Benchmarking zero-shot and few-shot approaches for tokenization, tagging, and dependency parsing of Tagalog text

The grammatical analysis of texts in any human language typically involves a number of basic processing tasks, such as tokenization, morphological tagging, and dependency parsing. State-of-the-art systems can achieve high accuracy on these tasks for languages with large datasets, but yield poor results for languages such as Tagalog which have little to no annotated data. To address this issue for the Tagalog language, we investigate the use of auxiliary data sources for creating task-specific models in the absence of annotated Tagalog data. We also explore the use of word embeddings and data augmentation to improve performance when only a small amount of annotated Tagalog data is available. We show that these zero-shot and few-shot approaches yield substantial improvements on grammatical analysis of both in-domain and out-of-domain Tagalog text compared to state-of-the-art supervised baselines.Comment: To appear at PACLIC 2022. 10 pages, 2 figures, 4 table

Relatório de estágio em farmácia comunitária

Relatório de estágio realizado no âmbito do Mestrado Integrado em Ciências Farmacêuticas, apresentado à Faculdade de Farmácia da Universidade de Coimbr

Soil erosion vulnerability in the southern part of the Meia Ponte watershed, Goias, Brazil

ABSTRACT Soil erosion is a serious environmental problem that can cause numerous types of damage to society and the environment, and thus require preventive measures. The objective of this study was to evaluate soil erosion vulnerability in the southern region of the Meia Ponte watershed, Goias, Brazil, considering two situations: natural and anthropic cover conditions. For the analysis, we used remote sensing data represented by several parameters: lithology, soil class, slope, rain intensity, vegetation index, vicinity of roads, and land use and occupation. For each variable, we established a scale of weights according to erosive susceptibility for natural and anthropogenic environments. Multicriteria analysis, which allows the combination of qualitative and quantitative information in the analysis of erosive susceptibility, was used through the relationship between soil use and land occupation. In the southern part of the Meia Ponte River watershed, the anthropic factor showed greater influence. The factors that increase erosive susceptibility were soil use and occupation, low vegetation index, and high slope. The southern part of the Meia Ponte watershed presents medium natural erosive susceptibility in most of the study area.RESUMEN La erosión de los suelos es un grave problema ambiental que puede ocasionar numerosos daños a la sociedad y al medio ambiente, siendo necesario la adopción de medidas preventivas. Este estudio tuvo por objetivo evaluar la vulnerabilidad erosiva de los suelos en la región sur de la cuenca Meia Ponte, Goiás, considerando dos situaciones: condiciones de cobertura natural y antrópica. Para la realización de los análisis, se utilizaron datos de sensoriamiento remoto representados por diversos parámetros: litología, clase de suelo, declividad, intensidad de lluvia, índice de vegetación, cercanías de vías y uso y ocupación del suelo. Se establecieron para cada variable, una escala de pesos de acuerdo con susceptibilidad erosiva para el medio natural y otra antrópica. Se utilizó el análisis multicriterio, por medio de la relación entre las actividades de uso y ocupación del suelo, la cual permite combinar informaciones cualitativas y cuantitativas en análisis en cuanto a la susceptibilidad erosiva. En la parte sur de la cuenca del Río Meia Ponte, el factor antrópico obtuvo mayor influencia. Los factores que aumentaron susceptibilidad erosiva fueron el uso y ocupación del suelo, bajo índice de vegetación y la alta declividad. La parte sur de la cuenca Media Ponte, presenta susceptibilidad erosiva natural media en la mayor parte del área de estudio

Expression and clinical implication of cyclooxygenase-2 and e-cadherin in oral squamous cell carcinomas

