Self-supervised Multi-view Stereo via Effective Co-Segmentation and Data-Augmentation

Recent studies have witnessed that self-supervised methods based on view synthesis obtain clear progress on multi-view stereo (MVS). However, existing methods rely on the assumption that the corresponding points among different views share the same color, which may not always be true in practice. This may lead to unreliable self-supervised signal and harm the final reconstruction performance. To address the issue, we propose a framework integrated with more reliable supervision guided by semantic co-segmentation and data-augmentation. Specially, we excavate mutual semantic from multi-view images to guide the semantic consistency. And we devise effective data-augmentation mechanism which ensures the transformation robustness by treating the prediction of regular samples as pseudo ground truth to regularize the prediction of augmented samples. Experimental results on DTU dataset show that our proposed methods achieve the state-of-the-art performance among unsupervised methods, and even compete on par with supervised methods. Furthermore, extensive experiments on Tanks&Temples dataset demonstrate the effective generalization ability of the proposed method.Comment: This paper is accepted by AAAI-21 with a Distinguished Paper Awar

Calcium channel α2δ1 proteins mediate trigeminal neuropathic pain states associated with aberrant excitatory synaptogenesis.

To investigate a potential mechanism underlying trigeminal nerve injury-induced orofacial hypersensitivity, we used a rat model of chronic constriction injury to the infraorbital nerve (CCI-ION) to study whether CCI-ION caused calcium channel α2δ1 (Cavα2δ1) protein dysregulation in trigeminal ganglia and associated spinal subnucleus caudalis and C1/C2 cervical dorsal spinal cord (Vc/C2). Furthermore, we studied whether this neuroplasticity contributed to spinal neuron sensitization and neuropathic pain states. CCI-ION caused orofacial hypersensitivity that correlated with Cavα2δ1 up-regulation in trigeminal ganglion neurons and Vc/C2. Blocking Cavα2δ1 with gabapentin, a ligand for the Cavα2δ1 proteins, or Cavα2δ1 antisense oligodeoxynucleotides led to a reversal of orofacial hypersensitivity, supporting an important role of Cavα2δ1 in orofacial pain processing. Importantly, increased Cavα2δ1 in Vc/C2 superficial dorsal horn was associated with increased excitatory synaptogenesis and increased frequency, but not the amplitude, of miniature excitatory postsynaptic currents in dorsal horn neurons that could be blocked by gabapentin. Thus, CCI-ION-induced Cavα2δ1 up-regulation may contribute to orofacial neuropathic pain states through abnormal excitatory synapse formation and enhanced presynaptic excitatory neurotransmitter release in Vc/C2