The dust masses of powerful radio galaxies: clues to the triggering of their activity

We use deep Herschel Space Observatory observations of a 90% complete sample of 32 intermediate-redshift 2Jy radio galaxies (0.05 < z < 0.7) to estimate the dust masses of their host galaxies and thereby investigate the triggering mechanisms for their quasar-like AGN. The dust masses derived for the radio galaxies (7.2x10^5 < M_d < 2.6x10^8 M_sun) are intermediate between those of quiescent elliptical galaxies on the one hand, and ultra luminous infrared galaxies (ULIRGs) on the other. Consistent with simple models for the co-evolution of supermassive black holes and their host galaxies, these results suggest that most of the radio galaxies represent the late time re-triggering of AGN activity via mergers between the host giant elliptical galaxies and companion galaxies with relatively low gas masses. However, a minority of the radio galaxies in our sample (~20%) have high, ULIRG-like dust masses, along with evidence for prodigious star formation activity. The latter objects are more likely to have been triggered in major, gas-rich mergers that represent a rapid growth phase for both their host galaxies and their supermassive black holes.Comment: 5 pages, 2 figures, accepted for publication in MNRAS Letter

A proposal to implement a quantum delayed choice experiment assisted by a cavity QED

We propose a scheme feasible with current technology to implement a quantum delayed-choice experiment in the realm of cavity QED. Our scheme uses two-level atoms interacting on and off resonantly with a single mode of a high Q cavity. At the end of the protocol, the state of the cavity returns to its ground state, allowing new sequential operations. The particle and wave behavior, which are verified in a single experimental setup, are postselected after the atomic states are selectively detected.Comment: 3 pages, 3 figures. arXiv admin note: text overlap with arXiv:1208.0802 by other author

Chronic alcohol consumption and withdrawal do not induce cell death in the suprachiasmatic nucleus, but lead to irreversible depression of peptide immunoreactivity and mRNA levels

There is evidence that chronic ethanol treatment (CET) disrupts the biological rhythms of various brain functions and behaviors. Because the suprachiasmatic nucleus (SCN) is widely recognized as the dominant pacemaker of the circadian system, we have examined the effects of CET and withdrawal on the main morphological features and chemoarchitecture of this hypothalamic nucleus. Groups of rats ethanol-treated for 6 and 12 months were compared with withdrawn rats (ethanol-treated for 6 months and then switched to a normal diet for an additional 6 months) and with groups of age-matched control and pair-fed control rats. The volume and the total number of neurons of the SCN were estimated from conventionally stained material, whereas the total number of astrocytes and of neurons containing vasopressin (AVP), vasoactive intestinal polypeptide (VIP), gastrin-releasing peptide (GRP), and somatostatin (SS) were estimated from immunostained sections. The estimates were obtained using unbiased stereological methods, based on Cavalieri’s principle and the optical fractionator. The volume of the SCN and the total number of SCN neurons and astrocytes did not vary among groups. We found, however, that CET induced a significant reduction in the total number of AVP-, VIP-, GRP-, and SS-containing neurons. Withdrawal from alcohol did not reduce but rather augmented the loss of VIP- and GRP-immunoreactive neurons. The CET-induced neurochemical alterations seem to result from a decrease in neuropeptide synthesis, as revealed by the reduction in AVP and VIP mRNA levels demonstrated byin situhybridization with radioactively labeled 48-mer AVP and 30-mer VIP probes. It is thus possible to conclude that the irreversible CET-induced changes in the neurochemistry of the SCN might underpin the disturbances in circadian rhythms observed after long-term alcohol consumption.</jats:p