Epithelial-Mesenchymal Transition (EMT) and angiogenesis are crucial events for development of aggressive and often fatal Oral Squamous Cell Carcinomas (OSCCs). Both promote cancer progression and metastasis development, but while the former induces the loss of E-cadherin expression and, hence cadherin switching; the latter produces haematic blood vessel neo-formation and contribute to OSCC cell growth, tumor mass development, and dissemination. Cyclooxygenase-2 (COX-2) has an important role, not only in angiogenic mechanisms, but also in favoring cancer invasion. Indeed it decreases the expression of E-cadherin and leads to phenotypic changes in epithelial cells (EMT) enhancing their carcinogenic potential. Our aim is to evaluate the interplay between E-cadherin cytoplasmic delocalization, COX-2 up-regulation and COX-2 induced neo-angiogenesis in 120 cases of OSCC. We have analyzed the distribution and the number of neo-formed endothelial buds surrounding infiltrating cells that express COX-2, as well as the neo-formed vessels in chronic inflammatory infiltrate, which surround the tumor. A double immunostaining method was employed in order to verify co-localization of endothelial cell marker (CD34) and COX-2. IHC has also been used to assess E-cadherin expression. Our data demonstrate that the OSCC cells, which lose membranous E-cadherin staining, acquiring a cytoplasmic delocalization, overexpress COX-2. Moreover, we find a new CD34+ vessel formation (sprouting angiogenesis). Only basaloid type of OSCC showes low level of COX-2 expression together with very low level of neo-angiogenesis and consequent tumor necrosis. The well-known anti-metastatic effect of certain COX-2 inhibitors suggests that these molecules might have clinical utility in the management of advanced cancers

Expression and clinical implication of cyclooxygenase-2 and e-cadherin in oral squamous cell carcinomas

Epithelial-Mesenchymal Transition (EMT) and angiogenesis are crucial events for development of aggressive and often fatal Oral Squamous Cell Carcinomas (OSCCs). Both promote cancer progression and metastasis development, but while the former induces the loss of E-cadherin expression and, hence cadherin switching; the latter produces haematic blood vessel neo-formation and contribute to OSCC cell growth, tumor mass development, and dissemination. Cyclooxygenase-2 (COX-2) has an important role, not only in angiogenic mechanisms, but also in favoring cancer invasion. Indeed it decreases the expression of E-cadherin and leads to phenotypic changes in epithelial cells (EMT) enhancing their carcinogenic potential. Our aim is to evaluate the interplay between E-cadherin cytoplasmic delocalization, COX-2 up-regulation and COX-2 induced neo-angiogenesis in 120 cases of OSCC. We have analyzed the distribution and the number of neo-formed endothelial buds surrounding infiltrating cells that express COX-2, as well as the neo-formed vessels in chronic inflammatory infiltrate, which surround the tumor. A double immunostaining method was employed in order to verify co-localization of endothelial cell marker (CD34) and COX-2. IHC has also been used to assess E-cadherin expression. Our data demonstrate that the OSCC cells, which lose membranous E-cadherin staining, acquiring a cytoplasmic delocalization, overexpress COX-2. Moreover, we find a new CD34+ vessel formation (sprouting angiogenesis). Only basaloid type of OSCC showes low level of COX-2 expression together with very low level of neo-angiogenesis and consequent tumor necrosis. The well-known anti-metastatic effect of certain COX-2 inhibitors suggests that these molecules might have clinical utility in the management of advanced cancers

Expression and clinical implication of cyclooxygenase-2 and e-cadherin in oral squamous cell carcinomas

Epithelial-Mesenchymal Transition (EMT) and angiogenesis are crucial events for development of aggressive and often fatal Oral Squamous Cell Carcinomas (OSCCs). Both promote cancer progression and metastasis development, but while the former induces the loss of E-cadherin expression and, hence cadherin switching; the latter produces haematic blood vessel neo-formation and contribute to OSCC cell growth, tumor mass development, and dissemination. Cyclooxygenase-2 (COX-2) has an important role, not only in angiogenic mechanisms, but also in favoring cancer invasion. Indeed it decreases the expression of E-cadherin and leads to phenotypic changes in epithelial cells (EMT) enhancing their carcinogenic potential. Our aim is to evaluate the interplay between E-cadherin cytoplasmic delocalization, COX-2 up-regulation and COX-2 induced neo-angiogenesis in 120 cases of OSCC. We have analyzed the distribution and the number of neo-formed endothelial buds surrounding infiltrating cells that express COX-2, as well as the neo-formed vessels in chronic inflammatory infiltrate, which surround the tumor. A double immunostaining method was employed in order to verify co-localization of endothelial cell marker (CD34) and COX-2. IHC has also been used to assess E-cadherin expression. Our data demonstrate that the OSCC cells, which lose membranous E-cadherin staining, acquiring a cytoplasmic delocalization, overexpress COX-2. Moreover, we find a new CD34+ vessel formation (sprouting angiogenesis). Only basaloid type of OSCC showes low level of COX-2 expression together with very low level of neo-angiogenesis and consequent tumor necrosis. The well-known anti-metastatic effect of certain COX-2 inhibitors suggests that these molecules might have clinical utility in the management of advanced cancers