74 research outputs found
Advances in the proposed electromagnetic zero-point field theory of inertia
A NASA-funded research effort has been underway at the Lockheed Martin
Advanced Technology Center in Palo Alto and at California State University in
Long Beach to develop and test a recently published theory that Newton's
equation of motion can be derived from Maxwell's equations of electrodynamics
as applied to the zero-point field (ZPF) of the quantum vacuum. In this
ZPF-inertia theory, mass is postulated to be not an intrinsic property of
matter but rather a kind of electromagnetic drag force that proves to be
acceleration dependent by virtue of the spectral characteristics of the ZPF.
The theory proposes that interactions between the ZPF and matter take place at
the level of quarks and electrons, hence would account for the mass of a
composite neutral particle such as the neutron. An effort to generalize the
exploratory study of Haisch, Rueda and Puthoff (1994) into a proper
relativistic formulation has been successful. Moreover the principle of
equivalence implies that in this view gravitation would also be electromagnetic
in origin along the lines proposed by Sakharov (1968). With regard to exotic
propulsion we can definitively rule out one speculatively hypothesized
mechanism: matter possessing negative inertial mass, a concept originated by
Bondi (1957) is shown to be logically impossible. On the other hand, the linked
ZPF-inertia and ZPF-gravity concepts open the conceptual possibility of
manipulation of inertia and gravitation, since both are postulated to be
electromagnetic phenomena. It is hoped that this will someday translate into
actual technological potential. A key question is whether the proposed
ZPF-matter interactions generating the phenomenon of mass might involve one or
more resonances. This is presently under investigation.Comment: Revised version of invited presentation at 34th AIAA/ASME/SAE/ASEE
Joint Propulsion Conference, July 13-15, 1998, Cleveland, OH, 10 pages, no
figure
The Zero-Point Field and Inertia
A brief overview is presented of the basis of the electromagnetic zero-point
field in quantum physics and its representation in stochastic electrodynamics.
Two approaches have led to the proposal that the inertia of matter may be
explained as an electromagnetic reaction force. The first is based on the
modeling of quarks and electrons as Planck oscillators and the method of
Einstein and Hopf to treat the interaction of the zero-point field with such
oscillators. The second approach is based on analysis of the Poynting vector of
the zero-point field in accelerated reference frames. It is possible to derive
both Newton's equation of motion, F=ma, and its relativistic co-variant form
from Maxwell's equations as applied to the zero-point field of the quantum
vacuum. This appears to account, at least in part, for the inertia of matter.Comment: 8 pages, no fig
Roles of Nutrient Limitation on Western Lake Erie CyanoHAB Toxin Production
Cyanobacterial harmful algal bloom (CyanoHAB) proliferation is a global problem impacting ecosystem and human health. Western Lake Erie (WLE) typically endures two highly toxic CyanoHABs during summer: a Microcystis spp. bloom in Maumee Bay that extends throughout the western basin, and a Planktothrix spp. bloom in Sandusky Bay. Recently, the USA and Canada agreed to a 40% phosphorus (P) load reduction to lessen the severity of the WLE blooms. To investigate phosphorus and nitrogen (N) limitation of biomass and toxin production in WLE CyanoHABs, we conducted in situ nutrient addition and 40% dilution microcosm bioassays in June and August 2019. During the June Sandusky Bay bloom, biomass production as well as hepatotoxic microcystin and neurotoxic anatoxin production were N and P co-limited with microcystin production becoming nutrient deplete under 40% dilution. During August, the Maumee Bay bloom produced microcystin under nutrient repletion with slight induced P limitation under 40% dilution, and the Sandusky Bay bloom produced anatoxin under N limitation in both dilution treatments. The results demonstrate the importance of nutrient limitation effects on microcystin and anatoxin production. To properly combat cyanotoxin and cyanobacterial biomass production in WLE, both N and P reduction efforts should be implemented in its watershed
Severe neurological outcomes after very early bilateral nephrectomies in patients with autosomal recessive polycystic kidney disease (ARPKD)
To test the association between bilateral nephrectomies in patients with autosomal recessive polycystic kidney disease (ARPKD) and long-term clinical outcome and to identify risk factors for severe outcomes, a dataset comprising 504 patients from the international registry study ARegPKD was analyzed for characteristics and complications of patients with very early (� 3 months; VEBNE) and early (4�15 months; EBNE) bilateral nephrectomies. Patients with very early dialysis (VED, onset � 3 months) without bilateral nephrectomies and patients with total kidney volumes (TKV) comparable to VEBNE infants served as additional control groups. We identified 19 children with VEBNE, 9 with EBNE, 12 with VED and 11 in the TKV control group. VEBNE patients suffered more frequently from severe neurological complications in comparison to all control patients. Very early bilateral nephrectomies and documentation of severe hypotensive episodes were independent risk factors for severe neurological complications. Bilateral nephrectomies within the first 3 months of life are associated with a risk of severe neurological complications later in life. Our data support a very cautious indication of very early bilateral nephrectomies in ARPKD, especially in patients with residual kidney function, and emphasize the importance of avoiding severe hypotensive episodes in this at-risk cohort. © 2020, The Author(s)
Vascular Remodeling in Health and Disease
The term vascular remodeling is commonly used to define the structural changes in blood vessel geometry that occur in response to long-term physiologic alterations in blood flow or in response to vessel wall injury brought about by trauma or underlying cardiovascular diseases.1, 2, 3, 4 The process of remodeling, which begins as an adaptive response to long-term hemodynamic alterations such as elevated shear stress or increased intravascular pressure, may eventually become maladaptive, leading to impaired vascular function. The vascular endothelium, owing to its location lining the lumen of blood vessels, plays a pivotal role in regulation of all aspects of vascular function and homeostasis.5 Thus, not surprisingly, endothelial dysfunction has been recognized as the harbinger of all major cardiovascular diseases such as hypertension, atherosclerosis, and diabetes.6, 7, 8 The endothelium elaborates a variety of substances that influence vascular tone and protect the vessel wall against inflammatory cell adhesion, thrombus formation, and vascular cell proliferation.8, 9, 10 Among the primary biologic mediators emanating from the endothelium is nitric oxide (NO) and the arachidonic acid metabolite prostacyclin [prostaglandin I2 (PGI2)], which exert powerful vasodilatory, antiadhesive, and antiproliferative effects in the vessel wall
Zero point field (ZPF) effects in the interaction of the ultra-short laser pulses with matter
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