388 research outputs found
Strong spin-photon coupling in silicon
We report the strong coupling of a single electron spin and a single
microwave photon. The electron spin is trapped in a silicon double quantum dot
and the microwave photon is stored in an on-chip high-impedance superconducting
resonator. The electric field component of the cavity photon couples directly
to the charge dipole of the electron in the double dot, and indirectly to the
electron spin, through a strong local magnetic field gradient from a nearby
micromagnet. This result opens the way to the realization of large networks of
quantum dot based spin qubit registers, removing a major roadblock to scalable
quantum computing with spin qubits
Electrochemical study of H3PMo12 retention on Vulcan carbon grafted with NH2 and OH groups
In this work, we show a comparative study based on the effects of specific chemical functional groups (–OH, –NH2), grafted on Vulcan carbon (VC) with the incorporation of a specific polyoxometalate (POM), PMo12 (H3PMo12O40), to improve electrochemical performance. We observed a decrease in the specific surface area of the grafted matrices (VC-OH and VC-NH2) [1], and the same trend was observed for PMo12 (POM) incorporation. Our electrochemical studies showed low concentrations of POM in unmodified VCs and higher POM concentrations for grafted matrices (VC-OH and VC-NH2) after 500 voltammetric cycles, especially for the VC grafted with –OH groups (VC-OH-POM). Mechanisms have been proposed for POM interaction with the grafted groups in carbon, emphasizing the role of aqueous medium and redox activity of POM. Cyclic voltammograms suggested the POM anchoring through –OH groups with a strong interaction as a covalent bond, resulting in a surface coverage of 1.66 × 10−11 mol cm−2. Surface modifications could be extrapolated to other carbons, and the materials could be employed for different potential applications such as photocatalysis, amperometric sensors, fuel cells, and supercapacitors.Fil: Cuentas Gallegos, Ana Karina. Universidad Nacional Autónoma de México; México. Consejo Nacional de Investigaciones CientÃficas y Técnicas; ArgentinaFil: López Cortina, S.. Universidad Autonoma de Nuevo Leon; MéxicoFil: Brousse, T.. Université de Nantes. Institut Des Materiaux Jean Rouxel; FranciaFil: Pacheco Catalán, D.. Centro de Investigación CientÃfica de Yucatán; MéxicoFil: Fuentes Quezada, Eduardo. Centro de Investigación y Desarrollo Tecnológico en ElectroquÃmica; México. Universidad Nacional Autónoma de México; México. Consejo Nacional de Investigaciones CientÃficas y Técnicas; ArgentinaFil: Mosqueda, H.. Universidad Autonoma de Nuevo Leon; MéxicoFil: Orozco Gamboa, G.. Centro de Investigación y Desarrollo Tecnológico en ElectroquÃmica; Méxic
Cytophagic histiocytic panniculitis: is it a macrophage activation syndrome in situ?
International audiencepas de résum
Small bowel adenocarcinoma in a patient with Coeliac disease: A case report
Coeliac disease is a chronic inflammatory disease of the gut with increased risk of gastrointestinal malignancy. Although enteropathy T-lymphoma is the most common neoplasm in patient affected by coeliac disease, an increased frequency of small bowel carcinoma has been described. We present a case of jejunal carcinoma in a patient suffering for coeliac disease in which gastrointestinal and extraintestinal symptoms of disease developed although he was treated with a gluten-free diet
A psicanálise aplicada ao sintoma: uma resposta ética aos impasses enfrentados pelos psicanalistas na atualidade
Long-term follow up of the FL2000 study comparing CHVP-interferon to CHVP-interferon plus rituximab in follicular lymphoma
Development and characterization of acid-doped polybenzimidazole/sulfonated polysulfone blend polymer electrolytes for fuel cells
Activation-Induced Cytidine Deaminase (AID) Deficiency Causes the Autosomal Recessive Form of the Hyper-IgM Syndrome (HIGM2)
AbstractThe activation-induced cytidine deaminase (AID) gene, specifically expressed in germinal center B cells in mice, is a member of the cytidine deaminase family. We herein report mutations in the human counterpart of AID in patients with the autosomal recessive form of hyper-IgM syndrome (HIGM2). Three major abnormalities characterize AID deficiency: (1) the absence of immunoglobulin class switch recombination, (2) the lack of immunoglobulin somatic hypermutations, and (3) lymph node hyperplasia caused by the presence of giant germinal centers. The phenotype observed in HIGM2 patients (and in AID−/− mice) demonstrates the absolute requirement for AID in several crucial steps of B cell terminal differentiation necessary for efficient antibody responses
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