Avaliação de cultivares de milho nas regiões nordeste e oeste do Pará.

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Avaliação dos impactos econômicos de quatro forrageiras lançadas pela Embrapa.

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Feijoeiro comum: opção de sustentabilidade para agricultura familiar no Estado de Goiás.

O objetivo deste trabalho foi avaliar a viabilidade de diferentes cultivares e linhagens de feijoeiro-comum ao sistema de produção dos pequenos agricultores no Estado de Goiás. Para isso foram implementados ensaios de VCU em propriedades de agricultores familiares nos municípios de Rubiataba-GO e Ipiranga-GO

Refinement of Monoclinic ReO 2 Structure from XRD by Rietveld Method

ReO 2 presents two crystalline variants, with monoclinic and orthorhombic structures. The former is metastable and irreversibly transforms to an orthorhombic structure above 460 o C. The structure of the latter was determined from studies on monocrystalline samples, whereas for the monoclinic variant there are no single crystals available so far. It was found only one monoclinic variant and the structure associated with this variant is based on studies on polycrystals. We analyzed a monoclinic oxide powder sample by X-ray diffraction and refined its pattern by means of the Rietveld Method. We obtained that the monoclinic variant belongs to space group P 21/c, with lattice parameters a = 5.615(3), b = 4.782(2), c = 5.574(2)Å, β = 120.1

Infections with Avian Pathogenic and Fecal Escherichia coli Strains Display Similar Lung Histopathology and Macrophage Apoptosis

The purpose of this study was to compare histopathological changes in the lungs of chickens infected with avian pathogenic (APEC) and avian fecal (Afecal) Escherichia coli strains, and to analyze how the interaction of the bacteria with avian macrophages relates to the outcome of the infection. Chickens were infected intratracheally with three APEC strains, MT78, IMT5155, and UEL17, and one non-pathogenic Afecal strain, IMT5104. The pathogenicity of the strains was assessed by isolating bacteria from lungs, kidneys, and spleens at 24 h post-infection (p.i.). Lungs were examined for histopathological changes at 12, 18, and 24 h p.i. Serial lung sections were stained with hematoxylin and eosin (HE), terminal deoxynucleotidyl dUTP nick end labeling (TUNEL) for detection of apoptotic cells, and an anti-O2 antibody for detection of MT78 and IMT5155. UEL17 and IMT5104 did not cause systemic infections and the extents of lung colonization were two orders of magnitude lower than for the septicemic strains MT78 and IMT5155, yet all four strains caused the same extent of inflammation in the lungs. The inflammation was localized; there were some congested areas next to unaffected areas. Only the inflamed regions became labeled with anti-O2 antibody. TUNEL labeling revealed the presence of apoptotic cells at 12 h p.i in the inflamed regions only, and before any necrotic foci could be seen. The TUNEL-positive cells were very likely dying heterophils, as evidenced by the purulent inflammation. Some of the dying cells observed in avian lungs in situ may also be macrophages, since all four avian E. coli induced caspase 3/7 activation in monolayers of HD11 avian macrophages. In summary, both pathogenic and non-pathogenic fecal strains of avian E. coli produce focal infections in the avian lung, and these are accompanied by inflammation and cell death in the infected areas