2,831 research outputs found

    Using a cognitive architecture to examine what develops

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    Different theories of development propose alternative mechanisms by which development occurs. Cognitive architectures can be used to examine the influence of each proposed mechanism of development while keeping all other mechanisms constant. An ACT-R computational model that matched adult behavior in solving a 21-block pyramid puzzle was created. The model was modified in three ways that corresponded to mechanisms of development proposed by developmental theories. The results showed that all the modifications (two of capacity and one of strategy choice) could approximate the behavior of 7-year-old children on the task. The strategy-choice modification provided the closest match on the two central measures of task behavior (time taken per layer, r = .99, and construction attempts per layer, r = .73). Modifying cognitive architectures is a fruitful way to compare and test potential developmental mechanisms, and can therefore help in specifying “what develops.

    Off-Critical Logarithmic Minimal Models

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    We consider the integrable minimal models M(m,m;t){\cal M}(m,m';t), corresponding to the φ1,3\varphi_{1,3} perturbation off-criticality, in the {\it logarithmic limit\,} m,mm, m'\to\infty, m/mp/pm/m'\to p/p' where p,pp, p' are coprime and the limit is taken through coprime values of m,mm,m'. We view these off-critical minimal models M(m,m;t){\cal M}(m,m';t) as the continuum scaling limit of the Forrester-Baxter Restricted Solid-On-Solid (RSOS) models on the square lattice. Applying Corner Transfer Matrices to the Forrester-Baxter RSOS models in Regime III, we argue that taking first the thermodynamic limit and second the {\it logarithmic limit\,} yields off-critical logarithmic minimal models LM(p,p;t){\cal LM}(p,p';t) corresponding to the φ1,3\varphi_{1,3} perturbation of the critical logarithmic minimal models LM(p,p){\cal LM}(p,p'). Specifically, in accord with the Kyoto correspondence principle, we show that the logarithmic limit of the one-dimensional configurational sums yields finitized quasi-rational characters of the Kac representations of the critical logarithmic minimal models LM(p,p){\cal LM}(p,p'). We also calculate the logarithmic limit of certain off-critical observables Or,s{\cal O}_{r,s} related to One Point Functions and show that the associated critical exponents βr,s=(2α)Δr,sp,p\beta_{r,s}=(2-\alpha)\,\Delta_{r,s}^{p,p'} produce all conformal dimensions Δr,sp,p<(pp)(9pp)4pp\Delta_{r,s}^{p,p'}<{(p'-p)(9p-p')\over 4pp'} in the infinitely extended Kac table. The corresponding Kac labels (r,s)(r,s) satisfy (pspr)2<8p(pp)(p s-p' r)^2< 8p(p'-p). The exponent 2α=p2(pp)2-\alpha ={p'\over 2(p'-p)} is obtained from the logarithmic limit of the free energy giving the conformal dimension Δt=1α2α=2ppp=Δ1,3p,p\Delta_t={1-\alpha\over 2-\alpha}={2p-p'\over p'}=\Delta_{1,3}^{p,p'} for the perturbing field tt. As befits a non-unitary theory, some observables Or,s{\cal O}_{r,s} diverge at criticality.Comment: 18 pages, 5 figures; version 3 contains amplifications and minor typographical correction

    Adolescent Trajectories of Aerobic Fitness and Adiposity as Markers of Cardiometabolic Risk in Adulthood

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    Purpose. The aim of this study was to investigate whether adolescent growth trajectories of aerobic fitness and adiposity were associated with mid-adulthood cardiometabolic risk (CMR). Methods. Participants were drawn from the Saskatchewan Growth and Development Study (1963–1973). Adolescent growth trajectories for maximal aerobic capacity (absolute VO2 (AbsVO2)), skinfolds (SF), representing total body (Sum6SF) and central adiposity (TrunkSF), and body mass index (BMI) were determined from 7 to 17 years of age. In mid-adulthood (40 to 50 years of age), 61 individuals (23 females) returned for follow-ups. A CMR score was calculated to group participants as displaying either high or a low CMR. Multilevel hierarchical models were constructed, comparing the adolescent growth trajectories of AbsVO2, Sum6SF, TrunkSF, and BMI between CMR groupings. Results. There were no significant differences in the adolescent development of AbsVO2, Sum6SF, TrunkSF, and BMI between adult CMR groupings (p>0.05). Individuals with high CMR accrued 62% greater adjusted total body fat percentage from adolescence to adulthood (p=0.03). Conclusions. Growth trajectories of adolescent aerobic fitness and adiposity do not appear to be associated with mid-adulthood CMR. Individuals should be encouraged to participate in behaviours that promote healthy aerobic fitness and adiposity levels throughout life to reduce lifelong CMR

    The Halo Mass of Optically Luminous Quasars at z ,F≈ ,F1-2 Measured via Gravitational Deflection of the Cosmic Microwave Background

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    © 2019. The American Astronomical Society. All rights reserved.We measure the average deflection of cosmic microwave background photons by quasars at 〈Z〉= 1.7. Our sample is selected from the Sloan Digital Sky Survey to cover the redshift range 0.9 ≤z≤2.2 with absolute i-band magnitudes of M i ≤-24 (K-corrected to z = 2). A stack of nearly 200,000 targets reveals an 8δ detection of Planck's estimate of the lensing convergence toward the quasars. We fit the signal with a model comprising a Navarro-Frenk-White density profile and a two-halo term accounting for correlated large-scale structure, which dominates the observed signal. The best-fitting model is described by an average halo mass log 10 (M h h -1 M)12.6 ±0.2 = and linear bias b=2.7±0.3 at 〈Z 〉= 1.7, in excellent agreement with clustering studies. We also report a hint, at a 90% confidence level, of a correlation between the convergence amplitude and luminosity, indicating that quasars brighter than Mi≲ -26 reside in halos of typical mass M h ≈ 10 13 h -1 M, scaling roughly as M h ∞ L opt 3/4 at M i ≲-24 mag, in good agreement with physically motivated quasar demography models. Although we acknowledge that this luminosity dependence is a marginal result, the observed Mh-L opt relationship could be interpreted as a reflection of the cutoff in the distribution of black hole accretion rates toward high Eddington ratios: the weak trend of Mh with Lopt observed at low luminosity becomes stronger for the most powerful quasars, which tend to be accreting close to the Eddington limit.Peer reviewedFinal Accepted Versio

