41 research outputs found

    Vaginismus After Epilepsy Surgery

    No full text

    Performance Evaluation of Pansharpening Methods on GPU for RASAT Images

    No full text
    Turkey is among the countries which could develop earth observation satellites. RASAT and Gokturk-2 satellites are still operational and continuously acquire images of the Earth. Their images are processed before sharing with end users. Pansharpening, at which high resolution pan and low resolution multi-spectral images are fused, is an important step in image processing chain. As the resolution and number of images increase, pansharpening of satellite images take considerable amount of time. Multithread programming and General Purpose GPU (GPGPU) programming implementation improve performance of image processing applications, where most operations carried out on individual pixels. In this paper, we compared pansharpening applications and their CPU and GPU implementations for RASAT images. GPU implementations of pansharpening algorithms provides 20-25 times speed-up compared to CPU implementations

    Anti-Mullerian hormone levels as a predictor of the pregnancy rate in women of advanced reproductive age

    No full text
    To investigate whether serum anti-mullerian hormone (AMH), follicle stimulating hormone (FSH), or antral follicle count (AFC) are predictive for clinical pregnancy in women who underwent IVF cycles at the age of 35 and olde

    Transient Gyral Enhancement After Epileptic Seizure in a Patient with CREST Syndrome

    No full text
    WOS: 000419248500012Limited scleroderma, also termed CREST syndrome (calcinosis cutis, Raynaud's phenomenon, esophageal dysmotility, sclerodactyly and telangiectasia), is a form of scleroderma which is an autoimmune connective tissue disease. Neurological involvement is less common than other system involvements in scleroderma. Epileptic seizure is a rare neurological complication. Gyral enhancement is not an expected finding during disease. Gyral enhancement is contrast enhancement of superficial brain parenchyma and it usually occurs as a result of vascular, inflammatory, infectious and neoplastic processes. A-sixty-seven-year old female patient, who was diagnosed as CREST syndrome for three years was admitted to our hospital. She had an epileptic seizure three days ago and behavioral changes after the seizure. There was not epilepsy diagnosis in her medical history. In neurological examination; she was apathic and deep tendon reflexes were hyperactive, Hoffman's and Babinski reflexes were positive on the right side of her body. In laboratory studies; anti-nuclear antibody was positive at 1/1000 and anti-centromere antibody was positive. On contrast-enhanced cranial magnetic resonance imaging (MRI); gyral enhancement was observed in the left parieto-occipital region. Cerebrospinal fluid tests were within normal limits. Electroencephalography demonstrated diffuse cerebral dysfunction. We administrated antiepileptic drug to the patient during hospitalization. On the follow up, apathy and restriction of the cooperation were improved. Epileptic seizure did not recur. After ten days, in the control cranial MRI, gyral enhancement was reduced significantly. Neurological involvement in CREST syndrome is rare and epileptic seizures have been appeared in a limited number in the literature. Inflammatory and vascular processes can cause gyral enhancement, but also it must be kept in mind that gyral enhancement can be observed transiently after epileptic seizure

    Prostate specific antigen levels after acute myocardial infarction

    No full text
    WOS: 000298781000014PubMed: 22187678Prostate Specific Antigen (PSA), a member of kallikrein family, is a specific serine protease of prostatic tissue. In some case reports, changes in PSA levels after acute myocardial infarction (AMI) have been reported. In this study we evaluated variations in PSA levels post-AMI. Twenty-six male patients who had PSA levels within reference limits were included in the study. The diagnosis of AMI was confirmed by clinical findings, ECG (electrocardiogram) and cardiac marker studies. Serum total PSA (tPSA) and free PSA (fPSA) levels were measured at days 0 (day of admission), 1, 2 and 3 after AMI. PSA/albumin ratio was also calculated in order to evaluate the effect of dilution. A statistical analysis of the results of all patients revealed significant decrease in tPSA levels and tPSA/Albumin ratio at day 2 when compared to days 0 and 3, which showed a similar pattern. Changes of fPSA and fPSA/Albumin ratio according to days were not found significant. In only four patients we found increased levels of tPSA and increased fPSA levels in three of them. These patients displayed severe problems such as renal failure, cardiac failure, ventricular aneurysm and cerebral ischemia due to cardiac arrest. The lower tPSA levels on day 2 suggest that tPSA can be eliminated rapidly from the circulation on days 1 and 2, probably through the formation of complexes of tPSA with acute phase proteins

