118 research outputs found
Can tonne-scale direct detection experiments discover nuclear dark matter?
Models of nuclear dark matter propose that the dark sector contains large
composite states consisting of dark nucleons in analogy to Standard Model
nuclei. We examine the direct detection phenomenology of a particular class of
nuclear dark matter model at the current generation of tonne-scale liquid noble
experiments, in particular DEAP-3600 and XENON1T. In our chosen nuclear dark
matter scenario distinctive features arise in the recoil energy spectra due to
the non-point-like nature of the composite dark matter state. We calculate the
number of events required to distinguish these spectra from those of a standard
point-like WIMP state with a decaying exponential recoil spectrum. In the most
favourable regions of nuclear dark matter parameter space, we find that a few
tens of events are needed to distinguish nuclear dark matter from WIMPs at the
level in a single experiment. Given the total exposure time of
DEAP-3600 and XENON1T we find that at best a distinction is
possible by these experiments individually, while sensitivity is
reached for a range of parameters by the combination of the two experiments. We
show that future upgrades of these experiments have potential to distinguish a
large range of nuclear dark matter models from that of a WIMP at greater than
.Comment: 23 pages, 7 multipanel figure
Simulated Milky Way analogues: implications for dark matter direct searches
We study the implications of galaxy formation on dark matter direct detection using high resolution hydrodynamic simulations of Milky Way-like galaxies simulated within the eagle and apostle projects. We identify MilkyWay analogues that satisfy observational constraints on the Milky Way rotation curve and total stellar mass. We then extract the dark matter density and velocity distribution in the Solar neighbourhood for this set of Milky Way analogues, and use them to analyse the results of current direct detection experiments. For most Milky Way analogues, the event rates in direct detection experiments obtained from the best _t Maxwellian distribution (with peak speed of 223 { 289 km=s) are similar to those obtained directly from the simulations. As a consequence, the allowed regions and exclusion limits set by direct detection experiments in the dark matter mass and spin-independent cross section plane shift by a few GeV compared to the Standard Halo Model, at low dark matter masses. For each dark matter mass, the halo-to-halo variation of the local dark matter density results in an overall shift of the allowed regions and exclusion limits for the cross section. However, the compatibility of the possible hints for a dark matter signal from
DAMA and CDMS-Si and null results from LUX and SuperCDMS is not improved
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Epstein-Barr virus: clinical and epidemiological revisits and genetic basis of oncogenesis
Epstein-Barr virus (EBV) is classified as a member in the order herpesvirales, family herpesviridae, subfamily gammaherpesvirinae and the genus lymphocytovirus. The virus is an exclusively human pathogen and thus also termed as human herpesvirus 4 (HHV4). It was the first oncogenic virus recognized and has been incriminated in the causation of tumors of both lymphatic and epithelial nature. It was reported in some previous studies that 95% of the population worldwide are serologically positive to the virus. Clinically, EBV primary infection is almost silent, persisting as a life-long asymptomatic latent infection in B cells although it may be responsible for a transient clinical syndrome called infectious mononucleosis. Following reactivation of the virus from latency due to immunocompromised status, EBV was found to be associated with several tumors. EBV linked to oncogenesis as detected in lymphoid tumors such as Burkitt's lymphoma (BL), Hodgkin's disease (HD), post-transplant lymphoproliferative disorders (PTLD) and T-cell lymphomas (e.g. Peripheral T-cell lymphomas; PTCL and Anaplastic large cell lymphomas; ALCL). It is also linked to epithelial tumors such as nasopharyngeal carcinoma (NPC), gastric carcinomas and oral hairy leukoplakia (OHL). In vitro, EBV many studies have demonstrated its ability to transform B cells into lymphoblastoid cell lines (LCLs). Despite these malignancies showing different clinical and epidemiological patterns when studied, genetic studies have suggested that these EBV- associated transformations were characterized generally by low level of virus gene expression with only the latent virus proteins (LVPs) upregulated in both tumors and LCLs. In this review, we summarize some clinical and epidemiological features of EBV- associated tumors. We also discuss how EBV latent genes may lead to oncogenesis in the different clinical malignancie
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