13 research outputs found
Chagas' disease: IgA, IgM and IgG antibodies to T. cruzi amastigote, trypomastigote and epimastigote antigens in acute and in different chronic forms of the disease
Full text linkMicro-angiopathic haemolysis, thrombocytopenia and nephrotic syndrome associated with membranous nephropathy in a Vietnamese boy
No full textRole of Trypanosoma cruzi Autoreactive T Cells in the Generation of Cardiac Pathology
No full textChagas disease, caused by Trypanosoma cruzi, affects several million people in Central and South America. About 30% of chronic patients develop cardiomyopathy probably caused by parasite persistence and/or autoimmunity. While several cross-reactive antibodies generated during mammal T. cruzi infection have been described, very few cross-reactive T cells have been identified. We performed adoptive transfer experiments of T cells isolated from chronically infected mice. The results showed the generation of cardiac pathology in the absence of parasites. We also transferred cross-reactive SAPA-specific T cells and observed unspecific alterations in heart repolarization, cardiac inflammatory infiltration, and tissue damage.S
Complement-fixing ability of antinuclear factors. Studies in adult and juvenile rheumatoid arthritis and systemic lupus erythematosus.
No full textModulation of Both Interleukin 2 Receptor Expression and Interleukin 2 Production during Experimental Murine Trypanosoma cruzi Infection
No full textDirect lysis of Trypanosoma cruzi: a novel effector mechanism of protection mediated by human anti-gal antibodies
No full textAntibody-dependent cytotoxicity of Trypanosoma cruzi antigen-coated mouse cell lines by eosinophils and neutrophils
No full textPrevention of electrocardiographic and histopathologic alterations in the murine model of Chagas' disease by preinoculation of an attenuated Trypanosoma cruzi strain
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