4,268 research outputs found
Microgastrinae (Hymenoptera: Braconidae) parasitizing Epirrita autumnata (Lepidoptera: Geometridae) larvae in Fennoscandia with description of Cotesia autumnatae Shaw, sp. n.
The microgastrine subset of hymenopteran parasitoids of the geometrid Epirrita
autumnata is investigated in Fennoscandia. Ecology, including population dynamics,
of the moth has been intensively studied in northern and mountainous
Finland, Norway and Sweden. Recently supported hypotheses about the causes
of its cyclic population dynamics stress the role of parasitoids, while the
parasitoid complex with some 15 species is insufficiently known. The complex
includes four solitarymicrogastrine species, Protapanteles anchisiades (Nixon),
P. immunis (Wesmael), Cotesia salebrosa (Marshall) and C. autumnatae Shaw,
sp. n. Here, we provide detailed figures for the latter, which is morphologically
close to C. jucunda (Marshall), and describe the species as new to science. We
also providemore general habitus figures of the other three species, as well as an
identification key for the four species, aiming to aid recognition of these species
by ecologists dealingwithmicrogastrine parasitoids of E. autumnata and their alternative
geometrid hosts
Probability distributions for locations of calling animals, receivers, sound speeds, winds, and data from travel time differences
The Gabor wave front set of compactly supported distributions
We show that the Gabor wave front set of a compactly supported distribution
equals zero times the projection on the second variable of the classical wave
front set
Conormal distributions in the Shubin calculus of pseudodifferential operators
We characterize the Schwartz kernels of pseudodifferential operators of
Shubin type by means of an FBI transform. Based on this we introduce as a
generalization a new class of tempered distributions called Shubin conormal
distributions. We study their transformation behavior, normal forms and
microlocal properties.Comment: 23 page
Tubuloglomerular feedback and interstitial pressure in obstructive nephropathy
Tubuloglomerular feedback and interstitial pressure in obstructive nephropathy. The possible role of the tubuloglomerular feedback (TGF) mechanism in the altered glomerular hemodynamics and tubular reabsorption which occur with prolonged (24-hr) ureteral obstruction and the changes in renal interstitial hydrostatic and oncotic pressure which may modulate TGF sensitivity were examined. The proximal tubule stop-flow pressure (PSF) response to increased distal tubular flow rates (TGF activity) was determined in rats with sham operation, 24-hr unilateral ureteral obstruction (UUO), or 24-hr bilateral ureteral obstruction (BUO), both before and for 2 hr after relief of obstruction. Subcapsular hydrostatic pressure, lymph flow and oncotic pressure, clearance and excretory data were measured in the second series of animals. During and after release of UUO, TGF sensitivity was increased, as indicated by the marked decrease in the loop perfusion rate at which 50% of the maximum decrease in PSF occurred (the turning point of TGF activation). Interstitial oncotic pressure but not hydrostatic pressure was significantly increased in UUO kidneys. In BUO rats, the turning point for TGF activation was slightly higher than the controls and the change in PSF with maximum loop perfusion rates was reduced, indicating a blunting of the TGF response before and particularly during postobstructive diuresis after release of BUO. Interstitial hydrostatic and oncotic pressures were both slightly increased resulting in no changes in net interstitial Starling forces. We conclude that enhanced TGF sensitivity after release of prolonged UUO, associated with increased interstitial oncotic pressure, may play a role in preventing postobstructive diuresis, while the blunting of TGF sensitivity after BUO may contribute to this phenomenon.Rétrocontrôle glomérulo-tubulaire et pression interstitielle au cours de la néphropathie obstructive. Le rôle possible du mécanisme de rétrocontrôle glomérulo-tubulaire (TGF) dans l'altération de l'hémadynamique glomérulaire et la réabsorption tubulaire qui se produisent lors d'une obstruction urétérale prolongée (24 heures) et les modifications des pressions hydrostatiques et oncotiques interstitielles rénales qui pourraient moduler la sensibilité TGF ont été étudiés. La réponse de pression en flux interrompu (PSF) du tubule proximal à une augmentation des débits tubulaires distaux a été déterminée chez des rats ayant subi un simulacre d'intervention, lors d'une obstruction urétérale unilatérale de 24 heures (UUO) ou lors d'une obstruction urétérale bilatérale de 24 heures (BUO), avant et 2 heures après la levée de l'obstruction. La pression hydrostatique sous-capsulaire, le flux et la pression oncotique lymphatiques, les paramètres de clearance et d'excrétion ont été mesurés chez une deuxième série d'animaux. Pendant et après levée de l'UUO, la sensibilité TGF a augmenté, comme le montrait la diminution marquée du débit de perfusion de l'anse pour lequel 50% de la chute maximale de PSF se produisait (le point d'inflexion de l'activation TGF). La pression oncotique mais non la pression hydrostatique interstitielle était significativement accrue dans les reins UUO. Chez les rats BUO, le point d'inflexion de l'activation TGF était légèrement plus élevé que chez les contrôles, et la modification de PSF aux débits de perfusion de l'anse maxima était diminuée, indiquant une altération de la réponse TGF avant et surtout pendant la diurèse post-obstructive, après levée de la BUO. Les pressions interstitielles hydrostatiques et oncotiques légèrement augmentées, d'où l'absence de modification des forces de Starling interstitielles nettes. Nous concluons que l'augmentation de la sensibilité TGF après levée d'une UUO prolongée associée à une élévation de la pression oncotique interstitielle, pourrait jouer un rôle pour prévenir la diurèse post-obstructive, alors que l'altération de la sensibilité TGF après BUO pourrait contribuer à ce phénomène
Observers Data Only Fault Detection
Abstract: Most fault detection algorithms are based on residuals, i.e.the difference between a measured signal and the corresponding model based prediction. However, in many more advanced sensors the raw measurements are internally processed before refined information is provided to the user. The contribution of this paper is to study the problem of fault detection when only the state estimate from an observer/Kalman filter is available and not the direct measured quantities. The idea is to look at an extended state space model where the true states and the observer states are combined. This extended model is then used to generate residuals viewing the observer outputs as measurements. Results for fault observability of such extended models are given. The approach is rather straightforward in case the internal structure of the observer is exactly known. For the Kalman filter this corresponds to knowing the observer gain. If this is not the case certain model approximations can be done to generate a simplified model to be used for standard fault detection. The corresponding methods are evaluated on a DC motor example. The next step is a real data robotics demonstrator
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