23 research outputs found
On the decomposition of stochastic cellular automata
In this paper we present two interesting properties of stochastic cellular
automata that can be helpful in analyzing the dynamical behavior of such
automata. The first property allows for calculating cell-wise probability
distributions over the state set of a stochastic cellular automaton, i.e.
images that show the average state of each cell during the evolution of the
stochastic cellular automaton. The second property shows that stochastic
cellular automata are equivalent to so-called stochastic mixtures of
deterministic cellular automata. Based on this property, any stochastic
cellular automaton can be decomposed into a set of deterministic cellular
automata, each of which contributes to the behavior of the stochastic cellular
automaton.Comment: Submitted to Journal of Computation Science, Special Issue on
Cellular Automata Application
A comprehensive analysis of the epidemiology and clinical characteristics of anti-LRP4 in myasthenia gravis
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VH GENE FAMILY UTILIZATION OF ANTIACETYLCHOLINE RECEPTOR ANTIBODIES IN EXPERIMENTAL AUTOIMMUNE MYASTHENIA-GRAVIS
VH GENE FAMILY UTILIZATION OF ANTIACETYLCHOLINE RECEPTOR ANTIBODIES IN EXPERIMENTAL AUTOIMMUNE MYASTHENIA-GRAVIS
Inherited congenital goiter in mice.
Congenital goiter (cog), a new autosomal recessive mutation in mice, has been mapped to the central region of chromosome 15. Young adult mutant mice are characterized by a reduced rate of growth, mild anemia, hypothyroidism, as indicated by significantly lower total serum T4 and T3, and elevated serum TSH. Thyroids from mutant mice are hypertrophied, deficient in colloid, show a reduced accumulation of iodine that is partially susceptible to perchlorate ion discharge, have modestly elevated serum immunoreactive thyroglobulin (Tg) levels, but are markedly deficient in glandular immunoreactive Tg content. Thyroid hormone therapy corrects the growth deficiency and prevents the thyroid hypertrophy resulting from excessive stimulation by TSH. These findings suggest that the cog mutant gene results in primary hypothyroidism in response to either defective synthesis or processing of Tg
A Tonality-oriented Symbolic Representation of Musical Audio Generated by Classification Trees
info:eu-repo/semantics/publishe