404 research outputs found
Reduction of quantum noise in optical interferometers using squeezed light
We study the photon counting noise in optical interferometers used for
gravitational wave detection. In order to reduce quantum noise a squeezed
vacuum state is injected into the usually unused input port. Here, we
specifically investigate the so called `dark port case', when the beam splitter
is oriented close to 90{\deg} to the incoming laser beam, such that nearly all
photons go to one output port of the interferometer, and only a small fraction
of photons is seen in the other port (`dark port'). For this case it had been
suggested that signal amplification is possible without concurrent noise
amplification [R.Barak and Y.Ben-Aryeh, J.Opt.Soc.Am.B25(361)2008]. We show
that by injection of a squeezed vacuum state into the second input port,
counting noise is reduced for large values of the squeezing factor, however the
signal is not amplified. Signal strength only depends on the intensity of the
laser beam.Comment: 8 pages, 1 figur
Theoretical study of the dynamic structure factor of superfluid 4He
We study the dynamic structure factor of superfluid 4He
at zero temperature in the roton momentum region and beyond using
field-theoretical Green's function techniques. We start from the
Gavoret-Nozi\`{e}res two-particle propagator and introduce the concept of
quasiparticles. We treat the residual (weak) interaction between quasiparticles
as being local in coordinate space and weakly energy dependent. Our
quasiparticle model explicitly incorporates the Bose-Einstein condensate. A
complete formula for the dynamic susceptibility, which is related to , is derived. The structure factor is numerically calculated
in a self-consistent way in the special case of a momentum independent
interaction between quasiparticles. Results are compared with experiment and
other theoretical approaches.Comment: 17 pages, 16 figure
Radiative decays of decuplet hyperons
We calculate the radiative decay widths of decuplet hyperons in a chiral
constituent quark model including electromagnetic exchange currents between
quarks. Exchange currents contribute significantly to the E2 transition
amplitude, while they largely cancel for the M1 transition amplitude.
Strangeness suppression of the radiative hyperon decays is found to be weakened
by exchange currents. Differences and similarities between our results and
other recent model predictions are discussed.Comment: 11 pages, 1 eps figure, revtex, accepted for publication in Phys.
Rev.
Nucleon Spin-Polarisabilities from Polarisation Observables in Low-Energy Deuteron Compton Scattering
We investigate the dependence of polarisation observables in elastic deuteron
Compton scattering below the pion production threshold on the spin-independent
and spin-dependent iso-scalar dipole polarisabilities of the nucleon. The
calculation uses Chiral Effective Field Theory with dynamical Delta(1232)
degrees of freedom in the Small Scale Expansion at next-to-leading order.
Resummation of the NN intermediate rescattering states and including the Delta
induces sizeable effects. The analysis considers cross-sections and the
analysing power of linearly polarised photons on an unpolarised target, and
cross-section differences and asymmetries of linearly and circularly polarised
beams on a vector-polarised deuteron. An intuitive argument helps one to
identify kinematics in which one or several polarisabilities do not contribute.
Some double-polarised observables are only sensitive to linear combinations of
two of the spin-polarisabilities, simplifying a multipole-analysis of the data.