    Baxterization, dynamical systems, and the symmetries of integrability

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    We resolve the `baxterization' problem with the help of the automorphism group of the Yang-Baxter (resp. star-triangle, tetrahedron, \dots) equations. This infinite group of symmetries is realized as a non-linear (birational) Coxeter group acting on matrices, and exists as such, {\em beyond the narrow context of strict integrability}. It yields among other things an unexpected elliptic parametrization of the non-integrable sixteen-vertex model. It provides us with a class of discrete dynamical systems, and we address some related problems, such as characterizing the complexity of iterations.Comment: 25 pages, Latex file (epsf style). WARNING: Postscript figures are BIG (600kB compressed, 4.3MB uncompressed). If necessary request hardcopy to [email protected] and give your postal mail addres

    Adolescent biological maturity and physical activity: biology meets behavior

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    The decline in physical activity (PA) across adolescence is well established but influence of biological maturity on the process has been largely overlooked. This paper reviews the limited number of studies which examine the relationship between timing of biological maturity and PA. Results are generally inconsistent among studies. Other health-related behaviors are also considered in an effort to highlight the complexity of relationships between biological maturation and behavior and to provide future research directions.</jats:p

    James Hutton’s geological tours of Scotland : romanticism, literary strategies, and the scientific quest

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    This article explores a somewhat neglected part of the story of the emergence of geology as a science and discourse in the late eighteenth century – James Hutton’s posthumously published accounts of the geological tours of Scotland that he undertook in the years 1785 to 1788 in search of empirical evidence in support of his theory of the Earth and that he intended to include in the projected third volume of his Theory of the Earth of 1795. The article brings some of the assumptions and techniques of literary criticism to bear on Hutton’s scientific travel writing in order to open up new connections between geology, Romantic aesthetics and eighteenth-century travel writing about Scotland. Close analysis of Hutton’s accounts of his field trips to Glen Tilt, Galloway and Arran, supplemented by later accounts of the discoveries at Jedburgh and Siccar Point, reveals the interplay between desire, travel and the scientific quest and foregrounds the textual strategies that Hutton uses to persuade his readers that they share in the experience of geological discovery and interpretation as ‘virtual witnesses’. As well as allowing us to revisit the interrelation between scientific theory and discovery, this article concludes that Hutton was a much better writer than he has been given credit for and suggests that if these geological tours had been published in 1795 they would have made it impossible for critics to dismiss him as an armchair geologist

    Completeness of classical spin models and universal quantum computation

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    We study mappings between distinct classical spin systems that leave the partition function invariant. As recently shown in [Phys. Rev. Lett. 100, 110501 (2008)], the partition function of the 2D square lattice Ising model in the presence of an inhomogeneous magnetic field, can specialize to the partition function of any Ising system on an arbitrary graph. In this sense the 2D Ising model is said to be "complete". However, in order to obtain the above result, the coupling strengths on the 2D lattice must assume complex values, and thus do not allow for a physical interpretation. Here we show how a complete model with real -and, hence, "physical"- couplings can be obtained if the 3D Ising model is considered. We furthermore show how to map general q-state systems with possibly many-body interactions to the 2D Ising model with complex parameters, and give completeness results for these models with real parameters. We also demonstrate that the computational overhead in these constructions is in all relevant cases polynomial. These results are proved by invoking a recently found cross-connection between statistical mechanics and quantum information theory, where partition functions are expressed as quantum mechanical amplitudes. Within this framework, there exists a natural correspondence between many-body quantum states that allow universal quantum computation via local measurements only, and complete classical spin systems.Comment: 43 pages, 28 figure

    Genetic variation at MECOM, TERT, JAK2 and HBS1L-MYB predisposes to myeloproliferative neoplasms

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    Clonal proliferation in myeloproliferative neoplasms (MPN) is driven by somatic mutations in JAK2, CALR or MPL, but the contribution of inherited factors is poorly characterized. Using a three-stage genome-wide association study of 3,437 MPN cases and 10,083 controls, we identify two SNPs with genome-wide significance in JAK2V617F-negative MPN: rs12339666 (JAK2; meta-analysis P=1.27 × 10−10) and rs2201862 (MECOM; meta-analysis P=1.96 × 10−9). Two additional SNPs, rs2736100 (TERT) and rs9376092 (HBS1L/MYB), achieve genome-wide significance when including JAK2V617F-positive cases. rs9376092 has a stronger effect in JAK2V617F-negative cases with CALR and/or MPL mutations (Breslow–Day P=4.5 × 10−7), whereas in JAK2V617F-positive cases rs9376092 associates with essential thrombocythemia (ET) rather than polycythemia vera (allelic χ2 P=7.3 × 10−7). Reduced MYB expression, previously linked to development of an ET-like disease in model systems, associates with rs9376092 in normal myeloid cells. These findings demonstrate that multiple germline variants predispose to MPN and link constitutional differences in MYB expression to disease phenotype
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