be broken with little fine-tuning, the simplest version of the model fails to bring the Higgs mass above the LEP bound due to a suppressed tree-level quartic coupling. To cure this problem, we consider the possibility of additional SU(3)-breaking contributions to the Higgs potential, which results in a moderate fine-tuning. The model predicts a rather low messenger scale, a small tan beta value, a light Higgs boson with Standard Model-like properties, and heavy higgsinos.Comment: 19 pages, 6 figures. New section 3.3 on the mu/Bmu problem, more detailed analytic computation in section 4.1, error in Fig. 5 corrected, significant redactional changes (including abstract, introduction and conclusion) in order to better emphasize the main results of the paper. Title changed in journal. Final version to appear in Eur. Phys. J.

    Kaluza-Klein gravitino production with a single photon at e^+ e^- colliders

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    In a supersymmetric large extra dimension scenario, the production of Kaluza-Klein gravitinos accompanied by a photino at e^+ e^- colliders is studied. We assume that a bulk supersymmetry is softly broken on our brane such that the low-energy theory resembles the MSSM. Low energy supersymmetry breaking is further assumed as in GMSB, leading to sub-eV mass shift in each KK mode of the gravitino from the corresponding graviton KK mode. Since the photino decays within a detector due to its sufficiently large inclusive decay rate into a photon and a gravitino, the process e^+ e^- -> photino + gravitino yields single photon events with missing energy. Even if the total cross section can be substantial at sqrt(s)=500 GeV, the KK graviton background of e^+ e^- -> photon + graviton is kinematically advantageous and thus much larger. It is shown that the observable, sigma(e^-_L)-sigma(e^-_R), can completely eliminate the KK graviton background but retain most of the KK gravitino signal, which provides a unique and robust method to probe the supersymmetric bulk.Comment: Reference added and typos correcte

    Genome-wide meta-analysis uncovers novel loci influencing circulating leptin levels.

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    Leptin is an adipocyte-secreted hormone, the circulating levels of which correlate closely with overall adiposity. Although rare mutations in the leptin (LEP) gene are well known to cause leptin deficiency and severe obesity, no common loci regulating circulating leptin levels have been uncovered. Therefore, we performed a genome-wide association study (GWAS) of circulating leptin levels from 32,161 individuals and followed up loci reaching P&lt;10(-6) in 19,979 additional individuals. We identify five loci robustly associated (P&lt;5 × 10(-8)) with leptin levels in/near LEP, SLC32A1, GCKR, CCNL1 and FTO. Although the association of the FTO obesity locus with leptin levels is abolished by adjustment for BMI, associations of the four other loci are independent of adiposity. The GCKR locus was found associated with multiple metabolic traits in previous GWAS and the CCNL1 locus with birth weight. Knockdown experiments in mouse adipose tissue explants show convincing evidence for adipogenin, a regulator of adipocyte differentiation, as the novel causal gene in the SLC32A1 locus influencing leptin levels. Our findings provide novel insights into the regulation of leptin production by adipose tissue and open new avenues for examining the influence of variation in leptin levels on adiposity and metabolic health

    Gaia Early Data Release 3: acceleration of the solar system from Gaia astrometry

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    Stars and planetary system

    Gaia early data release 3: summary of the contents and survey properties (Corrigendum)

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    ERRATUMThis article is an erratum for:[https://doi.org/10.1051/0004-6361/202039657]​​​​​​​Instrumentatio

    Gaia Early Data Release 3: the Gaia catalogue of nearby stars

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    Stars and planetary system
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