L'Écho : grand quotidien d'information du Centre Ouest

28 juillet 19231923/07/28 (A52).Appartient à l’ensemble documentaire : PoitouCh

MOESM5 of Identification and characterization of a new type of inhibitor against the human immunodeficiency virus type-1 nucleocapsid protein

Additional file 5: Figure S4. Inhibition of HIV-1 gRNA packaging in a high concentration of A1752. MT4 cells were infected with treatment of the inhibitors indicated. The viral genomic RNA was isolated from concentrated viral supernatant followed by northern blot analysis using Gag-specific probes

MOESM1 of Identification and characterization of a new type of inhibitor against the human immunodeficiency virus type-1 nucleocapsid protein

Additional file 1: Figure S1 . Effect of the A1752 on reverse transcriptase activity in vitro. The percentage inhibition of in vitro RT activity by the compounds indicated at different concentrations is shown. Nevirapine and Etravirine, both HIV-1 non-nucleoside reverse transcriptase inhibitors, were used as positive controls. Data are the mean ¹ SEM of three separate experiments

MOESM1 of Association between brachial-ankle pulse wave velocity and progression of coronary artery calcium: a prospective cohort study

Additional file 1: Table 1. The Risk of Progression of Coronary Calcium Score According to Baseline Brachial-Ankle Pulse Wave Velocity: Odds Ratio (95 % Confidence Interval) of Difference [√CAC(follow-up) − √CAC(baseline)] >2.5 by Pulse Wave Velocity quartiles—The SQRT Analysis. Table 2. The Risk of Progression of Coronary Calcium Score According to Pulse Pressure and Brachial-ankle Pulse Wave Velocity: Odds ratio (95 % Confidence Interval) of Difference [√CAC(follow-up) − √CAC(baseline)] >2.5 by Pulse Wave Velocity 50 % and pulse pressure 50 %—The SQRT Analysis. Table 3. The Risk of Progression of Coronary Calcium Score According to Baseline Brachial-Ankle Pulse Wave Velocity: Odds ratio (95 % Confidence Interval) of Coronary Calcium Score Change change >=10 by Pulse Wave Velocity Quartiles

The most important factors cause the avalanche falls

Job title: The most important factors cause the avalanche falls Purpose of work: The aim of this thesis was to gather and create a table of information on avalanches and to provide better guidance in layman issues avalanche hazards and risks associated with it, not only the Czech Republic but also abroad. Method: All necessary data were obtained from information resources through guided research and subsequently applied to my work. Obtained data were statistically processed and interpreted in both graphic and verbal form. Results: The biggest danger is the tipping point man on the slopes of the slope to 30ř- 50ř, most between 35ř- 45ř, on the leeward slopes with overblow snow less roughness and poor and very bad stability in alpine altitude level. Key words: Snow avalanche, freeride skiing, factor, subsidence, crawling, flake, tongues

Additional file 1: of An adaptive detection method for fetal chromosomal aneuploidy using cell-free DNA from 447 Korean women

Figure S1 showed optimally adaptive reference samples extracted from all reference samples. Figure S2 showed that GC correction played an important role in reducing the CV. Figures S3.1, S3.2, S4.1, S4.2, S5 and S6 represented similar results to our adaptive sample selection. Figure S7 represented the relationship of the reads fractions and the GC contents of samples. (DOCX 2063 kb

Additional file 2: Table S1. of Local compartment changes and regulatory landscape alterations in histone H1-depleted cells

The number of loci at which changes are observed for various combinations of epigenetic marks. (XLS 13 kb

Additional file 1: Figure S1. of Local compartment changes and regulatory landscape alterations in histone H1-depleted cells

DNA methylation changes per chromosome in histone H1-depleted ES cells. Figure S2. Hypomethylation in TKO cells occurs preferentially in gene-dense TADs regardless of GC content. Figure S3. ChIP-seq of four different histone marks in wild-type and TKO mouse ES cells. Figure S4. Large-scale changes in H3K4me1 and H3K4me3 sites upon depletion of histone H1. Figure S5. Analysis of DNA binding motifs at de novo formed DHSs in TKO cells. Figure S6. Over 10 % of de novo formed DHSs also show loss in CpG methylation in H1 TKO cells. Figure S7. Active chromatin marks accumulate in the most gene-dense TADs. Figure S8. Higher-order genome topology is very similar between wild-type and histone H1 TKO cells. Figure S9. Changes in compartment organization are not related to gene content, TAD size or differential expression. (PDF 4637 kb