    Prostate specific antigen levels after acute myocardial infarction

    No full text
    Prostate Specific Antigen (PSA), a member of kallikrein family, is a specific serine protease of prostatic tissue. In some case reports, changes in PSA levels after acute myocardial infarction (AMI) have been reported. In this study we evaluated variations in PSA levels post-AMI. Twenty-six male patients who had PSA levels within reference limits were included in the study. The diagnosis of AMI was confirmed by clinical findings, ECG (electrocardiogram) and cardiac marker studies. Serum total PSA (tPSA) and free PSA (fPSA) levels were measured at days 0 (day of admission), 1, 2 and 3 after AMI. PSA/albumin ratio was also calculated in order to evaluate the effect of dilution. A statistical analysis of the results of all patients revealed significant decrease in tPSA levels and tPSA/Albumin ratio at day 2 when compared to days 0 and 3, which showed a similar pattern. Changes of fPSA and fPSA/Albumin ratio according to days were not found significant. In only four patients we found increased levels of tPSA and increased fPSA levels in three of them. These patients displayed severe problems such as renal failure, cardiac failure, ventricular aneurysm and cerebral ischemia due to cardiac arrest. The lower tPSA levels on day 2 suggest that tPSA can be eliminated rapidly from the circulation on days 1 and 2, probably through the formation of complexes of tPSA with acute phase proteins

    Maternal serum and fetal cord blood irisin levels in gestational diabetes mellitus

    No full text
    Aim: To investigate the relationship between maternal and cord blood irisin in gestational diabetes mellitus (GDM)

    The relationship between maternal and umbilical cord adropin levels with the presence and severity of preeclampsia

    No full text
    Aim: To investigate both maternal and umbilical cord adropin levels in patients with preeclampsia and the possible relations with its severity and perinatal outcomes. Materials and methods: In this study, a total of 38 preeclamptic and 40 age- matched healthy pregnant women between January and June 2016 were included. Serum and cord adropin levels were measured using an enzymelinked immunosorbent assay (ELISA). Results: The maternal and umbilical cord adropin levels were significantly lower in the preeclamptic group compared to controls [71.19 +/- 22.21 vs. 100.76 +/- 27.02 ng/L and 92.39 (59.77:129.89) vs. 106.20 (74.42: 208.02) ng/L, P < 0.001, respectively]. While maternal adropin levels were significantly lower in the severe preeclampsia group as compared to the mild preeclamptic group [66.45 (21.49:98.02) vs. 76.17 (58.06:109.58), P = 0.007], umbilical cord adropin levels did not differ between each group [91.32 (59.77:113.34) vs. 92.87 (63.12:129.89), P = 0.750]. Maternal adropin level was negatively correlated with systolic and diastolic blood pressures (r = - 0.60, P < 0.001 and r = - 0.58, P < 0.001, respectively) and positively correlated with platelet count (r = 0.27, P = 0.016). Moreover, umbilical cord adropin levels were weakly correlated with gestational age at delivery (r = 0.28, P = 0.012) and birth weight (r = 0.28, P = 0.014). Conclusion: The present study is the first to demonstrate a significant association between maternal and umbilical adropin levels and the presence and severity of preeclampsia. Adropin might be a useful parameter for predicting the presence and severity of preeclampsia

    The relationship between maternal and umbilical cord adropin levels with the presence and severity of preeclampsia

    No full text
    Aim: To investigate both maternal and umbilical cord adropin levels in patients with preeclampsia and the possible relations with its severity and perinatal outcomes
    corecore