Biblical Humanism and Roman Catholic Reform: (1501-1542) Contarini, Pole, and Giberti

Reginald Pole deserves careful attention by students of the Reformation period. Pole delivered one and shared in a second of three confessions which assumed Roman Catholic guilt for the splintering of Christendom. Adrian VI excoriated the Curia in his Instructio of Jan. 3, 1523, read to the Diet of Nuremberg by Francesco Chieregati

Laurentius Valla (1407-1457): Renaissance Critic and Biblical Theologian

When Laurentius Valla penned those words, he was writing the fuse scientific treatise on Latin grammar since John Duns Scotus. Leonardo Bruni died in the same year Valla’s treatise appeared. The year 1444 marks the return of Renaissance scholars to a philological analysis of classical texts. This method, which Valla soon applied to Biblical study, revolutionized medieval Biblical scholarship in the century before Trent. Valla\u27s purpose was to revitalize Catholic faith. Protestants and Catholics still owe their fresh awareness of Scripture to the labors of Valla

Brief Studies

Professor Gordon Rupp: The Optimism of Grace Toward Lutheran Unit

Capacity of Second-Growth Douglas-fir and Western Hemlock Stump Anchors for Cable Logging

The use of small instead of large stumps for cable logging anchors will usually result in applied loads approaching the load capacity of the anchors more closely. The use of small stump anchors is then contingent on better means of assessing their capacity. The results of field load tests of Douglas-fir and western hemlock stump anchors are reported. Ultimate loads were modeled as power functions of DBH. In addition, the relation between load and movement relationships for the stumps are modeled using a hyperbolic function that also provides an estimate of ultimate load. Practical use of the model equations requires knowledge of failure statistics and the acceptance of a probabilistic anchor capacity. Probability is applied to the re-rigging required when an anchor fails to perform adequately and to total pull-out failure

CORE and the Haldane Conjecture

The Contractor Renormalization group formalism (CORE) is a real-space renormalization group method which is the Hamiltonian analogue of the Wilson exact renormalization group equations. In an earlier paper\cite{QGAF} I showed that the Contractor Renormalization group (CORE) method could be used to map a theory of free quarks, and quarks interacting with gluons, into a generalized frustrated Heisenberg antiferromagnet (HAF) and proposed using CORE methods to study these theories. Since generalizations of HAF's exhibit all sorts of subtle behavior which, from a continuum point of view, are related to topological properties of the theory, it is important to know that CORE can be used to extract this physics. In this paper I show that despite the folklore which asserts that all real-space renormalization group schemes are necessarily inaccurate, simple Contractor Renormalization group (CORE) computations can give highly accurate results even if one only keeps a small number of states per block and a few terms in the cluster expansion. In addition I argue that even very simple CORE computations give a much better qualitative understanding of the physics than naive renormalization group methods. In particular I show that the simplest CORE computation yields a first principles understanding of how the famous Haldane conjecture works for the case of the spin-1/2 and spin-1 HAF.Comment: 36 pages, 4 figures, 5 tables, latex; extensive additions to conten

CORE Technology and Exact Hamiltonian Real-Space Renormalization Group Transformations

The COntractor REnormalization group (CORE) method, a new approach to solving Hamiltonian lattice systems, is presented. The method defines a systematic and nonperturbative means of implementing Kadanoff-Wilson real-space renormalization group transformations using cluster expansion and contraction techniques. We illustrate the approach and demonstrate its effectiveness using scalar field theory, the Heisenberg antiferromagnetic chain, and the anisotropic Ising chain. Future applications to the Hubbard and t-J models and lattice gauge theory are discussed.Comment: 65 pages, 9 Postscript figures, uses epsf.st

Solar System Processes Underlying Planetary Formation, Geodynamics, and the Georeactor

Only three processes, operant during the formation of the Solar System, are responsible for the diversity of matter in the Solar System and are directly responsible for planetary internal-structures, including planetocentric nuclear fission reactors, and for dynamical processes, including and especially, geodynamics. These processes are: (i) Low-pressure, low-temperature condensation from solar matter in the remote reaches of the Solar System or in the interstellar medium; (ii) High-pressure, high-temperature condensation from solar matter associated with planetary-formation by raining out from the interiors of giant-gaseous protoplanets, and; (iii) Stripping of the primordial volatile components from the inner portion of the Solar System by super-intense solar wind associated with T-Tauri phase mass-ejections, presumably during the thermonuclear ignition of the Sun. As described herein, these processes lead logically, in a causally related manner, to a coherent vision of planetary formation with profound implications including, but not limited to, (a) Earth formation as a giant gaseous Jupiter-like planet with vast amounts of stored energy of protoplanetary compression in its rock-plus-alloy kernel; (b) Removal of approximately 300 Earth-masses of primordial gases from the Earth, which began Earth's decompression process, making available the stored energy of protoplanetary compression for driving geodynamic processes, which I have described by the new whole-Earth decompression dynamics and which is responsible for emplacing heat at the mantle-crust-interface at the base of the crust through the process I have described, called mantle decompression thermal-tsunami; and, (c)Uranium accumulations at the planetary centers capable of self-sustained nuclear fission chain reactions.Comment: Invited paper for the Special Issue of Earth, Moon and Planets entitled Neutrino Geophysics Added final corrections for publicatio

Precision Determination of the Neutron Spin Structure Function g1n

We report on a precision measurement of the neutron spin structure function $g^n_1$ using deep inelastic scattering of polarized electrons by polarized ^3He. For the kinematic range 0.014<x<0.7 and 1 (GeV/c)^2< Q^2< 17 (GeV/c)^2, we obtain $\int^{0.7}_{0.014} g^n_1(x)dx = -0.036 \pm 0.004 (stat) \pm 0.005 (syst)$ at an average $Q^2=5 (GeV/c)^2$. We find relatively large negative values for $g^n_1$ at low $x$. The results call into question the usual Regge theory method for extrapolating to x=0 to find the full neutron integral $\int^1_0 g^n_1(x)dx$, needed for testing quark-parton model and QCD sum rules.Comment: 5 pages, 3 figures To be published in Phys. Rev. Let

Research priorities in hypertrophic cardiomyopathy: report of a Working Group of the National Heart, Lung, and Blood Institute.

Hypertrophic cardiomyopathy (HCM) is a myocardial disorder characterized by left ventricular (LV) hypertrophy without dilatation and without apparent cause (ie, it occurs in the absence of severe hypertension, aortic stenosis, or other cardiac or systemic diseases that might cause LV hypertrophy). Numerous excellent reviews and consensus documents provide a wealth of additional background.1–8 HCM is the leading cause of sudden death in young people and leads to significant disability in survivors. It is caused by mutations in genes that encode components of the sarcomere. Cardiomyocyte and cardiac hypertrophy, myocyte disarray, interstitial and replacement fibrosis, and dysplastic intramyocardial arterioles characterize the pathology of HCM. Clinical manifestations include impaired diastolic function, heart failure, tachyarrhythmia (both atrial and ventricular), and sudden death. At present, there is a lack of understanding of how the mutations in genes encoding sarcomere proteins lead to the phenotypes described above. Current therapeutic approaches have focused on the prevention of sudden death, with implantable cardioverter defibrillator placement in high-risk patients. But medical therapies have largely focused on alleviating symptoms of the disease, not on altering its natural history. The present Working Group of the National Heart, Lung, and Blood Institute brought together clinical, translational, and basic scientists with the overarching goal of identifying novel strategies to prevent the phenotypic expression of disease. Herein, we identify research initiatives that we hope will lead to novel therapeutic approaches for patients with HCM