13,183 research outputs found

    Eliashberg's proof of Cerf's theorem

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    Following a line of reasoning suggested by Eliashberg, we prove Cerf's theorem that any diffeomorphism of the 3-sphere extends over the 4-ball. To this end we develop a moduli-theoretic version of Eliashberg's filling-with-holomorphic-discs method.Comment: 32 page

    Translationally invariant treatment of pair correlations in nuclei: II. Tensor correlations

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    We study the extension of our translationally invariant treatment of few-body nuclear systems to include tensor forces and correlations. It is shown that a direct application of our method is not as successful for realistic V6 interactions as our previous results for V4 potentials suggested. We investigate the cause in detail for the case of 4^4He, and show that a combination of our method with that of Jastrow-correlated wave functions seems to be a lot more powerful, thereby suggesting that for mildly to strongly repulsive forces such a hybrid procedure may be an appropriate description.Comment: 19 pages, 3 ps figures. uses elsart, graphicx, amssym

    Spartan Daily, February 7, 1938

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    Volume 26, Issue 79https://scholarworks.sjsu.edu/spartandaily/2716/thumbnail.jp

    Spartan Daily, March 11, 1935

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    Volume 23, Issue 101https://scholarworks.sjsu.edu/spartandaily/2279/thumbnail.jp

    Spartan Daily, April 12, 1938

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    Volume 26, Issue 114https://scholarworks.sjsu.edu/spartandaily/2751/thumbnail.jp

    v. 43, no. 11, December 9, 1977

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    Feline hypersomatotropism and acromegaly tumorigenesis: a potential role for the AIP gene

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    Acromegaly in humans is usually sporadic, however up to 20% of familial isolated pituitary adenomas are caused by germline sequence variants of the aryl-hydrocarbon-receptor interacting protein (AIP) gene. Feline acromegaly has similarities to human acromegalic families with AIP mutations. The aim of this study was to sequence the feline AIP gene, identify sequence variants and compare the AIP gene sequence between feline acromegalic and control cats, and in acromegalic siblings. The feline AIP gene was amplified through PCR using whole blood genomic DNA from 10 acromegalic and 10 control cats, and 3 sibling pairs affected by acromegaly. PCR products were sequenced and compared with the published predicted feline AIP gene. A single nonsynonymous SNP was identified in exon 1 (AIP:c.9T > G) of two acromegalic cats and none of the control cats, as well as both members of one sibling pair. The region of this SNP is considered essential for the interaction of the AIP protein with its receptor. This sequence variant has not previously been reported in humans. Two additional synonymous sequence variants were identified (AIP:c.481C > T and AIP:c.826C > T). This is the first molecular study to investigate a potential genetic cause of feline acromegaly and identified a nonsynonymous AIP single nucleotide polymorphism in 20% of the acromegalic cat population evaluated, as well as in one of the sibling pairs evaluated
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