160 research outputs found

    New Twist on Shibori: How an Old Tradition Survives in the New World When Japanese Wooden Poles Are Replaced by American PVC Pipes

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    The subject of my talk is arashi shibori or pole wrap resist Although at times it is hard to recognize some arashi effects as such, technically arashi shibori is one of many forms of tie-dye. After thirty years of its exploration through art-to-wear, dyed and painted tapestries, three dimensional sculptures, and mixed media in the United States, various forms of tie-dye have now become part of the lexicon of American fabric design and fiberarts vocabulary. On the one hand, there has been much effort by textile specialists to circumvent the term tie-dye, due to its association with the Grateful Dead, the rock band cult figures with their dead heads dressed in tie-dyed T shirts. Or conversely, some textile scholars apply the term tie-dyed fabric to both ikat and shibori fabrics of various ethnic origins. However, tie-dyeing yarns to weave cloth and tie-dyeing cloth itself present two very different circumstances which require different processes, and therefore result in two dissimilar effects. Plangi and tritik have also been used to refer to some of the tie-dyed textiles, although I have often found an inconsistent use of the term plangi. As far as I can tell, the term tritik is always used to indicate stitch-resist technique, but plangi seems to refer to gathered and bound resist, or stitched and bound—sometimes capped resist, as well as to many other tie-dye processes. On the other hand, a majority of Japanese shibori terms are quite particular as to the process required to create a specific pattern. For those unfamiliar with Japanese terms, I choose to use English terms, such as clamp-resist, stitch and bound resist, and pole wrap resist, etc. Nonetheless, this confusion in the technical terms used in one area of surface design vocabulary is a good example of just how the contact and crossover of these culturally specific fiber arts practices may need further articulation to continue in a new context

    Arimatsu to Africa: Shibori Textiles Developed for African Trade in 1948–49

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    Shibori is a traditional Japanese textile term now widely used to classify a variety of patterns created on cloth by plucking, stitching, folding and then tightly knotting, binding, or clamping to compress and selectively resist dye penetration. The resulting patterns record the memory on cloth of the processes it sustained. Reading the resist marks on the cloth, shibori artisans can recreate the process or interpret various patterns. For the Textile Society of America’s Fifteenth Biennial Symposium in 2016 I organized a session with papers contributed by Françoise Cousin, Annie Ringuedé, and Ana Lisa Hedstrom and an exhibition titled “Arimatsu to Africa—examining shibori trade, techniques, and patterns,” which I curated in collaboration with Hiroshi Murase, a traditional artisan turned shibori producer and community leader, who was unable to attend the symposium. Joining me in Savannah were Ana Lisa Hedstrom and Annie Ringuedé. Ana Lisa is an internationally renowned American textile artist and contemporary interpreter and teacher of traditional shibori techniques. Her talk was titled “Ingenious and Practical: Parallels in the production of Arimatsu trade cloth and contemporary artists\u27 textiles.” Annie is a social anthropologist and urban planner from France, who amid extensive work on development and humanitarian projects in North Africa and West Africa became fascinated by the rich weaving and cloth-dyeing traditions in those regions, especially in Guinea. She shared a presentation on “West African Indigo Textiles under Influences, Case-studies: the Fouta-Jallon wrapper & the Mauritanian melhafa.” In addition, Françoise Cousin, retired curator of textiles at the musée du quai Branly in Paris and scholar, researcher, and author, was not able to attend the symposium but submitted a paper, “Indigo and resist-dyed textiles in Central and West Africa.

    Introduction to the Panel

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    The socio-historical factors of this era, including the effect of foreign culture, influenced consumer choice and production decisions regarding meisen textiles. Industrialization brought by the West generated demand for a new urban labor force, thereby providing for women work and educational opportunities that had not previously existed The cloth produced duringJhis time in history spoke of a kind of freedom and a conspicuous popular taste that celebrated women is changing position. Looking at the way women expressed themselves through textiles provides a wonderful approach to understanding this period of recent Japanese history. Meisen was a commercial term for a popular, widely distributed textile used by middle class women and children for every day wear (kimono and haori), by working class women for special or festive occasions, and by many people for coverlets and sitting cushions (futon and zabuton). The term meisen appeared in a wide range of written materials from the late Edo period (18th century) until the early 1960s

    Temporal convolutional neural networks to generate a head-related impulse response from one direction to another

