348 research outputs found

    Positive-partial-transpose distinguishability for lattice-type maximally entangled states

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    We study the distinguishability of a particular type of maximally entangled states -- the "lattice states" using a new approach of semidefinite program. With this, we successfully construct all sets of four ququad-ququad orthogonal maximally entangled states that are locally indistinguishable and find some curious sets of six states having interesting property of distinguishability. Also, some of the problems arose from \cite{CosentinoR14} about the PPT-distinguishability of "lattice" maximally entangled states can be answered.Comment: It's rewritten. We deleted the original section II about PPT-distinguishability of three ququad-ququad MESs. Moreover, we have joined new section V which discuss PPT-distinguishability of lattice MESs for cases t=3t=3 and t=4t=4 . As a result, the sequence of the theorems in our article has been changed. And we revised the title of our articl

    Correlated Quantum Memory: Manipulating Atomic Entanglement via Electromagnetically Induced Transparency

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    We propose a feasible scheme of quantum state storage and manipulation via electromagnetically induced transparency (EIT) in flexibly unitedunited multi-ensembles of three-level atoms. For different atomic array configurations, one can properly steer the signal and the control lights to generate different forms of atomic entanglement within the framework of linear optics. These results shed new light on designing the versatile quantum memory devices by using, e.g., an atomic grid.Comment: 5 pages, 1 figur

    Twist-teleportation based local discrimination of maximally entangled states

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    In this work, we study the local distinguishability of maximally entangled states (MESs). In particular, we are concerned with whether any fixed number of MESs can be locally distinguishable for sufficiently large dimensions. Fan and Tian \emph{et al.} have already obtained two satisfactory results for the generalized Bell states (GBSs) and the qudit lattice states when applied to prime or prime power dimensions. We construct a general twist-teleportation scheme for any orthonormal basis with MESs that is inspired by the method used in [Phys. Rev. A \textbf{70}, 022304 (2004)]. Using this teleportation scheme, we obtain a sufficient and necessary condition for one-way distinguishable sets of MESs, which include the GBSs and the qudit lattice states as special cases. Moreover, we present a generalized version of the results in [Phys. Rev. A \textbf{92}, 042320 (2015)] for the arbitrary dimensional case.Comment: 7 pages, 2 figure

    Plant toxin β-ODAP activates integrin β1 and focal adhesion : a critical pathway to cause neurolathyrism

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    Neurolathyrism is a unique neurodegeneration disease caused by beta-N-oxalyl-L-alpha, beta-diaminopropionic (beta-ODAP) present in grass pea seed (Lathyrus stativus L.) and its pathogenetic mechanism is unclear. This issue has become a critical restriction to take full advantage of drought-tolerant grass pea as an elite germplasm resource under climate change. We found that, in a human glioma cell line, beta-ODAP treatment decreased mitochondrial membrane potential, leading to outside release and overfall of Ca2+ from mitochondria to cellular matrix. Increased Ca2+ in cellular matrix activated the pathway of ECM, and brought about the overexpression of beta 1 integrin on cytomembrane surface and the phosphorylation of focal adhesion kinase (FAK). The formation of high concentration of FA units on the cell microfilaments further induced overexpression of paxillin, and then inhibited cytoskeleton polymerization. This phenomenon turned to cause serious cell microfilaments distortion and ultimately cytoskeleton collapse. We also conducted qRT-PCR verification on RNA-sequence data using 8 randomly chosen genes of pathway enrichment, and confirmed that the data was statistically reliable. For the first time, we proposed a relatively complete signal pathway to neurolathyrism. This work would help open a new window to cure neurolathyrism, and fully utilize grass pea germplasm resource under climate change

    荆州区农村人口初发糖尿病胰岛功能的现状跟踪调查*

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    Objective: To study the change of islet function in patients with incipient diabetic characteristics through incipient diabetic tracking observation of the islet function in patients of Jingzhou area. Methods: Selection of 1220 cases of patients with diabetes mellitus in Jingzhou area as research object at the beginning, 12 months follow-up, the clinic after 3 months, 6 months and 12 months, all patients to detect blood sugar change, c-peptide release quantity, calculate insulin secretion index (HOMA -β) and insulin resistance index (HOMA IR), summarizes the characteristic of islet function in patients with changes. Results: ① The patients restored to basic standard blood sugar in 3 months by drug treatment, and the patient's blood glucose levels not seen obvious fluctuation after 6 months and 12 months; ② During follow-up, patients with diabetes sustained c-peptide release quantity reduction, and in three months after treatment, c-peptide release decreased obviously, and see a doctor at 6 months and 12 months after the comparison, the difference was statistically significant (P < 0.05). ③ During follow-up, insulin capacity was decreasing among patients with diabetes, within three months after the doctor had the greatest reduction, the difference was statistically significant (P < 0.05); ④ During follow-up, island hormone decreasing index, insulin resistance index continued to rise among patients with diabetes, and 6 months and 12 months, the most significant variations in 3 months (P < 0.05). Conclusion: With the extension of course, the pancreatic islet function in patients with early onset diabetes decreased gradually. It could be proved that there is a significant correlation between the two and especially seen in obvious function decline of pancreatic islets among the patients within 3 months.目的  通过对荆州区初发糖尿病患者的胰岛功能进行跟踪观察,探讨发现初发糖尿病患者胰岛功能的变化特点。方法  选取荆州区1220例初发糖尿病患者作为观察对象,跟踪随访12个月,在就诊后的3个月、6个月及12个月时,全部患者检测血糖变化、C-肽释放量,计算胰岛素分泌指数(HOMA-β)及胰岛素抵抗指数(HOMA-IR),观察总结患者的胰岛功能变化特点。结果  (1)通过药物治疗,患者血糖在3个月时基本达标,6个月及12个月时,患者的血糖水平未见明显波动;(2)随访期间,糖尿病患者C-肽释放量持续降低,且在就诊后3个月内,C-肽释放量下降明显,与就诊后6个月时及12个月时比较,差异有统计学意义(P<0.05)。(3)随访期间,糖尿病患者胰岛素放量持续降低,就诊后3个月内下降最明显,差异有统计学意义(P<0.05);(4)随访期间,糖尿病患者胰岛素分泌指数持续降低,胰岛素抵抗指数持续升高,且与6个月时和12个月时比较,3个月时变化幅度最为显著(P<0.05)。结论  随着病程的延长,初发糖尿病患者胰岛功能逐渐降低,二者具有显著相关性,且3个月内患者的胰岛功能下降最为显著
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