Narratives of Feminist Resistance: Women\u27s Bodily Autonomy and the Dystopian Mode

This undergraduate thesis examines how dystopian fiction has responded to the sociopolitical issue of restrictions on women’s bodily autonomy, a question that has become more timely since the reversal of Roe v. Wade in Summer 2022. Particularly, I aim to understand how readers can use dystopian novels to shape real-world dialogue and how authors can use narrative strategies to encourage readers to resist oppression. My first chapter takes a broad approach, tracing the development of dystopian fiction from a genre to a mode and using Marge Piercy’s Woman on the Edge of Time (1976) as a case study of how an author can use the dystopian mode to model resistance. My second chapter analyzes how, with its plausible predictions and intimate first-person stream-of-consciousness narrative style, Margaret Atwood’s The Handmaid’s Tale (1985) has become a cultural intervention that many women still turn to as a symbol of protest almost four decades after its publication. Finally, my third chapter considers how Octavia Butler’s Dawn (1987) has become a feminist resistor’s narrative–especially for Black women like its protagonist, Lilith, who resists the aftereffects of slavery as well as patriarchal oppression–that shows readers how women can gain power, especially by using their voice in protest, exhibiting empathy, and never losing hope. My thesis is prefaced and concluded by a personal memoir, tracing my identity as a feminist in twenty-first century America

Resumption of mass accretion in RS Oph

The latest outburst of the recurrent nova RS Oph occurred in 2006 February. Photometric data presented here show evidence of the resumption of optical flickering, indicating re-establishment of accretion by day 241 of the outburst. Magnitude variations of up to 0.32 mag in V band and 0.14 mag in B band on time-scales of 600–7000 s are detected. Over the two-week observational period, we also detect a 0.5 mag decline in the mean brightness, from V≈ 11.4 to 11.9, and record B≈ 12.9 mag. Limits on the mass accretion rate of [inline image] are calculated, which span the range of accretion rates modelled for direct wind accretion and Roche lobe overflow mechanisms. The current accretion rates make it difficult for thermonuclear runaway models to explain the observed recurrence interval, and this implies average accretion rates are typically higher than seen immediately post-outburst

A Scattered Light Echo around SN 1993J in M81

A light echo around SN 1993J was observed 8.2 years after explosion by a HST WFPC2 observation, adding to the small family of supernovae with light echoes. The light echo was formed by supernova light scattered from a dust sheet, which lies 220 parsecs away from the supernova, 50 parsecs thick along the line of sight, as inferred from radius and width of the light echo. The dust inferred from the light echo surface brightness is 1000 times denser than the intercloud dust. The graphite to silicate fraction can not be determined by our BVI photometric measurements, however, a pure graphite model can be excluded based on comparison with the data. With future observations, it will be possible to measure the expansion rate of the light echo, from which an independent distance to M81 can be obtained.Comment: 10 pages, 6 figures, in AASTeX format, submitted to ApJ Part

Suppression of mitochondrial respiration through recruitment of p160 myb binding protein to PGC-1α : modulation by p38 MAPK

The transcriptional coactivator PPAR gamma coactivator 1 α (PGC-1α) is a key regulator of metabolic processes such as mitochondrial biogenesis and respiration in muscle and gluconeogenesis in liver. Reduced levels of PGC-1α in humans have been associated with type II diabetes. PGC-1α contains a negative regulatory domain that attenuates its transcriptional activity. This negative regulation is removed by phosphorylation of PGC-1α by p38 MAPK, an important kinase downstream of cytokine signaling in muscle and β-adrenergic signaling in brown fat. We describe here the identification of p160 myb binding protein (p160MBP) as a repressor of PGC-1α. The binding and repression of PGC-1α by p160MBP is disrupted by p38 MAPK phosphorylation of PGC-1α. Adenoviral expression of p160MBP in myoblasts strongly reduces PGC-1α's ability to stimulate mitochondrial respiration and the expression of the genes of the electron transport system. This repression does not require removal of PGC-1α from chromatin, suggesting that p160MBP is or recruits a direct transcriptional suppressor. Overall, these data indicate that p160MBP is a powerful negative regulator of PGC-1α function and provide a molecular mechanism for the activation of PGC-1α by p38 MAPK. The discovery of p160MBP as a PGC-1α regulator has important implications for the understanding of energy balance and diabetes