An origin-based model for unique shortest path routing

Link weights are the main parameters of shortest path routing protocols, the most commonly used protocols for IP networks. The problem of optimally setting link weights for unique shortest path routing is addressed. Due to the complexity of the constraints involved, there exist challenges to formulate the problem in such a way based on which a more efficient solution algorithm than the existing ones may be developed. In this paper, an exact formulation is first introduced and then mathematically proved correct. It is further illustrated that the formulation has advantages over a prior one in terms of both constraint structure and model size for a proposed decomposition method to solve the problem

Low-complexity iterative frequency domain decision feedback equalization

Single-carrier transmission with frequency domain equalization (SC-FDE) offers a viable design alternative to the classic orthogonal frequency division multiplexing technique. However, SC-FDE using a linear equalizer may suffer from serious performance deterioration for transmission over severely frequency-selective fading channels. An effective method of solving this problem is to introduce non-linear decision feedback equalization (DFE) to SC-FDE. In this contribution, a low complexity iterative decision feedback equalizer operating in the frequency domain of single-carrier systems is proposed. Based on the minimum mean square error criterion, a simplified parameter estimation method is introduced to calculate the coefficients of the feed-forward and feedback filters, which significantly reduces the implementation complexity of the equalizer. Simulation results show that the performance of the proposed simplified design is similar to the traditional iterative block DFE under various multipath fading channels but it imposes a much lower complexity than the latter

Weak euler scheme for lévy-driven stochastic differential equations

This paper studies the rate of convergence of the weak Euler approximation for solutions to Lévy-driven stochastic differential equations with nondegenerate main part driven by a spherically symmetric stable process, under the assumption of Hölder continuity. The rate of convergence is derived for a full regularity scale based on solving the associated backward Kolmogorov equation and investigating the dependence of the rate on the regularity of the coefficients and driving processes

MEASURING AND VISUALIZING USER SENTIMENT CHANGES OVER MULTIPLE CHANNELS

Techniques are provided to simultaneously infer approximately where a speaker is looking and the speaker\u27s emotion during a conversation. Due to privacy concerns, only the speaker\u27s approximate facial features may be estimated. The inferred face may be converted into a cartoon face that retains the main facial features. This may enhance user interaction experience even when a speaker does not turn on video in teleconference

Loss of ATF3 exacerbates liver damage through the activation of mTOR/p70S6K/ HIF-1α signaling pathway in liver inflammatory injury.

Activating transcription factor 3 (ATF3) is a stress-induced transcription factor that plays important roles in regulating immune and metabolic homeostasis. Activation of the mechanistic target of rapamycin (mTOR) and hypoxia-inducible factor (HIF) transcription factors are crucial for the regulation of immune cell function. Here, we investigated the mechanism by which the ATF3/mTOR/HIF-1 axis regulates immune responses in a liver ischemia/reperfusion injury (IRI) model. Deletion of ATF3 exacerbated liver damage, as evidenced by increased levels of serum ALT, intrahepatic macrophage/neutrophil trafficking, hepatocellular apoptosis, and the upregulation of pro-inflammatory mediators. ATF3 deficiency promoted mTOR and p70S6K phosphorylation, activated high mobility group box 1 (HMGB1) and TLR4, inhibited prolyl-hydroxylase 1 (PHD1), and increased HIF-1α activity, leading to Foxp3 downregulation and RORγt and IL-17A upregulation in IRI livers. Blocking mTOR or p70S6K in ATF3 knockout (KO) mice or bone marrow-derived macrophages (BMMs) downregulated HMGB1, TLR4, and HIF-1α and upregulated PHD1, increasing Foxp3 and decreasing IL-17A levels in vitro. Silencing of HIF-1α in ATF3 KO mice ameliorated IRI-induced liver damage in parallel with the downregulation of IL-17A in ATF3-deficient mice. These findings demonstrated that ATF3 deficiency activated mTOR/p70S6K/HIF-1α signaling, which was crucial for the modulation of TLR4-driven inflammatory responses and T cell development. The present study provides potential therapeutic targets for the treatment of liver IRI followed by liver transplantation

On the Rate of Convergence of Weak Euler Approximation for Nondegenerate SDEs Driven by Levy Processes

The paper studies the rate of convergence of the weak Euler approximation for solutions to SDEs driven by Levy processes, with Hoelder-continuous coefficients. It investigates the dependence of the rate on the regularity of coefficients and driving processes. The equation considered has a non-degenerate main part driven by a spherically-symmetric stable process