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    Virtual sound synthesis is a technology that allows users to perceive spatial sound through headphones or earphones. However, accurate virtual sound requires an individual head-related transfer function (HRTF), which can be difficult to measure due to the need for a specialized environment. In this study, we proposed a method to generate HRTFs from one direction to the other. To this end, we used temporal convolutional neural networks (TCNs) to generate head-related impulse responses (HRIRs). To train the TCNs, publicly available datasets in the horizontal plane were used. Using the trained networks, we successfully generated HRIRs for directions other than the front direction in the dataset. We found that the proposed method successfully generated HRIRs for publicly available datasets. To test the generalization of the method, we measured the HRIRs of a new dataset and tested whether the trained networks could be used for this new dataset. Although the similarity evaluated by spectral distortion was slightly degraded, behavioral experiments with human participants showed that the generated HRIRs were equivalent to the measured ones. These results suggest that the proposed TCNs can be used to generate personalized HRIRs from one direction to another, which could contribute to the personalization of virtual sound

    Cilostazol Attenuates AngII-Induced Cardiac Fibrosis in apoE Deficient Mice

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    Cardiac fibrosis is characterized by the net accumulation of extracellular matrix in the myocardium and is an integral component of most pathological cardiac conditions. Cilostazol, a selective inhibitor of phosphodiesterase type III with anti-platelet, anti-mitogenic, and vasodilating properties, is widely used to treat the ischemic symptoms of peripheral vascular disease. Here, we investigated whether cilostazol has a protective effect against Angiotensin II (AngII)-induced cardiac fibrosis. Male apolipoprotein E-deficient mice were fed either a normal diet or a diet containing cilostazol (0.1% wt/wt). After 1 week of diet consumption, the mice were infused with saline or AngII (1000 ng kg(-1) min(-1)) for 28 days. AngII infusion increased heart/body weight ratio (p < 0.05), perivascular fibrosis (p < 0.05), and interstitial cardiac fibrosis (p < 0.0001), but were significantly attenuated by cilostazol treatment (p < 0.05, respectively). Cilostazol also reduced AngII-induced increases in fibrotic and inflammatory gene expression (p < 0.05, respectively). Furthermore, cilostazol attenuated both protein and mRNA abundance of osteopontin induced by AngII in vivo. In cultured human cardiac myocytes, cilostazol reduced mRNA expression of AngII-induced osteopontin in dose-dependent manner. This reduction was mimicked by forskolin treatment but was cancelled by co-treatment of H-89. Cilostazol attenuates AngII-induced cardiac fibrosis in mice through activation of the cAMP-PKA pathway

    Comparative genomics of Glandirana rugosa using unsupervised AI reveals a high CG frequency

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    The Japanese wrinkled frog (Glandirana rugosa) is unique in having both XX-XY and ZZ-ZW types of sex chromosomes within the species. The genome sequencing and comparative genomics with other frogs should be important to understand mechanisms of turnover of sex chromosomes within one species or during a short period. In this study, we analyzed the newly sequenced genome of G. rugosa using a batch-learning self-organizing map which is unsupervised artificial intelligence for oligonucleotide compositions. To clarify genome characteristics of G. rugosa, we compared its short oligonucleotide compositions in all 1-Mb genomic fragments with those of other six frog species (Pyxicephalus adspersus, Rhinella marina, Spea multiplicata, Leptobrachium leishanense, Xenopus laevis, and Xenopus tropicalis). In G. rugosa, we found an Mb-level large size of repeat sequences having a high identity with the W chromosome of the African bullfrog (P. adspersus). Our study concluded that G. rugosa has unique genome characteristics with a high CG frequency, and its genome is assumed to heterochromatinize a large size of genome via methylataion of CG

    Edaravone Attenuated Angiotensin II-Induced Atherosclerosis and Abdominal Aortic Aneurysms in Apolipoprotein E-Deficient Mice