Spin-polarisabilities can be extracted at photon energies \gtrsim 100 MeV,
after measurements at lower energies of \lesssim 70 MeV provide high-accuracy
determinations of the spin-independent ones. An interactive Mathematica 7.0
notebook of our findings is available from [email protected]: 30 pages LaTeX2e, including 22 figures as 66 .eps file embedded with
includegraphicx; three errors in initial submission corrected. This
submission includes ot the erratum to be published in EPJA (2012) and the
corrections in the tex
Non-perturbative Gluons and Pseudoscalar Mesons in Baryon Spectroscopy
We study baryon spectroscopy including the effects of pseudoscalar meson
exchange and one gluon exchange potentials between quarks, governed by
. The non-perturbative, hyperspherical method calculations show that
one can obtain a good description of the data by using a quark-meson coupling
constant that is compatible with the measured pion-nucleon coupling constant,
and a reasonably small value of .Comment: 12 pages; Submitted to Phys. Rev. C. Rapid Communication
Muscle-specific ablation of glucose transporter 1 (GLUT1) does not impair basal or overload-stimulated skeletal muscle glucose uptake
Glucose transporter 1 (GLUT1) is believed to solely mediate basal (insulin-independent) glucose uptake in skeletal muscle; yet recent work has demonstrated that mechanical overload, a model of resistance exercise training, increases muscle GLUT1 levels. The primary objective of this study was to determine if GLUT1 is necessary for basal or overload-stimulated muscle glucose uptake. Muscle-specific GLUT1 knockout (mGLUT1KO) mice were generated and examined for changes in body weight, body composition, metabolism, systemic glucose regulation, muscle glucose transporters, and muscle
Genetically Increasing Flux Through b-Oxidation in Skeletal Muscle Increases Mitochondrial Reductive Stress and Glucose Intolerance
Elevated mitochondrial hydrogen peroxide (H(2)O(2)) emission and an oxidative shift in cytosolic redox environment have been linked to high-fat-diet-induced insulin resistance in skeletal muscle. To test specifically whether increased flux through mitochondrial fatty acid oxidation, in the absence of elevated energy demand, directly alters mitochondrial function and redox state in muscle, two genetic models characterized by increased muscle β-oxidation flux were studied. In mice overexpressing peroxisome proliferator-activated receptor-α in muscle (MCK-PPARα), lipid-supported mitochondrial respiration, membrane potential (ΔΨ(m)), and H(2)O(2) production rate (JH(2)O(2)) were increased, which coincided with a more oxidized cytosolic redox environment, reduced muscle glucose uptake, and whole body glucose intolerance despite an increased rate of energy expenditure. Similar results were observed in lipin-1-deficient, fatty-liver dystrophic mice, another model characterized by increased β-oxidation flux and glucose intolerance. Crossing MCAT (mitochondria-targeted catalase) with MCK-PPARα mice normalized JH(2)O(2) production, redox environment, and glucose tolerance, but surprisingly, both basal and absolute insulin-stimulated rates of glucose uptake in muscle remained depressed. Also surprising, when placed on a high-fat diet, MCK-PPARα mice were characterized by much lower whole body, fat, and lean mass as well as improved glucose tolerance relative to wild-type mice, providing additional evidence that overexpression of PPARα in muscle imposes more extensive metabolic stress than experienced by wild-type mice on a high-fat diet. Overall, the findings suggest that driving an increase in skeletal muscle fatty acid oxidation in the absence of metabolic demand imposes mitochondrial reductive stress and elicits multiple counterbalance metabolic responses in an attempt to restore bioenergetic homeostasis. NEW & NOTEWORTHY Prior work has suggested that mitochondrial dysfunction is an underlying cause of insulin resistance in muscle because it limits fatty acid oxidation and therefore leads to the accumulation of cytotoxic lipid intermediates. The implication has been that therapeutic strategies to accelerate β-oxidation will be protective. The current study provides evidence that genetically increasing flux through β-oxidation in muscle imposes reductive stress that is not beneficial but rather detrimental to metabolic regulation
Reconstructing the Indian Origin and Dispersal of the European Roma: A Maternal Genetic Perspective
Previous genetic, anthropological and linguistic studies have shown that Roma (Gypsies) constitute a founder population dispersed throughout Europe whose origins might be traced to the Indian subcontinent. Linguistic and anthropological evidence point to Indo-Aryan ethnic groups from North-western India as the ancestral parental population of Roma. Recently, a strong genetic hint supporting this theory came from a study of a private mutation causing primary congenital glaucoma. In the present study, complete mitochondrial control sequences of Iberian Roma and previously published maternal lineages of other European Roma were analyzed in order to establish the genetic affinities among Roma groups, determine the degree of admixture with neighbouring populations, infer the migration routes followed since the first arrival to Europe, and survey the origin of Roma within the Indian subcontinent. Our results show that the maternal lineage composition in the Roma groups follows a pattern of different migration routes, with several founder effects, and low effective population sizes along their dispersal. Our data allowed the confirmation of a North/West migration route shared by Polish, Lithuanian and Iberian Roma. Additionally, eleven Roma founder lineages were identified and degrees of admixture with host populations were estimated. Finally, the comparison with an extensive database of Indian sequences allowed us to identify the Punjab state, in North-western India, as the putative ancestral homeland of the European Roma, in agreement with previous linguistic and anthropological studies
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