7,027 research outputs found
Renormalized parameters and perturbation theory for an n-channel Anderson model with Hund's rule coupling: Asymmetric case
We explore the predictions of the renormalized perturbation theory for an
n-channel Anderson model, both with and without Hund's rule coupling, in the
regime away from particle-hole symmetry. For the model with n=2 we deduce the
renormalized parameters from numerical renormalization group calculations, and
plot them as a function of the occupation at the impurity site, nd. From these
we deduce the spin, orbital and charge susceptibilities, Wilson ratios and
quasiparticle density of states at T=0, in the different parameter regimes,
which gives a comprehensive overview of the low energy behavior of the model.
We compare the difference in Kondo behaviors at the points where nd=1 and nd=2.
One unexpected feature of the results is the suppression of the charge
susceptibility in the strong correlation regime over the occupation number
range 1 <nd <3.Comment: 9 pages, 17 figure
Duramycin-induced calcium release in cancer cells
Introduction: Duramycin through binding with phosphatidylethanolamine (PE) has shown potential to be an effective anti-tumour agent. However its mode of action in relation to tumour cells is not fully understood. Methods: PE expression on the surface of a panel of cancer cell lines was analysed using duramycin and subsequent antibody labelling then analysed by flow cytometry. Cell viability was also assessed via flow cytometry using annexin V and propidium iodide (PI). Calcium ion (Ca²⁺) release by tumour cells in response to duramycin was determined by spectrofluorometry following incubation with Fluo-3, AM. Confocal microscopy was performed on the cancer cell line AsPC-1 to assess real time cell response to duramycin treatment. Results: Duramycin was able to detect cell surface PE expression on all 15 cancer cell lines screened, which was shown to be duramycin concentration dependent. However higher concentrations induced necrotic cell death. Duramycin induced calcium ion (Ca²⁺) release from the cancer cell lines also in a concentration and time dependent manner. Confocal microscopy showed an influx of PI into the cells over time and induced morphological changes. Conclusion: Duramycin induces Ca²⁺ release from cancer cell lines in a time and concentration dependent relationship
Special studies of AROD system concepts and designs
Signal processing techniques for range and range rate measurements in airborne range and orbit determinatio
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