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    Background: The aim of the study was to define whether edaravone, a free-radical scavenger, influenced angiotensin II (AngII)-induced atherosclerosis and abdominal aortic aneurysms (AAAs) formation. Methods: Male apolipoprotein E-deficient mice (8-12 weeks old) were fed with a normal diet for 5 weeks. Either edaravone (10 mg/kg/day) or vehicle was injected intraperitoneally for 5 weeks. After 1 week of injections, mice were infused subcutaneously with either AngII (1000 ng/kg/min, n = 16-17 per group) or saline (n = 5 per group) by osmotic minipumps for 4 weeks. Results: AngII increased systolic blood pressure equivalently in mice administered with either edaravone or saline. Edaravone had no effect on plasma total cholesterol concentrations and body weights. AngII infusion significantly increased ex vivo maximal diameters of abdominal aortas and en face atherosclerosis but was significantly attenuated by edaravone administration. Edaravone also reduced the incidence of AngII-induced AAAs. In addition, edaravone diminished AngII-induced aortic MMP-2 activation. Quantitative RT-PCR revealed that edaravone ameliorated mRNA abundance of aortic MCP-1 and IL-1 beta. Immunostaining demonstrated that edaravone attenuated oxidative stress and macrophage accumulation in the aorta. Furthermore, edaravone administration suppressed thioglycolate-induced mice peritoneal macrophages (MPMs) accumulation and mRNA abundance of MCP-1 in MPMs in male apolipoprotein E-deficient mice. In vitro, edaravone reduced LPS-induced mRNA abundance of MCP-1 in MPMs. Conclusions: Edaravone attenuated AngII-induced AAAs and atherosclerosis in male apolipoprotein E-deficient mice via anti-oxidative action and anti-inflammatory effect

    The Protective Effect of Chlorogenic Acid on Vascular Senescence via the Nrf2/HO-1 Pathway

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    The world faces the serious problem of aging. In this study, we aimed to investigate the effect of chlorogenic acid (CGA) on vascular senescence. C57/BL6 female mice that were 14 +/- 3 months old were infused with either Angiotensin II (AngII) or saline subcutaneously for two weeks. These mice were administered CGA of 20 or 40 mg/kg/day, or saline via oral gavage. AngII infusion developed vascular senescence, which was confirmed by senescence associated-beta-galactosidase (SA-beta-gal) staining. CGA administration attenuated vascular senescence in a dose-dependent manner, in association with the increase of Sirtuin 1 (Sirt1) and endothelial nitric oxide synthase (eNOS), and with the decrease of p-Akt, PAI-1, p53, and p21. In an in vitro study, with or without pre-treatment of CGA, Human Umbilical Vein Endothelial Cells (HUVECs) were stimulated with H(2)O(2)for an hour, then cultured in the absence or presence of 0.5-5.0 mu M CGA for the indicated time. Endothelial cell senescence was induced by H2O2, which was attenuated by CGA treatment. Pre-treatment of CGA increased Nrf2 in HUVECs. After H(2)O(2)treatment, translocation of Nrf2 into the nucleus and the subsequent increase of Heme Oxygenase-1 (HO-1) were observed earlier in CGA-treated cells. Furthermore, the HO-1 inhibitor canceled the beneficial effect of CGA on vascular senescence in mice. In conclusion, CGA exerts a beneficial effect on vascular senescence, which is at least partly dependent on the Nuclear factor erythroid 2-factor 2 (Nrf2)/HO-1 pathway

    Inhibition of interleukin-6 signaling attenuates aortitis, left ventricular hypertrophy and arthritis in interleukin-1 receptor antagonist deficient mice

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    The aim of the present study was to examine whether inhibition of Interleukin (IL)-6 signaling by MR16-1, an IL-6 receptor antibody, attenuates aortitis, cardiac hypertrophy, and arthritis in IL-1 receptor antagonist deficient (IL-1RA KO) mice. Four weeks old mice were intraperitoneally administered with either MR16-1 or non-immune IgG at dosages that were adjusted over time for 5 weeks. These mice were stratified into four groups: MR16-1 treatment groups, KO/MR low group (first 2.0 mg, following 0.5 mg/week, n=14) and KO/MR high group (first 4.0 mg, following 2.0 mg/week, n=19) in IL-1RA KO mice, and IgG treatment groups, KO/IgG group (first 2.0 mg, following 1.0 mg/week, n=22) in IL-1RA KO mice, and wild/IgG group (first 2.0 mg, following 1.0 mg/week, n=17) in wild mice. Aortitis, cardiac hypertrophy and arthropathy were histologically analyzed. Sixty-eight percent of the KO/IgG group developed aortitis (53% developed severe aortitis). In contrast, only 21% of the KO/MR high group developed mild aortitis, without severe aortitis (P<0.01, vs KO/IgG group). Infiltration of inflammatory cells, such as neutrophils, T cells, and macrophages, was frequently observed around aortic sinus of the KO/IgG group. Left ventricle and cardiomyocyte hypertrophy were observed in IL-1RA KO mice. Administration of high dosage of MR16-1 significantly suppressed cardiomyocyte hypertrophy. MR16-1 attenuated the incidence and severity of arthritis in IL-1RA KO mice in a dose-dependent manner. In conclusion, blockade of IL-6 signaling may exert a beneficial effect to attenuate severe aortitis, left ventricle hypertrophy, and arthritis
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