Role Strain of Nursing Students in Obstetrical Clinical Rotations: Is Role Strain Greater for Male Students?

As increasing numbers of men enter the career of nursing, it is important that nursing educators identify areas of role strain and learning needs that may be different from their female counterparts This research project examined the role strain factors of nursing students who have completed the maternaVnewbom (obstetrical) clinical experience in a baccalaureate nursing program. The research question addressed in this research project was: Do male nursing students experience greater role strain than female nursing students in the matemaVnewbom setting? A convenience sample of male students (N=23) and a random sampling of female students (N=23) participated in this study. The Junior and Senior level nursing students who had completed the obstetrical rotation within the last 3 semesters were recruited to complete the Sherrod Role Strain Scale ( 40 question Likert scale survey tool) at specified times during fall semester 2008. Data was analyzed as grouped data and female/male responses were also compared. The overall score showed no gender differences in role strain (p value= 0.917). The four subcategories (Overload, Conflict, Incongruity, and Ambiguity) also did not reflect gender differences in this sample. The findings of this study did not support the concept of male students experiencing greater role strain than their female counterparts. Further studies examining role strain factors, student perceptions of role strain, and comparing men students in various nursing programs are recommended

Environmental study of ERTS-1 imagery Lake Champlain and Vermont

There are no author-identified significant results in this report

The Morita equivalence between parametrized spectra and module spectra

We give a Quillen equivalence between May and Sigurdsson's model category of parametrized spectra over BG, and Mandell, May, Schwede, and Shipley's model category of modules over the orthogonal ring spectrum \Sigma^\infty_+ G, for each topological group G. More generally, for a topological category C we introduce an "aggregate" model structure on the category of diagrams of spectra indexed by C, and prove that it is Quillen equivalent to spectra over BC. This lifts several earlier results, and leads to a complete characterization of the dualizable parametrized spectra, answering a question of May and Sigurdsson.Comment: 24 pages, to appear in Contemporary Mathematic

Horizontal-branch morphology and multiple stellar populations in the anomalous globular cluster M22

M22 is an anomalous globular cluster that hosts two groups of stars with different metallicity and s-element abundance. The star-to-star light-element variations in both groups, with the presence of individual Na-O and C-N anticorrelations, demonstrates that this Milky-Way satellite has experienced a complex star-formation history. We have analysed FLAMES/UVES spectra for seven stars covering a small color interval, on the reddest horizontal-branch (HB) portion of this cluster and investigated possible relations between the chemical composition of a star and its location along the HB. Our chemical abundance analysis takes into account effects introduced by deviations from the local-thermodynamic equilibrium (NLTE effects), that are significant for the measured spectral lines in the atmospheric parameters range spanned by our stars. We find that all the analysed stars are barium-poor and sodium-poor, thus supporting the idea that the position of a star along the HB is strictly related to the chemical composition, and that the HB-morphology is influenced by the presence of different stellar populations.Comment: 21 pages, 3 figures, accepted for publication in Ap

A shared mechanism of muscle wasting in cancer and Huntington's disease.

Skeletal muscle loss and dysfunction in aging and chronic diseases is one of the major causes of mortality in patients, and is relevant for a wide variety of diseases such as neurodegeneration and cancer. Muscle loss is accompanied by changes in gene expression and metabolism that lead to contractile impairment and likely affect whole-body metabolism and function. The changes may be caused by inactivity, inflammation, age-related factors or unbalanced nutrition. Although links with skeletal muscle loss have been found in diseases with disparate aetiologies, for example both in Huntingtons disease (HD) and cancer cachexia, the outcome is a similar impairment and mortality. This short commentary aims to summarize recent achievements in the identification of common mechanisms leading to the skeletal muscle wasting syndrome seen in diseases as different as cancer and HD. The latter is the most common hereditary neurodegenerative disorder and muscle wasting is an important component of its pathology. In addition, possible therapeutic strategies for anti-cachectic treatment will be also discussed in the light of their translation into possible therapeutic approaches for HD