E-cadherin immunostaining on tissue of epithelial type of meningioma (WHO I grade), with amplification of 200X.

<p>Expression level was determined as +.</p

Creación de la población PQUI-1 desarrollada con tolerancia a frío mediante selección recurrente en arroz = PQUI-1 rice population developed with cold tolerance by recurrent selection

<p>Expression level was determined as ++.</p

β-catenin immunostaining on tissue of epithelia type of meningioma (WHO I grade), with amplification of 200X.

<p>Expression level was determined as +.</p

β-catenin immunostaining on tissue of malignant type of meningioma (WHO III grade), with amplification of 200X.

<p>Expression level was determined as +.</p

The expression levels of E-cadherin and β-catenin in different pathological grades of meningioma.

<p>The expression levels of E-cadherin and β-catenin in different pathological grades of meningioma.</p

The expression levels of E-cadherin and β-catenin with the postoperative recurrence of meningioma.

<p>The expression levels of E-cadherin and β-catenin with the postoperative recurrence of meningioma.</p

The expression of E-cadherin and β-catenin with the different pathological types of meningioma.

<p>The expression of E-cadherin and β-catenin with the different pathological types of meningioma.</p

DataSheet_1_Construction and validation of a prognostic model with RNA binding protein-related mRNAs for the HBV-related hepatocellular carcinoma patients.docx

Hepatocellular carcinoma (HCC) is a common malignancy worldwide with poor clinical outcomes, and the infection of hepatitis B virus (HBV) is the leading cause of this disease. Mounting evidence shows that RNA binding proteins (RBPs) can modulate the progression of cancers. However, the functions and clinical implications of RBP-related mRNAs in HBV-related HCC remain largely unclear. Therefore, we aim to develop a prognostic model based on the RBP-related mRNAs for HBV-related HCC patients. Firstly, we identified 626 differentially expressed RBP-related mRNAs in the HBV-related HCC through the Pearson correlation analysis. Subsequently, the Kaplan-Meier survival, univariate, Least Absolute Shrinkage and Selection Operator (LASSO), and multivariate Cox regression analyses were used to construct a prognostic model comprised of five RBP-related mRNAs. Furthermore, the patients were categorized into the high- and low-risk groups by the prognostic model and the patients in the high-risk group had a poor prognosis. Additionally, the prognostic model was an independent predictor of prognosis, and the accuracy of the prognostic model was proved by the receiver operator characteristic (ROC) analysis. Furthermore, the functional enrichment analysis revealed that various cancer-promoting processes were enriched in the high-risk group. Taken together, our study may provide the HBV-related HCC biomarkers of prognosis to improve the clinical outcomes of patients.</p

Double Immunofluorescence staining for Bax and Cyto C in hDPCs exposed to dental monomers in the absence or presence of NAC.

<p>G<i>reen fluorescence</i> represents Bax, whereas <i>red green fluorescence</i> represents Cyto C. <i>Yellow color</i> in the overlay of these two images indicates co-localization of Bax and Cyto C (presumably in mitochondria).</p

Changes of mitochondrial morphology observed by transmission electron microscopic (TEM).

<p>(A, E) Control hDPCs and (I, M) hDPCs treated with 10 mM NAC showing a well preserved morphology. (B-D, F-H) Following 24 h dental monomer treatment (1mM HEMA, 5mM MMA or 1mM TEGDMA), some cells showed nuclear and cytoplasmic signs of apoptosis (condensed/peripheralized nuclear chromatin and/or cytoplasm, cytoplasmic vacuolization). Larger and elongated mitochondria (black arrow) with impaired mitochondrial membrane integrity (white arrow), reduced number of cristae and deformed cristae (wedged) could be observed. (J-L, N-P) The presence of 10 mM NAC restored cell and mitochondrial morphology. Although larger and elongated mitochondria (black arrow), and deformed cristae (wedged) could still be observed, the structural integrity of mitochondrial membrane was preserved.</p