Disordered Quantum Spin Chains with Long-Range Antiferromagnetic Interactions

We investigate the magnetic susceptibility $\chi(T)$ of quantum spin chains of $N=1280$ spins with power-law long-range antiferromagnetic coupling as a function of their spatial decay exponent $\alpha$ and cutoff length $\xi$. The calculations are based on the strong disorder renormalization method which is used to obtain the temperature dependence of $\chi(T)$ and distribution functions of couplings at each renormalization step. For the case with only algebraic decay ($\xi = \infty$) we find a crossover at $\alpha^*=1.066$ between a phase with a divergent low-temperature susceptibility $\chi(T\rightarrow 0)$ for $\alpha > \alpha^*$ to a phase with a vanishing $\chi(T\rightarrow 0)$ for $\alpha < \alpha^*$. For finite cutoff lengths $\xi$, this crossover occurs at a smaller $\alpha^*(\xi)$. Additionally we study the localization of spin excitations for $\xi = \infty$ by evaluating the distribution function of excitation energies and we find a delocalization transition that coincides with the opening of the pseudo-gap at $\alpha_c=\alpha^*$.Comment: 6 pages, 7 figure

Valence Bond Solids and Their Quantum Melting in Hard-Core Bosons on the Kagome Lattice

Using large scale quantum Monte Carlo simulations and dual vortex theory we analyze the ground state phase diagram of hard-core bosons on the kagome lattice with nearest neighbor repulsion. In contrast to the case of a triangular lattice, no supersolid emerges for strong interactions. While a uniform superfluid prevails at half-filling, two novel solid phases emerge at densities $\rho=1/3$ and $\rho=2/3$. These solids exhibit an only partial ordering of the bosonic density, allowing for local resonances on a subset of hexagons of the kagome lattice. We provide evidence for a weakly first-order phase transition at the quantum melting point between these solid phases and the superfluid.Comment: 4 pages, 7 figure

S-Lemma with Equality and Its Applications

Let $f(x)=x^TAx+2a^Tx+c$ and $h(x)=x^TBx+2b^Tx+d$ be two quadratic functions having symmetric matrices $A$ and $B$. The S-lemma with equality asks when the unsolvability of the system $f(x)<0, h(x)=0$ implies the existence of a real number $\mu$ such that $f(x) + \mu h(x)\ge0, ~\forall x\in \mathbb{R}^n$. The problem is much harder than the inequality version which asserts that, under Slater condition, $f(x)<0, h(x)\le0$ is unsolvable if and only if $f(x) + \mu h(x)\ge0, ~\forall x\in \mathbb{R}^n$ for some $\mu\ge0$. In this paper, we show that the S-lemma with equality does not hold only when the matrix $A$ has exactly one negative eigenvalue and $h(x)$ is a non-constant linear function ($B=0, b\not=0$). As an application, we can globally solve $\inf\{f(x)\vert h(x)=0\}$ as well as the two-sided generalized trust region subproblem $\inf\{f(x)\vert l\le h(x)\le u\}$ without any condition. Moreover, the convexity of the joint numerical range $\{(f(x), h_1(x),\ldots, h_p(x)):~x\in\Bbb R^n\}$ where $f$ is a (possibly non-convex) quadratic function and $h_1(x),\ldots,h_p(x)$ are affine functions can be characterized using the newly developed S-lemma with equality.Comment: 34 page

A linear theory for control of non-linear stochastic systems

We address the role of noise and the issue of efficient computation in stochastic optimal control problems. We consider a class of non-linear control problems that can be formulated as a path integral and where the noise plays the role of temperature. The path integral displays symmetry breaking and there exist a critical noise value that separates regimes where optimal control yields qualitatively different solutions. The path integral can be computed efficiently by Monte Carlo integration or by Laplace approximation, and can therefore be used to solve high dimensional stochastic control problems.Comment: 5 pages, 3 figures. Accepted to PR

Evolution of the single-hole spectral function across a quantum phase transition in the anisotropic-triangular-lattice antiferromagnet

We study the evolution of the single-hole spectral function when the ground state of the anisotropic-triangular-lattice antiferromagnet changes from the incommensurate magnetically-ordered phase to the spin-liquid state. In order to describe both of the ground states on equal footing, we use the large-N approach where the transition between these two phases can be obtained by controlling the quantum fluctuations via an 'effective' spin magnitude. Adding a hole into these ground states is described by a t-J type model in the slave-fermion representation. Implications of our results to possible future ARPES experiments on insulating frustrated magnets, especially Cs$_2$CuCl$_4$, are discussed.Comment: 8 pages, 7 figure

Extracellular signal-regulated kinases mediate the enhancing effects of inflammatory mediators on resurgent currents in dorsal root ganglion neurons

Previously we reported that a group of inflammatory mediators significantly enhanced resurgent currents in dorsal root ganglion neurons. To understand the underlying intracellular signaling mechanism, we investigated the effects of inhibition of extracellular signal-regulated kinases and protein kinase C on the enhancing effects of inflammatory mediators on resurgent currents in rat dorsal root ganglion neurons. We found that the extracellular signal-regulated kinases inhibitor U0126 completely prevented the enhancing effects of the inflammatory mediators on both Tetrodotoxin-sensitive and Tetrodotoxin-resistant resurgent currents in both small and medium dorsal root ganglion neurons. U0126 substantially reduced repetitive firing in small dorsal root ganglion neurons exposed to inflammatory mediators, consistent with prevention of resurgent current amplitude increases. The protein kinase C inhibitor Bisindolylmaleimide I also showed attenuating effects on resurgent currents, although to a lesser extent compared to extracellular signal-regulated kinases inhibition. These results indicate a critical role of extracellular signal-regulated kinases signaling in modulating resurgent currents and membrane excitability in dorsal root ganglion neurons treated with inflammatory mediators. It is also suggested that targeting extracellular signal-regulated kinases-resurgent currents might be a useful strategy to reduce inflammatory pain