Are the media globalizing political discourse? The war on terrorism case study

The paper challenges the claim that an increasingly global media is creating a homogenisation of political discourses at the international level. In particular, it explores the extent to which the U.S. government managed to affect global perceptions of the War On Terrorism through the media in the immediate aftermath of the 9/11 events. The research starts from the consideration that the U.S. government created, through the repetition of consistent messages, a very specific interpretation of the 9/11 events (a War On Terrorism frame) and attempted to export it globally in order to support its own foreign policy objectives. The analysis then focuses on the comparison between the War On Terrorism frame as delivered by the U.S. government and its reproduction within both the political and media discourses in a range of local cases at the international level. They include the U.S., France, Italy and Pakistan. The research questions current literature on globalisation by drawing on political communications’ framing theory. More specifically, it suggests first that there is no evidence of an on-going globalisation of either political or media discourses; secondly that the local nation-state level plays a key role in understanding the mechanisms of frames’ spreading at the global level; and thirdly that national culture is a major determinant in defining local political and media discourses’ contents, even in presence of a strong persuasion attempt by a powerful international actor such as the U.S. government

Unamerican Views: Why US-developed models of press-state relations don't apply to the rest of the world

The article shows the limitations of the 'indexing' hypothesis, an influential conceptualization of state-press relations based on the notion that the media tend to reproduce the range of debate within political elites. The hypothesis, as confirmed by an international comparative investigation of the elite press coverage of 9/11 in the US, Italy, France, and Pakistan, cannot be applied outside the American context. The analysis finds that the variation in the levels of correlation between elite press coverage and governmental discourse are explained by previously neglected variables: national interest, national journalistic culture, and editorial policy within each media organization. The article argues that more international comparative research and multidisciplinary approaches are needed in order to renew old paradigms, especially at a time when the distinction between foreign and domestic politics is disappearing

Rebranding terror

Ten years after its most devastating attack, al-Qaeda has turned into a franchiser, publisher, and occasional climate-change activist. Can the world’s most deadly terrorist group go mainstream and keep its edge

A multidisciplinary understanding of news: Comparing elite press framing of 9/11 in the US, Italy, France and Pakistan

Political Communications, International Communications, News Sociology, all claim to offer an explanation for what shapes the news, but provide extremely different, if not contradictory suggestions. Political communications almost takes for granted the fact that official actors have a major role in shaping news stories at the national level. International Communications points at several possibilities: structural economic imbalances lead to unidirectional news flows from rich countries towards poor countries; globalization causes news to become homogenised on a worldwide scale; news is geared to the tastes of local audiences by national news producers. News sociology, instead, argues that the news product of each media organization is the unique output of patterns of social interactions among media professionals. An international comparative study of the elite press framing of 9/11 in the US, Italy, France, and Pakistan reveals the limits of these approaches: none of them alone is able to explain the patterns of news contents that were detected in the empirical investigation.The analysis suggests that the content of press coverage in the newspapers under analysis is more effectively explained in terms of selection of newsworthy sources, guided by national interest, journalistic culture, and editorial policy. The study points to the benefit of adopting international comparative research designs and fundamentally argues that, if we want to explain news in the information age, we need to approach its study in a multidisciplinary perspective

Reoptimization of the Maximum Weighted P k -Free Subgraph Problem under Vertex Insertion

International audienceThe reoptimization issue studied in this paper can be described as follows: given an instance I of some problem Π, an optimal solution OPT for Π in I and an instance I′ resulting from a local perturbation of I that consists of insertions or removals of a small number of data, we wish to use OPT in order to solve Π in I′, either optimally or by guaranteeing an approximation ratio better than that guaranteed by an ex nihilo computation and with running time better than that needed for such a computation. In this setting we study the weighted version of max weighted P k -free subgraph. We then show, how the technique we use allows us to handle also bin packing

Reoptimization of Some Maximum Weight Induced Hereditary Subgraph Problems

The reoptimization issue studied in this paper can be described as follows: given an instance I of some problem Π, an optimal solution OPT for Π in I and an instance I′ resulting from a local perturbation of I that consists of insertions or removals of a small number of data, we wish to use OPT in order to solve Π in I', either optimally or by guaranteeing an approximation ratio better than that guaranteed by an ex nihilo computation and with running time better than that needed for such a computation. We use this setting in order to study weighted versions of several representatives of a broad class of problems known in the literature as maximum induced hereditary subgraph problems. The main problems studied are max independent set, max k-colorable subgraph and max split subgraph under vertex insertions and deletion

Intronic CYP46 polymorphism along with ApoE genotype in sporadic Alzheimer Disease: from risk factors to disease modulators

Increasing biological and clinical findings argue for a link between brain cholesterol turnover and Alzheimer Disease (AD), high cerebral levels of the former increasing Abeta load. Cerebral cholesterol elimination involves two mechanisms dependent on Apolipoprotein E (ApoE) and cholesterol 24-hydroxylase (CYP46). The aim of this study was to evaluate an intronic variation in CYP46 (intron 2, T --> C ) along with ApoE genotype as risk factors for AD and to establish the correlation between CYP46/ApoE polymorphism and disease progression. One-hundred and fifty-seven AD patients, who had been followed periodically through 1-year follow-up after enrollment, and 134 age- and gender-matched controls entered the study. The distribution of CYP46 genotypes was significantly different in AD compared to controls (P<0.004), being CYP*C allele higher in AD patients ( P<0.002). ApoE 4 genotype was more frequent in AD (41.4%) than in controls (15.9%, P<0.0001). The odds ratio (OR) for AD risk in CYP46*C carriers was 2.8, and in ApoE epsilon4 carriers was 4.05; the OR for having both CYP46*C and ApoE epsilon4 was 17.75, demonstrating the their synergic effect on AD risk. In AD patients, CYP46*C along with ApoE epsilon4 genotype were associated with a higher cognitive decline at 1-year follow-up (P<0.02). These findings provide direct evidence that CYP46 and ApoE polymorphisms synergically increase the risk for AD development, and influence on the rate of cognitive decline

Evolutionary dynamics of tumor-stroma interactions in multiple myeloma

Cancer cells and stromal cells cooperate by exchanging diffusible factors that sustain tumor growth, a form of frequency-dependent selection that can be studied in the framework of evolutionary game theory. In the case of multiple myeloma, three types of cells (malignant plasma cells, osteoblasts and osteoclasts) exchange growth factors with different effects, and tumor-stroma interactions have been analysed using a model of cooperation with pairwise interactions. Here we show that a model in which growth factors have autocrine and paracrine effects on multiple cells, a more realistic assumption for tumor-stroma interactions, leads to different results, with implications for disease progression and treatment. In particular, the model reveals that reducing the number of malignant plasma cells below a critical threshold can lead to their extinction and thus to restore a healthy balance between osteoclast and osteoblast, a result in line with current therapies against multiple myeloma