Upper critical dimension of the KPZ equation

Numerical results for the Directed Polymer model in 1+4 dimensions in various types of disorder are presented. The results are obtained for system size considerably larger than that considered previously. For the extreme strong disorder case (Min-Max system), associated with the Directed Percolation model, the expected value of the meandering exponent, zeta = 0.5 is clearly revealed, with very week finite size effects. For the week disorder case, associated with the KPZ equation, finite size effects are stronger, but the value of seta is clearly seen in the vicinity of 0.57. In systems with "strong disorder" it is expected that the system will cross over sharply from Min-Max behavior at short chains to weak disorder behavior at long chains. This is indeed what we find. These results indicate that 1+4 is not the Upper Critical Dimension (UCD) in the week disorder case, and thus 4+1 does not seem to be the upper critical dimension for the KPZ equation

Airbag vent valve and system

An energy absorbing airbag system includes one or more vent valve assemblies for controlling the release of airbag inflation gases to maintain inflation gas pressure within an airbag at a substantially constant pressure during a ride-down of an energy absorbing event. Each vent valve assembly includes a cantilever spring that is flat in an unstressed condition and that has a free end portion. The cantilever spring is secured to an exterior surface of the airbag housing and flexed to cause the second free end portion of the cantilever spring to be pressed, with a preset force, against a vent port or a closure covering the vent port to seal the vent port until inflation gas pressure within the airbag reaches a preselected value determined by the preset force whereupon the free end portion of the cantilever spring is lifted from the vent port by the inflation gases within the airbag to vent the inflation gases from within the airbag. The resilience of the cantilever spring maintains a substantially constant pressure within the airbag during a ride-down portion of an energy absorbing event by causing the cantilever spring to vent gases through the vent port whenever the pressure of the inflation gases reaches the preselected value and by causing the cantilever spring to close the vent port whenever the pressure of the inflation gases falls below the preselected value

Coherence-enhanced imaging of a degenerate Bose gas

We present coherence-enhanced imaging, an in situ technique that uses Raman superradiance to probe the spatial coherence properties of an ultracold gas. Applying this method, we obtain a spatially resolved measurement of the condensate number and more generally, of the first-order spatial correlation function in a gas of $^{87}$Rb atoms. We observe the enhanced decay of propagating spin gratings in high density regions of a Bose condensate, a decay we ascribe to collective, non-linear atom-atom scattering. Further, we directly observe spatial inhomogeneities that arise generally in the course of extended sample superradiance.Comment: 4 pages, 4 figure

High-Resolution Magnetometry with a Spinor Bose-Einstein Condensate

We demonstrate a precision magnetic microscope based on direct imaging of the Larmor precession of a $^{87}$Rb spinor Bose-Einstein condensate. This magnetometer attains a field sensitivity of 8.3 pT/Hz$^{1/2}$ over a measurement area of 120 $\mu$m$^2$, an improvement over the low-frequency field sensitivity of modern SQUID magnetometers. The corresponding atom shot-noise limited sensitivity is estimated to be 0.15 pT/Hz$^{1/2}$ for unity duty cycle measurement. The achieved phase sensitivity is close to the atom shot-noise limit suggesting possibilities of spatially resolved spin-squeezed magnetometry. This magnetometer marks a significant application of degenerate atomic gases to metrology

Mapping the cellular electrophysiology of rat sympathetic preganglionic neurones to their roles in cardiorespiratory reflex integration:A whole cell recording study in situ

Sympathetic preganglionic neurones (SPNs) convey sympathetic activity flowing from the CNS to the periphery to reach the target organs. Although previous in vivo and in vitro cell recording studies have explored their electrophysiological characteristics, it has not been possible to relate these characteristics to their roles in cardiorespiratory reflex integration. We used the working heart–brainstem preparation to make whole cell patch clamp recordings from T3–4 SPNs (n = 98). These SPNs were classified by their distinct responses to activation of the peripheral chemoreflex, diving response and arterial baroreflex, allowing the discrimination of muscle vasoconstrictor-like (MVC(like), 39%) from cutaneous vasoconstrictor-like (CVC(like), 28%) SPNs. The MVC(like) SPNs have higher baseline firing frequencies (2.52 ± 0.33 Hz vs. CVC(like) 1.34 ± 0.17 Hz, P = 0.007). The CVC(like) have longer after-hyperpolarisations (314 ± 36 ms vs. MVC(like) 191 ± 13 ms, P < 0.001) and lower input resistance (346 ± 49  MΩ vs. MVC(like) 496 ± 41 MΩ, P < 0.05). MVC(like) firing was respiratory-modulated with peak discharge in the late inspiratory/early expiratory phase and this activity was generated by both a tonic and respiratory-modulated barrage of synaptic events that were blocked by intrathecal kynurenate. In contrast, the activity of CVC(like) SPNs was underpinned by rhythmical membrane potential oscillations suggestive of gap junctional coupling. Thus, we have related the intrinsic electrophysiological properties of two classes of SPNs in situ to their roles in cardiorespiratory reflex integration and have shown that they deploy different cellular mechanisms that are likely to influence how they integrate and shape the distinctive sympathetic outputs

Pisatin demethylation by fungal pathogens and nonpathogens of pea: Association with pisatin tolerance and virulence

Previous studies have indicated that detoxification of their hosts’ phytoalexins is a tolerance mechanism for some true fungi, but not the fungus-like Oomycota, and may be involved in determining the virulence of a pathogen. In the present study, the associations between demethylation of the pea phytoalexin pisatin, tolerance to pisatin, and viru­lence on pea were examined for 50 fungal isolates which represent 17 species of pathogens and nonpathogens of pea. All isolates of Pythium coloratum and P. irregulare failed to metabolize and were sensitive to pisatin, consistent with previous observations that members of the Oomycota generally lack the ability to metabolize and are sensitive to their hosts’ phytoalexins. Among true fungi tested, the ability to demethylate pisatin was common, regardless of whether the particular isolate was pathogenic on pea or not. However, when the rate of pisatin demethylation was compared to virulence, all but one of the moderate to highly virulent isolates rapidly demethylated pisatin. In addition, the more rapidly demethylating isolates were generally more tolerant of pisatin. These results suggest that a specialized en­zyme system for quickly detoxifying pisatin might be present in most pea pathogens. In previous studies a specific cy­tochrome P450 enzyme for demethylating pisatin was identified in the pea pathogen Nectria haematococca mating pop­ulation VI, and genes (PDA genes) encoding that enzyme have been cloned from this fungus. When DNA specific for these genes was used to probe genomic DNA from other fungi that demethylate pisatin, significant hybridization was detected with only one fungus, the pea pathogen Fusarium oxysporum f. sp. pisi. If the other pea pathogens possess a specific cytochrome P450 system for detoxification of pisatin, the genes encoding these enzymes apparently share lim­ited nucleotide similarity with N. haematococca PDA genes